Involvement of src-kinase activation in ischemic preconditioning induced protection of mouse brain.
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[1] T. G. Singh,et al. Involvement of cyclic adenosine diphosphoribose receptor activation in ischemic preconditioning induced protection in mouse brain , 2010, Brain Research.
[2] A. Ferrara,et al. The usefulness of operant conditioning procedures to assess long-lasting deficits following transient focal ischemia in mice , 2009, Behavioural Brain Research.
[3] K. Fukunaga,et al. Nobiletin improves brain ischemia-induced learning and memory deficits through stimulation of CaMKII and CREB phosphorylation , 2009, Brain Research.
[4] P. Gressens,et al. The Preconditioning Effect of Sevoflurane on the Oxygen Glucose-Deprived Hippocampal Slice: The Role of Tyrosine Kinases and Duration of Ischemia , 2009, Anesthesia and analgesia.
[5] Nirmal Singh,et al. Diethyl dithiocarbamic acid, a possible nuclear factor kappa B inhibitor, attenuates ischemic postconditioning-induced attenuation of cerebral ischemia-reperfusion injury in mice. , 2009, Canadian journal of physiology and pharmacology.
[6] T. G. Singh,et al. Pharmacological preconditioning of the brain: a possible interplay between opioid and calcitonin gene related peptide transduction systems. , 2008, Pharmacological reports : PR.
[7] Nirmal Singh,et al. Implication of mast cell degranulation in ischemic preconditioning‐induced prevention of cerebral injury , 2008, Fundamental & clinical pharmacology.
[8] P. Insel,et al. Caveolin‐1 expression is essential for N‐methyl‐D‐aspartate receptor‐mediated Src and extracellular signal‐regulated kinase 1/2 activation and protection of primary neurons from ischemic cell death , 2008, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[9] O. Ottersen,et al. Neuroprotective effects of inhibiting N-methyl-d-aspartate receptors, P2X receptors and the mitogen-activated protein kinase cascade: A quantitative analysis in organotypical hippocampal slice cultures subjected to oxygen and glucose deprivation , 2005, Neuroscience.
[10] L. Teves,et al. Inhibition of protein kinase C reduces ischemia‐induced tyrosine phosphorylation of the N‐methyl‐d‐aspartate receptor , 2003, Journal of neurochemistry.
[11] Sukhwinder Thandi,et al. Group‐I metabotropic glutamate receptors, mGlu1a and mGlu5a, couple to extracellular signal‐regulated kinase (ERK) activation via distinct, but overlapping, signalling pathways , 2002, Journal of neurochemistry.
[12] D. S. Cowen,et al. Activation of Extracellular-Regulated Kinase by 5-Hydroxytryptamine2A Receptors in PC12 Cells is Protein Kinase C-Independent and Requires Calmodulin and Tyrosine Kinases , 2002, Journal of Pharmacology and Experimental Therapeutics.
[13] R. Bolli,et al. Role of Src protein tyrosine kinases in late preconditioning against myocardial infarction. , 2002, American journal of physiology. Heart and circulatory physiology.
[14] M. Lazdunski,et al. Activation of the Nuclear Factor-κB Is a Key Event in Brain Tolerance , 2001, The Journal of Neuroscience.
[15] Michael Chopp,et al. Src deficiency or blockade of Src activity in mice provides cerebral protection following stroke , 2001, Nature Medicine.
[16] M. Gishizky,et al. SU6656, a Selective Src Family Kinase Inhibitor, Used To Probe Growth Factor Signaling , 2000, Molecular and Cellular Biology.
[17] R. Neumar. Molecular mechanisms of ischemic neuronal injury. , 2000, Annals of emergency medicine.
[18] L. Teves,et al. The Effect of Transient Global Ischemia on the Interaction of Src and Fyn with the N-Methyl-d-Aspartate Receptor and Postsynaptic Densities: Possible Involvement of Src Homology 2 Domains , 1999, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[19] M. Rudin,et al. Calcineurin inhibitors FK506 and SDZ ASM 981 alleviate the outcome of focal cerebral ischemic/reperfusion injury. , 1999, The Journal of pharmacology and experimental therapeutics.
[20] Y. Zou,et al. Oxidative stress activates extracellular signal-regulated kinases through Src and Ras in cultured cardiac myocytes of neonatal rats. , 1997, The Journal of clinical investigation.
[21] C. Velasco,et al. Endothelin-1 and myocardial preconditioning. , 1996, American heart journal.
[22] A. Hsu,et al. Morphine mimics the cardioprotective effect of ischemic preconditioning via a glibenclamide-sensitive mechanism in the rat heart. , 1996, Circulation research.
[23] Sheila M. Thomas,et al. Calcium influx induces neurite growth through a Src-Ras signaling cassette , 1995, Neuron.
[24] J. Downey,et al. Role of bradykinin in protection of ischemic preconditioning in rabbit hearts. , 1995, Circulation research.
[25] K. Mullane,et al. Harnessing an endogenous cardioprotective mechanism: cellular sources and sites of action of adenosine. , 1995, Journal of molecular and cellular cardiology.
[26] J. Downey,et al. Pretreatment with angiotensin II activates protein kinase C and limits myocardial infarction in isolated rabbit hearts. , 1995, Journal of molecular and cellular cardiology.
[27] G. Gross,et al. Role of nitric oxide, muscarinic receptors, and the ATP-sensitive K+ channel in mediating the effects of acetylcholine to mimic preconditioning in dogs. , 1993, Circulation research.
[28] C. Cairns,et al. Preconditioning against myocardial dysfunction after ischemia and reperfusion by an alpha 1-adrenergic mechanism. , 1993, Circulation research.
[29] G. Gross,et al. Acetylcholine mimics ischemic preconditioning via a glibenclamide-sensitive mechanism in dogs. , 1993, The American journal of physiology.
[30] B. Dobkin,et al. The rehabilitation of elderly stroke patients. , 1991, Clinics in geriatric medicine.
[31] K. Mikoshiba,et al. ‘Ischemic tolerance’ phenomenon found in the brain , 1990, Brain Research.
[32] Himori Norio,et al. Cerebral ischemia model with conscious mice. Involvement of NMDA receptor activation and derangement of learning and memory ability. , 1990 .
[33] T. Kameyama,et al. Utility of an elevated plus-maze for the evaluation of memory in mice: effects of nootropics, scopolamine and electroconvulsive shock , 1990, Psychopharmacology.
[34] R. Jennings,et al. Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium. , 1986, Circulation.
[35] H. Holländer,et al. Vergleichende licht- und fluoreszenzmikroskopische Untersuchungen der experimentellen allergischen Encephalomyelitis des Meerschweinchens , 1965, Acta Neuropathologica.
[36] N W DUNHAM,et al. A note on a simple apparatus for detecting neurological deficit in rats and mice. , 1957, Journal of the American Pharmaceutical Association. American Pharmaceutical Association.
[37] J. Miller,et al. The role of postischemic recirculation in the development of ischemic neuronal injury following complete cerebral ischemia , 2004, Acta Neuropathologica.
[38] S. Doctrow,et al. Synthetic combined superoxide dismutase/catalase mimetics are protective as a delayed treatment in a rat stroke model: a key role for reactive oxygen species in ischemic brain injury. , 1998, The Journal of pharmacology and experimental therapeutics.
[39] J. Whisnant. The decline of stroke. , 1984, Stroke.