Gelsolin is depleted in post-shock mesenteric lymph.

[1]  E. Moore,et al.  Normal mesenteric lymph blunts the pulmonary inflammatory response to endotoxin. , 2006, The Journal of surgical research.

[2]  E. Moore,et al.  BIOACTIVITY OF POSTSHOCK MESENTERIC LYMPH DEPENDS ON THE DEPTH AND DURATION OF HEMORRHAGIC SHOCK , 2006, Shock.

[3]  T. Stossel,et al.  Relationship of Plasma Gelsolin Levels to Outcomes in Critically Ill Surgical Patients , 2006, Annals of surgery.

[4]  P. Janmey,et al.  Inactivation of endotoxin by human plasma gelsolin. , 2005, Biochemistry.

[5]  E. Deitch,et al.  Hemorrhagic shock induces endothelial cell apoptosis, which is mediated by factors contained in mesenteric lymph , 2004, Critical care medicine.

[6]  E. Moore,et al.  Systemic neutrophil priming by lipid mediators in post-shock mesenteric lymph exists across species. , 2004, The Journal of trauma.

[7]  E. Deitch,et al.  A study of the biologic activity of trauma-hemorrhagic shock mesenteric lymph over time and the relative role of cytokines. , 2004, Surgery.

[8]  E. Deitch,et al.  Trauma-Hemorrhagic Shock Mesenteric Lymph Induces Endothelial Apoptosis That Involves Both Caspase-Dependent and Caspase-Independent Mechanisms , 2004, Annals of surgery.

[9]  I. Shkrob,et al.  Frequency-domain “single-shot” ultrafast transient absorption spectroscopy using chirped laser pulses , 2004 .

[10]  E. Deitch,et al.  Factors in intestinal lymph after shock increase neutrophil adhesion molecule expression and pulmonary leukosequestration. , 2003, The Journal of trauma.

[11]  E. Moore,et al.  Post-hemorrhagic shock mesenteric lymph activates human pulmonary microvascular endothelium for in vitro neutrophil-mediated injury: the role of intercellular adhesion molecule-1. , 2003, The Journal of trauma.

[12]  John M. Adams,et al.  Shock mesenteric lymph-induced rat polymorphonuclear neutrophil activation and endothelial cell injury is mediated by aqueous factors. , 2002, The Journal of trauma.

[13]  E. Moore,et al.  Mesenteric lymph is responsible for post-hemorrhagic shock systemic neutrophil priming. , 2001, The Journal of trauma.

[14]  E. Moore,et al.  Post-hemorrhagic shock mesenteric lymph lipids prime neutrophils for enhanced cytotoxicity via phospholipase A2. , 2001, Shock.

[15]  E. Goetzl,et al.  The lysophospholipids sphingosine-1-phosphate and lysophosphatidic acid enhance survival during hypoxia in neonatal rat cardiac myocytes. , 2001, Journal of molecular and cellular cardiology.

[16]  E. Moore,et al.  Phospholipase A(2)--derived neutral lipids from posthemorrhagic shock mesenteric lymph prime the neutrophil oxidative burst. , 2001, Surgery.

[17]  C. Turck,et al.  Gelsolin Binding and Cellular Presentation of Lysophosphatidic Acid* , 2000, The Journal of Biological Chemistry.

[18]  Helen L. Yin,et al.  Gelsolin, a Multifunctional Actin Regulatory Protein* , 1999, The Journal of Biological Chemistry.

[19]  K. Mounzer,et al.  Relationship of admission plasma gelsolin levels to clinical outcomes in patients after major trauma. , 1999, American journal of respiratory and critical care medicine.

[20]  D. Y. Tamura,et al.  Posthemorrhagic shock mesenteric lymph primes circulating neutrophils and provokes lung injury. , 1999, The Journal of surgical research.

[21]  E. Deitch,et al.  POST‐HEMORRHAGIC SHOCK MESENTERIC LYMPH IS CYTOTOXIC TO ENDOTHELIAL CELLS AND ACTIVATES NEUTROPHILS , 1998, Shock.

[22]  L. Magnotti,et al.  Gut-derived mesenteric lymph but not portal blood increases endothelial cell permeability and promotes lung injury after hemorrhagic shock. , 1998, Annals of surgery.

[23]  Simon C Watkins,et al.  PURE ENDOTOXIN DOES NOT PASS ACROSS THE INTESTINAL EPITHELIUM IN VITRO , 1998, Shock.

[24]  W. M. Lee,et al.  Decreased plasma gelsolin concentrations in acute liver failure, myocardial infarction, septic shock, and myonecrosis. , 1997, Critical care medicine.

[25]  M. Way,et al.  Evidence for functional homology in the F-actin binding domains of gelsolin and alpha-actinin: implications for the requirements of severing and capping , 1992, The Journal of cell biology.

[26]  E. Deitch,et al.  Multiple organ failure. Pathophysiology and potential future therapy. , 1992, Annals of surgery.

[27]  D. Kwiatkowski,et al.  Muscle is the major source of plasma gelsolin. , 1988, The Journal of biological chemistry.

[28]  D. B. Smith,et al.  Role of plasma gelsolin and the vitamin D-binding protein in clearing actin from the circulation. , 1986, The Journal of clinical investigation.

[29]  J. Tou,et al.  Lysophosphatidic acid increases phosphatidic acid formation, phospholipase D activity and degranulation by human neutrophils. , 2005, Cellular signalling.

[30]  William M. Lee,et al.  Recombinant plasma gelsolin infusion attenuates burn-induced pulmonary microvascular dysfunction. , 2004, Journal of applied physiology.

[31]  E. Deitch,et al.  A time course study of the protective effect of mesenteric lymph duct ligation on hemorrhagic shock-induced pulmonary injury and the toxic effects of lymph from shocked rats on endothelial cell monolayer permeability. , 2001, Surgery.