The combination of oral quercetin supplementation and exercise prevents brain mitochondrial biogenesis

[1]  A. Martínez-Amat,et al.  Oral quercetin supplementation hampers skeletal muscle adaptations in response to exercise training , 2014, Scandinavian journal of medicine & science in sports.

[2]  A. Martínez-Amat,et al.  Ergogenic effects of quercetin supplementation in trained rats , 2013, Journal of the International Society of Sports Nutrition.

[3]  Rick B. Vega,et al.  Transcriptional integration of mitochondrial biogenesis , 2012, Trends in Endocrinology & Metabolism.

[4]  M. Nikolaidis,et al.  Does Vitamin C and E Supplementation Impair the Favorable Adaptations of Regular Exercise? , 2012, Oxidative medicine and cellular longevity.

[5]  J. Spencer,et al.  Flavonoids as modulators of memory and learning: molecular interactions resulting in behavioural effects , 2012, Proceedings of the Nutrition Society.

[6]  J. Davis,et al.  Exercise training increases mitochondrial biogenesis in the brain. , 2011, Journal of applied physiology.

[7]  E. Bedrick,et al.  Thermotolerance and heat acclimation may share a common mechanism in humans. , 2011, American journal of physiology. Regulatory, integrative and comparative physiology.

[8]  M. Ristow,et al.  Extending life span by increasing oxidative stress. , 2011, Free Radical Biology & Medicine.

[9]  J. Auwerx,et al.  The secret life of NAD+: an old metabolite controlling new metabolic signaling pathways. , 2010, Endocrine reviews.

[10]  Michael Stumvoll,et al.  Antioxidants prevent health-promoting effects of physical exercise in humans , 2009, Proceedings of the National Academy of Sciences.

[11]  J. Davis,et al.  Quercetin increases brain and muscle mitochondrial biogenesis and exercise tolerance. , 2009, American journal of physiology. Regulatory, integrative and comparative physiology.

[12]  S. Powers,et al.  Exercise-induced oxidative stress: cellular mechanisms and impact on muscle force production. , 2008, Physiological reviews.

[13]  Christoph Handschin,et al.  The role of exercise and PGC1α in inflammation and chronic disease , 2008, Nature.

[14]  P. Puigserver,et al.  Metabolic adaptations through the PGC‐1α and SIRT1 pathways , 2008 .

[15]  J. Spencer.  The interactions of flavonoids within neuronal signalling pathways , 2007, Genes & Nutrition.

[16]  H. Naito,et al.  Effects of exercise on brain function: role of free radicals. , 2007, Applied physiology, nutrition, and metabolism = Physiologie appliquee, nutrition et metabolisme.

[17]  Andre Fischer,et al.  SIRT1 deacetylase protects against neurodegeneration in models for Alzheimer's disease and amyotrophic lateral sclerosis , 2007, The EMBO journal.

[18]  A. Bast,et al.  The quercetin paradox. , 2007, Toxicology and applied pharmacology.

[19]  P. Puigserver,et al.  Metabolic control of muscle mitochondrial function and fatty acid oxidation through SIRT1/PGC‐1α , 2007, The EMBO journal.

[20]  P. Puigserver,et al.  Resveratrol Improves Mitochondrial Function and Protects against Metabolic Disease by Activating SIRT1 and PGC-1α , 2006, Cell.

[21]  A. Bast,et al.  The reversibility of the glutathionyl-quercetin adduct spreads oxidized quercetin-induced toxicity. , 2005, Biochemical and biophysical research communications.

[22]  N. Güzel,et al.  The effects of vitamin C supplementation on oxidative stress and antioxidant content in the brains of chronically exercised rats , 2005, Molecular and Cellular Biochemistry.

[23]  D. Hovda,et al.  Voluntary exercise following traumatic brain injury: brain-derived neurotrophic factor upregulation and recovery of function , 2004, Neuroscience.

[24]  Liliana Jiménez,et al.  Polyphenols: food sources and bioavailability. , 2004, The American journal of clinical nutrition.

[25]  C. Rice-Evans,et al.  Cellular uptake and metabolism of flavonoids and their metabolites: implications for their bioactivity. , 2004, Archives of biochemistry and biophysics.

[26]  C. Manach,et al.  Quercetin, but not its glycosides, is absorbed from the rat stomach. , 2002, Journal of agricultural and food chemistry.

[27]  Thomas D. Schmittgen,et al.  Analysis of relative gene expression data using real-time quantitative PCR and the 2(-Delta Delta C(T)) Method. , 2001, Methods.

[28]  T. Kaneko,et al.  Regular exercise improves cognitive function and decreases oxidative damage in rat brain , 2001, Neurochemistry International.

[29]  C. Rice-Evans,et al.  Decomposition of cocoa procyanidins in the gastric milieu. , 2000, Biochemical and biophysical research communications.

[30]  C. Rice-Evans,et al.  The small intestine can both absorb and glucuronidate luminal flavonoids , 1999, FEBS letters.

[31]  M. M. Bradford.  A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding. , 1976, Analytical biochemistry.

[32]  I W SIZER,et al.  A spectrophotometric method for measuring the breakdown of hydrogen peroxide by catalase. , 1952, The Journal of biological chemistry.

[33]  R. Shephard.  Antioxidants prevent health-promoting effects of physical exercise in humans , 2010 .

[34]  J. Sastre,et al.  Oral administration of vitamin C decreases muscle mitochondrial biogenesis and hampers training-induced adaptations in endurance performance. , 2008, The American journal of clinical nutrition.

[35]  Christoph Handschin,et al.  The Role of Exercise and Pgc1alpha in Inflammation and Chronic Disease , 2022 .

[36]  E. Stadtman,et al.  Determination of carbonyl content in oxidatively modified proteins. , 1990, Methods in enzymology.

[37]  L. Flohé,et al.  Superoxide dismutase assays. , 1984, Methods in enzymology.