Loss of amyloid precursor protein exacerbates early inflammation in Niemann-Pick disease type C
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Christopher G. Wilson | S. Soriano | D. Bellinger | S. Shin | L. Siebold | Karina Mayagoitia | Alexandra Shin | M. Rubini
[1] M. Klein,et al. Depletion of regulatory T cells increases T cell brain infiltration, reactive astrogliosis, and interferon-γ gene expression in acute experimental traumatic brain injury , 2019, Journal of Neuroinflammation.
[2] Christopher G. Wilson,et al. Interferon downstream signaling is activated early in pre-symptomatic Niemann-Pick disease type C , 2019, Neuroscience Letters.
[3] Christopher G. Wilson,et al. Cellular hormetic response to 27-hydroxycholesterol promotes neuroprotection through AICD induction of MAST4 abundance and kinase activity , 2017, Scientific Reports.
[4] Fei Li,et al. Erythropoietin regulates immune/inflammatory reaction and improves neurological function outcomes in traumatic brain injury , 2017, Brain and behavior.
[5] A. Lucia,et al. Niemann-Pick disease treatment: a systematic review of clinical trials. , 2015, Annals of translational medicine.
[6] A. Suzumura,et al. CCL11 enhances excitotoxic neuronal death by producing reactive oxygen species in microglia , 2015, Glia.
[7] P. Das,et al. Altered Innate Immune and Glial Cell Responses to Inflammatory Stimuli in Amyloid Precursor Protein Knockout Mice , 2015, PloS one.
[8] C. Bocti,et al. β-Amyloid peptides display protective activity against the human Alzheimer’s disease-associated herpes simplex virus-1 , 2015, Biogerontology.
[9] C. Mackay. CXCR3⁺CCR5⁺ T cells and autoimmune diseases: guilty as charged? , 2014, The Journal of clinical investigation.
[10] Shweta Tripathi,et al. Alzheimer's Associated β-Amyloid Protein Inhibits Influenza A Virus and Modulates Viral Interactions with Phagocytes , 2014, PloS one.
[11] H. Lassmann,et al. Expression of Ccl11 Associates with Immune Response Modulation and Protection against Neuroinflammation in Rats , 2012, PloS one.
[12] Zhong-Hui Duan,et al. Fold change and p-value cutoffs significantly alter microarray interpretations , 2012, BMC Bioinformatics.
[13] J. Hibbert,et al. CXCL10/IP-10 in infectious diseases pathogenesis and potential therapeutic implications , 2011, Cytokine & growth factor reviews.
[14] J. Cooper,et al. Loss of amyloid precursor protein in a mouse model of Niemann–Pick type C disease exacerbates its phenotype and disrupts tau homeostasis , 2011, Neurobiology of Disease.
[15] I. Campbell,et al. Review: The chemokine receptor CXCR3 and its ligands CXCL9, CXCL10 and CXCL11 in neuroimmunity – a tale of conflict and conundrum , 2010, Neuropathology and applied neurobiology.
[16] F. Platt,et al. Lipids on Trial: The Search for the Offending Metabolite in Niemann‐Pick type C Disease , 2010, Traffic.
[17] B. Hyman,et al. The Alzheimer's Disease-Associated Amyloid β-Protein Is an Antimicrobial Peptide , 2010, PloS one.
[18] B. Fischer,et al. Abeta40, either soluble or aggregated, is a remarkably potent antioxidant in cell-free oxidative systems. , 2009, Biochemistry.
[19] R. Sidman,et al. TLR8: An Innate Immune Receptor in Brain, Neurons and Axons , 2007, Cell cycle.
[20] Pablo Tamayo,et al. Gene set enrichment analysis: A knowledge-based approach for interpreting genome-wide expression profiles , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[21] M. Diamond,et al. Neuronal CXCL10 Directs CD8+ T-Cell Recruitment and Control of West Nile Virus Encephalitis , 2005, Journal of Virology.
[22] Colin L Masters,et al. Gene knockout of amyloid precursor protein and amyloid precursor‐like protein‐2 increases cellular copper levels in primary mouse cortical neurons and embryonic fibroblasts , 2004, Journal of neurochemistry.
[23] K. Schroder,et al. Interferon- : an overview of signals, mechanisms and functions , 2004 .
[24] Oreste Acuto,et al. CD28-mediated co-stimulation: a quantitative support for TCR signalling , 2003, Nature Reviews Immunology.
[25] M. Vanier. Niemann-Pick disease type C , 2010, Orphanet journal of rare diseases.
[26] J. Mudgett,et al. Antibody‐mediated blockade of the CXCR3 chemokine receptor results in diminished recruitment of T helper 1 cells into sites of inflammation , 2003, Journal of leukocyte biology.
[27] M. Kawaguchi,et al. Differential regulation of eotaxin expression by IFN-γ in airway epithelial cells ☆ ☆☆ , 2003 .
[28] K. Zou,et al. A Novel Function of Monomeric Amyloid β-Protein Serving as an Antioxidant Molecule against Metal-Induced Oxidative Damage , 2002, The Journal of Neuroscience.
[29] M. Dziejman,et al. IFN-γ-Inducible Protein 10 (IP-10; CXCL10)-Deficient Mice Reveal a Role for IP-10 in Effector T Cell Generation and Trafficking1 , 2002, The Journal of Immunology.
[30] J. Flier,et al. Differential expression of CXCR3 targeting chemokines CXCL10, CXCL9, and CXCL11 in different types of skin inflammation , 2001, The Journal of pathology.
[31] M. Graeber,et al. Neuronal MCP-1 Expression in Response to Remote Nerve Injury , 2001, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[32] J. Farber. Mig and IP‐10: CXC chemokines that target lymphocytes , 1997, Journal of leukocyte biology.
[33] C. Bocti,et al. Protective Effect of Amyloid-β Peptides Against Herpes Simplex Virus-1 Infection in a Neuronal Cell Culture Model. , 2016, Journal of Alzheimer's disease : JAD.
[34] Martin Ingelsson,et al. The Alzheimer's Disease-Associated Amyloid \(\beta\)-Protein Is an Antimicrobial Peptide , 2010 .
[35] X. Bi,et al. Cholesterol in Niemann-Pick Type C disease. , 2010, Sub-cellular biochemistry.
[36] R. Ransohoff,et al. Chemokine receptor CXCR3: an unexpected enigma. , 2005, Current topics in developmental biology.
[37] K. Schroder,et al. Interferon-gamma: an overview of signals, mechanisms and functions. , 2004, Journal of leukocyte biology.
[38] M. Kawaguchi,et al. Differential regulation of eotaxin expression by IFN-gamma in airway epithelial cells. , 2003, The Journal of allergy and clinical immunology.