Crosstalk between glucocorticoid receptor and nutritional sensor mTOR in skeletal muscle.
暂无分享,去创建一个
Keiichi Fukuda | Makoto Suematsu | Hirotoshi Tanaka | Naoki Ito | M. Suematsu | C. Morimoto | K. Fukuda | J. Nakae | S. Takeda | K. Takehana | M. Sano | Hirotoshi Tanaka | N. Shimizu | N. Yoshikawa | Yuko Suzuki | Kenji Takehana | Motoaki Sano | Yuko Suzuki | Takako Maruyama | Jun Nakae | Noriaki Shimizu | Noritada Yoshikawa | Sin-ichi Takeda | Yusuke Tagata | Shinobu Nishitani | Chikao Morimoto | N. Ito | Shinobu Nishitani | T. Maruyama | Y. Tagata | Naoki Ito
[1] D. Sabatini,et al. Regulation of the mTOR complex 1 pathway by nutrients, growth factors, and stress. , 2010, Molecular cell.
[2] E. Olson,et al. Myogenin and Class II HDACs Control Neurogenic Muscle Atrophy by Inducing E3 Ubiquitin Ligases , 2010, Cell.
[3] D. Hailey,et al. Autophagy termination and lysosome reformation regulated by mTOR , 2010, Nature.
[4] D. Sabatini,et al. Ragulator-Rag Complex Targets mTORC1 to the Lysosomal Surface and Is Necessary for Its Activation by Amino Acids , 2010, Cell.
[5] A. Poupon,et al. The Translation Regulatory Subunit eIF3f Controls the Kinase-Dependent mTOR Signaling Required for Muscle Differentiation and Hypertrophy in Mouse , 2010, PloS one.
[6] L. Schaeffer,et al. Muscle inactivation of mTOR causes metabolic and dystrophin defects leading to severe myopathy , 2009, The Journal of cell biology.
[7] W. Mitch,et al. Endogenous glucocorticoids and impaired insulin signaling are both required to stimulate muscle wasting under pathophysiological conditions in mice. , 2009, The Journal of clinical investigation.
[8] S. Miyano,et al. Ligand-based gene expression profiling reveals novel roles of glucocorticoid receptor in cardiac metabolism. , 2009, American journal of physiology. Endocrinology and metabolism.
[9] Martin J Firth,et al. Glucocorticoid resistance in T-lineage acute lymphoblastic leukaemia is associated with a proliferative metabolism , 2009, British Journal of Cancer.
[10] K. Yamamoto,et al. DNA Binding Site Sequence Directs Glucocorticoid Receptor Structure and Activity , 2009, Science.
[11] Svati H Shah,et al. A branched-chain amino acid-related metabolic signature that differentiates obese and lean humans and contributes to insulin resistance. , 2009, Cell metabolism.
[12] E. Casanova,et al. Skeletal muscle-specific ablation of raptor, but not of rictor, causes metabolic changes and results in muscle dystrophy. , 2008, Cell metabolism.
[13] S. Bodine,et al. The glucocorticoid receptor and FOXO1 synergistically activate the skeletal muscle atrophy-associated MuRF1 gene. , 2008, American journal of physiology. Endocrinology and metabolism.
[14] Zhigui Ma,et al. Rapamycin reverses NPM-ALK-induced glucocorticoid resistance in lymphoid tumor cells by inhibiting mTOR signaling pathway, enhancing G1 cell cycle arrest and apoptosis , 2008, Leukemia.
[15] Marco Sandri,et al. Signaling in muscle atrophy and hypertrophy. , 2008, Physiology.
[16] J. Thissen,et al. Mechanisms of glucocorticoid-induced myopathy. , 2008, The Journal of endocrinology.
[17] Daniel J. Klionsky,et al. Autophagy fights disease through cellular self-digestion , 2008, Nature.
[18] N. LeBrasseur,et al. Fast/Glycolytic muscle fiber growth reduces fat mass and improves metabolic parameters in obese mice. , 2008, Cell metabolism.
[19] D. Sgroi,et al. Hypoxia regulates TSC1/2-mTOR signaling and tumor suppression through REDD1-mediated 14-3-3 shuttling. , 2008, Genes & development.
[20] A. Goldberg,et al. FoxO3 coordinately activates protein degradation by the autophagic/lysosomal and proteasomal pathways in atrophying muscle cells. , 2007, Cell metabolism.
[21] A. Goldberg,et al. FoxO3 controls autophagy in skeletal muscle in vivo. , 2007, Cell metabolism.
[22] V. Mootha,et al. mTOR controls mitochondrial oxidative function through a YY1–PGC-1α transcriptional complex , 2007, Nature.
[23] Pengxiang She,et al. Disruption of BCATm in mice leads to increased energy expenditure associated with the activation of a futile protein turnover cycle. , 2007, Cell metabolism.
[24] Y. Itoyama,et al. NO production results in suspension-induced muscle atrophy through dislocation of neuronal NOS. , 2007, The Journal of clinical investigation.
[25] Wei Wei,et al. Role of glucocorticoids in the molecular regulation of muscle wasting , 2007, Critical care medicine.
[26] Saptarsi M. Haldar,et al. Kruppel-like factor 15 is a regulator of cardiomyocyte hypertrophy , 2007, Proceedings of the National Academy of Sciences.
[27] Saptarsi M. Haldar,et al. Regulation of gluconeogenesis by Krüppel-like factor 15. , 2007, Cell metabolism.
[28] L. Ellisen,et al. Dexamethasone Represses Signaling through the Mammalian Target of Rapamycin in Muscle Cells by Enhancing Expression of REDD1* , 2006, Journal of Biological Chemistry.
[29] Rosa Bernardi,et al. PML inhibits HIF-1α translation and neoangiogenesis through repression of mTOR , 2006, Nature.
[30] G. Thomas,et al. Nutrient overload, insulin resistance, and ribosomal protein S6 kinase 1, S6K1. , 2006, Cell metabolism.
[31] Huajun Yan,et al. Mechanism by which mammalian target of rapamycin inhibitors sensitize multiple myeloma cells to dexamethasone-induced apoptosis. , 2006, Cancer research.
[32] R. Evans,et al. The Nuclear Receptor Superfamily: a Rosetta Stone for Physiology , 1985 .
[33] Eric P Hoffman,et al. Balancing muscle hypertrophy and atrophy , 2004, Nature Medicine.
[34] G. Yancopoulos,et al. The IGF-1/PI3K/Akt pathway prevents expression of muscle atrophy-induced ubiquitin ligases by inhibiting FOXO transcription factors. , 2004, Molecular cell.
[35] Marco Sandri,et al. Foxo Transcription Factors Induce the Atrophy-Related Ubiquitin Ligase Atrogin-1 and Cause Skeletal Muscle Atrophy , 2004, Cell.
[36] D. Glass. Signalling pathways that mediate skeletal muscle hypertrophy and atrophy , 2003, Nature Cell Biology.
[37] E. R. Sánchez,et al. Potentiation of glucocorticoid receptor-mediated gene expression by the immunophilin ligands FK506 and rapamycin. , 1993, The Journal of biological chemistry.
[38] M. Rennie,et al. Effects of corticosteroid on the transport and metabolism of glutamine in rat skeletal muscle. , 1991, Biochimica et biophysica acta.
[39] M. Buse,et al. Effects of diabetes and starvation on skeletal muscle branched-chain alpha-keto acid dehydrogenase activity. , 1988, The American journal of physiology.
[40] R. PérezDíaz,et al. Proteins and amino acids in surgery , 1949 .
[41] D. Attaix,et al. Myostatin Gene Deletion Prevents Glucocorticoid- Induced Muscle Atrophy , 2022 .
[42] Rosa Bernardi,et al. PML inhibits HIF-1alpha translation and neoangiogenesis through repression of mTOR. , 2006, Nature.
[43] S. Bhasin,et al. Characterization of the 5' upstream regulatory region of the human myostatin gene: regulation of myostatin gene transcription by dexamethasone in vitro , 2001 .
[44] A. Wagenmakers. Protein and amino acid metabolism in human muscle. , 1998, Advances in experimental medicine and biology.
[45] C. W. Wrigley,et al. Proteins and amino acids , 1988 .
[46] N. Holbrook,et al. Physiological functions of glucocorticoids in stress and their relation to pharmacological actions. , 1984, Endocrine reviews.