Upregulated TRIM32 correlates with enhanced cell proliferation and poor prognosis in hepatocellular carcinoma
暂无分享,去创建一个
Zhipeng Lin | R. Ni | Xiaopeng Cui | Wen-Kai Ni | Jinxia Liu | Yuyan Chen | Baoying Hu | Zhongyi Shen | C. Duan | Huiling Zhou | Xiaofei Mao | Xiaohang Shan | Lingling Chen | Jiale Lv
[1] Q. Ye,et al. Peritumoral Cbl is a strong independent prognostic marker after curative resection of hepatocellular carcinoma , 2015, Oncotarget.
[2] Qianshan Ding,et al. Downregulation of TRIM21 contributes to hepatocellular carcinoma carcinogenesis and indicates poor prognosis of cancers , 2015, Tumor Biology.
[3] Zheng Zhang,et al. The global burden of liver disease: The major impact of China , 2014, Hepatology.
[4] Y. Kaneko,et al. Trim32 facilitates degradation of MYCN on spindle poles and induces asymmetric cell division in human neuroblastoma cells. , 2014, Cancer research.
[5] V. Belyĭ,et al. E3 ubiquitin ligase TRIM32 negatively regulates tumor suppressor p53 to promote tumorigenesis , 2014, Cell Death and Differentiation.
[6] S. Gygi,et al. Trim32 reduces PI3K–Akt–FoxO signaling in muscle atrophy by promoting plakoglobin–PI3K dissociation , 2014, The Journal of cell biology.
[7] H. Shu,et al. TRIM32 Protein Modulates Type I Interferon Induction and Cellular Antiviral Response by Targeting MITA/STING Protein for K63-linked Ubiquitination* , 2012, The Journal of Biological Chemistry.
[8] S. Hatakeyama,et al. TRIM proteins and cancer , 2011, Nature Reviews Cancer.
[9] Eugene Berezikov,et al. The TRIM-NHL Protein TRIM32 Activates MicroRNAs and Prevents Self-Renewal in Mouse Neural Progenitors , 2009, Cell.
[10] K. Ozato,et al. TRIM family proteins and their emerging roles in innate immunity , 2008, Nature Reviews Immunology.
[11] A. Albor,et al. Novel initiation genes in squamous cell carcinomagenesis: A role for substrate‐specific ubiquitylation in the control of cell survival , 2007, Molecular carcinogenesis.
[12] T. de Baère,et al. Gemcitabine plus oxaliplatin (GEMOX) in patients with advanced hepatocellular carcinoma (HCC) , 2007, Cancer.
[13] A. Albor,et al. The Interaction of Piasy with Trim32, an E3-Ubiquitin Ligase Mutated in Limb-girdle Muscular Dystrophy Type 2H, Promotes Piasy Degradation and Regulates UVB-induced Keratinocyte Apoptosis through NFκB* , 2006, Journal of Biological Chemistry.
[14] G. Meroni,et al. TRIM/RBCC, a novel class of ‘single protein RING finger’ E3 ubiquitin ligases , 2005, BioEssays : news and reviews in molecular, cellular and developmental biology.
[15] W. Stremmel,et al. Enhanced caspase-8 recruitment to and activation at the DISC is critical for sensitisation of human hepatocellular carcinoma cells to TRAIL-induced apoptosis by chemotherapeutic drugs , 2004, Cell Death and Differentiation.
[16] G. Lozano,et al. RING protein Trim32 associated with skin carcinogenesis has anti-apoptotic and E3-ubiquitin ligase properties. , 2003, Carcinogenesis.
[17] Alessandro Guffanti,et al. The tripartite motif family identifies cell compartments , 2001, The EMBO journal.
[18] F. Slack,et al. A novel repeat domain that is often associated with RING finger and B-box motifs. , 1998, Trends in biochemical sciences.
[19] A. Weissman,et al. Regulating protein degradation by ubiquitination. , 1997, Immunology today.
[20] 加納 里志. Tripartite motif protein 32 facilitates cell growth and migration via degradation of Abl-interactor 2 , 2009 .