We Aim to Determine the Regulatory Mechanisms Involved in the Switch Between Normal and Aberrant Angiogenesis Induced by Different Doses of Vascular Endothelial Growth Factor
暂无分享,去创建一个
The 2 major prerequisites to pursue such a systematicinvestigation are a highly controlled angiogenesis modeland a technique that allows specific isolation of affectedvascular cells from their microenvironment in tissue.Therefore, Dr Banfi and his coresearchers have combineda unique cell-based platform for controlled in vivo geneexpression with laser capture microdissection to perform astage-specific and VEGF dose-dependent analysis of thevascular messenger RNA and microRNA transcriptome.The group has developed a unique model of VEGFdose-dependent angiogenesis in mouse skeletal musclebased on the implantation of monoclonal populations ofretrovirally transduced myoblasts, which ensure homoge-neous expression of a specific VEGF dose, alone or togetherwith PDGF-BB, in the microenvironment around eachengrafted fibre. The clinically relevant tissue for peripheralartery disease (ie, skeletal muscle) is used, in the absenceof ischaemia, to avoid the confounding factors of inflam-mation and upregulated endogenous factors. Normal andaberrant vascular structures developing in the areas ofimplantation over a previously defined time course aremicrodissected at the International Centre for GeneticEngineering and Biotechnology, Trieste, Italy. Total RNA
[1] M. Merchant,et al. Therapeutic angiogenesis due to balanced single‐vector delivery of VEGF and PDGF‐BB , 2012, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[2] R. Hlushchuk,et al. VEGF over-expression in skeletal muscle induces angiogenesis by intussusception rather than sprouting , 2012, Angiogenesis.
[3] H. Blau,et al. Microenvironmental VEGF concentration, not total dose, determines a threshold between normal and aberrant angiogenesis. , 2004, The Journal of clinical investigation.