Crosstalk between glucocorticoid receptor and nutritional sensor mTOR in skeletal muscle.

[1]  D. Sabatini,et al.  Regulation of the mTOR complex 1 pathway by nutrients, growth factors, and stress. , 2010, Molecular cell.

[2]  E. Olson,et al.  Myogenin and Class II HDACs Control Neurogenic Muscle Atrophy by Inducing E3 Ubiquitin Ligases , 2010, Cell.

[3]  D. Hailey,et al.  Autophagy termination and lysosome reformation regulated by mTOR , 2010, Nature.

[4]  D. Sabatini,et al.  Ragulator-Rag Complex Targets mTORC1 to the Lysosomal Surface and Is Necessary for Its Activation by Amino Acids , 2010, Cell.

[5]  A. Poupon,et al.  The Translation Regulatory Subunit eIF3f Controls the Kinase-Dependent mTOR Signaling Required for Muscle Differentiation and Hypertrophy in Mouse , 2010, PloS one.

[6]  L. Schaeffer,et al.  Muscle inactivation of mTOR causes metabolic and dystrophin defects leading to severe myopathy , 2009, The Journal of cell biology.

[7]  W. Mitch,et al.  Endogenous glucocorticoids and impaired insulin signaling are both required to stimulate muscle wasting under pathophysiological conditions in mice. , 2009, The Journal of clinical investigation.

[8]  S. Miyano,et al.  Ligand-based gene expression profiling reveals novel roles of glucocorticoid receptor in cardiac metabolism. , 2009, American journal of physiology. Endocrinology and metabolism.

[9]  Martin J Firth,et al.  Glucocorticoid resistance in T-lineage acute lymphoblastic leukaemia is associated with a proliferative metabolism , 2009, British Journal of Cancer.

[10]  K. Yamamoto,et al.  DNA Binding Site Sequence Directs Glucocorticoid Receptor Structure and Activity , 2009, Science.

[11]  Svati H Shah,et al.  A branched-chain amino acid-related metabolic signature that differentiates obese and lean humans and contributes to insulin resistance. , 2009, Cell metabolism.

[12]  E. Casanova,et al.  Skeletal muscle-specific ablation of raptor, but not of rictor, causes metabolic changes and results in muscle dystrophy. , 2008, Cell metabolism.

[13]  S. Bodine,et al.  The glucocorticoid receptor and FOXO1 synergistically activate the skeletal muscle atrophy-associated MuRF1 gene. , 2008, American journal of physiology. Endocrinology and metabolism.

[14]  Zhigui Ma,et al.  Rapamycin reverses NPM-ALK-induced glucocorticoid resistance in lymphoid tumor cells by inhibiting mTOR signaling pathway, enhancing G1 cell cycle arrest and apoptosis , 2008, Leukemia.

[15]  Marco Sandri,et al.  Signaling in muscle atrophy and hypertrophy. , 2008, Physiology.

[16]  J. Thissen,et al.  Mechanisms of glucocorticoid-induced myopathy. , 2008, The Journal of endocrinology.

[17]  Daniel J. Klionsky,et al.  Autophagy fights disease through cellular self-digestion , 2008, Nature.

[18]  N. LeBrasseur,et al.  Fast/Glycolytic muscle fiber growth reduces fat mass and improves metabolic parameters in obese mice. , 2008, Cell metabolism.

[19]  D. Sgroi,et al.  Hypoxia regulates TSC1/2-mTOR signaling and tumor suppression through REDD1-mediated 14-3-3 shuttling. , 2008, Genes & development.

[20]  A. Goldberg,et al.  FoxO3 coordinately activates protein degradation by the autophagic/lysosomal and proteasomal pathways in atrophying muscle cells. , 2007, Cell metabolism.

[21]  A. Goldberg,et al.  FoxO3 controls autophagy in skeletal muscle in vivo. , 2007, Cell metabolism.

[22]  V. Mootha,et al.  mTOR controls mitochondrial oxidative function through a YY1–PGC-1α transcriptional complex , 2007, Nature.

[23]  Pengxiang She,et al.  Disruption of BCATm in mice leads to increased energy expenditure associated with the activation of a futile protein turnover cycle. , 2007, Cell metabolism.

[24]  Y. Itoyama,et al.  NO production results in suspension-induced muscle atrophy through dislocation of neuronal NOS. , 2007, The Journal of clinical investigation.

[25]  Wei Wei,et al.  Role of glucocorticoids in the molecular regulation of muscle wasting , 2007, Critical care medicine.

[26]  Saptarsi M. Haldar,et al.  Kruppel-like factor 15 is a regulator of cardiomyocyte hypertrophy , 2007, Proceedings of the National Academy of Sciences.

[27]  Saptarsi M. Haldar,et al.  Regulation of gluconeogenesis by Krüppel-like factor 15. , 2007, Cell metabolism.

[28]  L. Ellisen,et al.  Dexamethasone Represses Signaling through the Mammalian Target of Rapamycin in Muscle Cells by Enhancing Expression of REDD1* , 2006, Journal of Biological Chemistry.

[29]  Rosa Bernardi,et al.  PML inhibits HIF-1α translation and neoangiogenesis through repression of mTOR , 2006, Nature.

[30]  G. Thomas,et al.  Nutrient overload, insulin resistance, and ribosomal protein S6 kinase 1, S6K1. , 2006, Cell metabolism.

[31]  Huajun Yan,et al.  Mechanism by which mammalian target of rapamycin inhibitors sensitize multiple myeloma cells to dexamethasone-induced apoptosis. , 2006, Cancer research.

[32]  R. Evans,et al.  The Nuclear Receptor Superfamily: a Rosetta Stone for Physiology , 1985 .

[33]  Eric P Hoffman,et al.  Balancing muscle hypertrophy and atrophy , 2004, Nature Medicine.

[34]  G. Yancopoulos,et al.  The IGF-1/PI3K/Akt pathway prevents expression of muscle atrophy-induced ubiquitin ligases by inhibiting FOXO transcription factors. , 2004, Molecular cell.

[35]  Marco Sandri,et al.  Foxo Transcription Factors Induce the Atrophy-Related Ubiquitin Ligase Atrogin-1 and Cause Skeletal Muscle Atrophy , 2004, Cell.

[36]  D. Glass.  Signalling pathways that mediate skeletal muscle hypertrophy and atrophy , 2003, Nature Cell Biology.

[37]  E. R. Sánchez,et al.  Potentiation of glucocorticoid receptor-mediated gene expression by the immunophilin ligands FK506 and rapamycin. , 1993, The Journal of biological chemistry.

[38]  M. Rennie,et al.  Effects of corticosteroid on the transport and metabolism of glutamine in rat skeletal muscle. , 1991, Biochimica et biophysica acta.

[39]  M. Buse,et al.  Effects of diabetes and starvation on skeletal muscle branched-chain alpha-keto acid dehydrogenase activity. , 1988, The American journal of physiology.

[40]  R. PérezDíaz,et al.  Proteins and amino acids in surgery , 1949 .

[41]  D. Attaix,et al.  Myostatin Gene Deletion Prevents Glucocorticoid- Induced Muscle Atrophy , 2022 .

[42]  Rosa Bernardi,et al.  PML inhibits HIF-1alpha translation and neoangiogenesis through repression of mTOR. , 2006, Nature.

[43]  S. Bhasin,et al.  Characterization of the 5' upstream regulatory region of the human myostatin gene: regulation of myostatin gene transcription by dexamethasone in vitro , 2001 .

[44]  A. Wagenmakers.  Protein and amino acid metabolism in human muscle. , 1998, Advances in experimental medicine and biology.

[45]  C. W. Wrigley,et al.  Proteins and amino acids , 1988 .

[46]  N. Holbrook,et al.  Physiological functions of glucocorticoids in stress and their relation to pharmacological actions. , 1984, Endocrine reviews.