Alternation of neurofilaments in immune-mediated injury of spinal cord motor neurons
暂无分享,去创建一个
Dong-xia Wu | Y.-S. Guo | Y. Liu | S-Y Wu | C.-Y. Li | C. Yang | L. Xu | Y. Zhang | Dong-xia Wu
[1] M. Strong,et al. The Pathobiology of Amyotrophic Lateral Sclerosis: A Proteinopathy? , 2005, Journal of neuropathology and experimental neurology.
[2] P. Shaw,et al. Molecular and cellular pathways of neurodegeneration in motor neurone disease , 2005, Journal of Neurology, Neurosurgery & Psychiatry.
[3] C. Shaw,et al. Neurofilament heavy chain side arm phosphorylation regulates axonal transport of neurofilaments , 2003, The Journal of cell biology.
[4] B. Liu,et al. Role of Nitric Oxide in Inflammation‐Mediated Neurodegeneration , 2002, Annals of the New York Academy of Sciences.
[5] S Cluskey,et al. Mechanisms of neurodegeneration in amyotrophic lateral sclerosis , 2001, Molecular pathology : MP.
[6] H. Pant,et al. Cyclin-dependent protein kinase 5 (Cdk5) and the regulation of neurofilament metabolism. , 2001, European journal of biochemistry.
[7] T. Gotow,et al. Neurofilaments in health and disease , 2000, Medical Electron Microscopy.
[8] C. Geula,et al. Motor neurons are rich in non-phosphorylated neurofilaments: cross-species comparison and alterations in ALS , 2000, Brain Research.
[9] M. Strong. Neurofilament metabolism in sporadic amyotrophic lateral sclerosis , 1999, Journal of the Neurological Sciences.
[10] Jean-Pierre Julien,et al. Neurofilament functions in health and disease , 1999, Current Opinion in Neurobiology.
[11] P. Leigh,et al. Cyclin dependent kinase-5 (CDK-5) phosphorylates neurofilament heavy (NF-H) chain to generate epitopes for antibodies that label neurofilament accumulations in amyotrophic lateral sclerosis (ALS) and is present in affected motor neurones in ALS , 1999, Progress in Neuro-Psychopharmacology and Biological Psychiatry.
[12] Guy A. Rouleau,et al. SOD1 mutation is assosiated with accumulation of neurofilaments in amyotrophic lateral scelaries , 1996 .
[13] A. Clark,et al. SOD1 mutation is associated with accumulation of neurofilaments in amyotrophic lateral sclerosis. , 1996, Annals of neurology.
[14] S. Brady. Mice overexpressing the human neurofilament heavy gene as a model of ALS , 1995, Neurobiology of Aging.
[15] E. Stefani,et al. Amyotrophic lateral sclerosis patient antibodies label Ca2+ channel α1 subunit , 1994 .
[16] E. Stefani,et al. Amyotrophic lateral sclerosis patient antibodies label Ca2+ channel alpha 1 subunit. , 1994, Annals of neurology.
[17] J. Coyle,et al. Oxidative stress, glutamate, and neurodegenerative disorders. , 1993, Science.
[18] E. Stefani,et al. Serum antibodies to L-type calcium channels in patients with amyotrophic lateral sclerosis. , 1992, The New England journal of medicine.
[19] S. Appel,et al. IgG reactivity in the spinal cord and motor cortex in amyotrophic lateral sclerosis. , 1990, Archives of neurology.
[20] S. Appel,et al. Motor neuron destruction in guinea pigs immunized with bovine spinal cord ventral horn homogenate: experimental autoimmune gray matter disease , 1990, Journal of Neuroimmunology.
[21] S. Appel,et al. Experimental autoimmune motoneuron disease , 1989, Annals of neurology.
[22] S. Appel,et al. Amyotrophic lateral sclerosis. Associated clinical disorders and immunological evaluations. , 1986, Archives of Neurology.