Direct evidence for a role of the mast cell in the nasal response to aspirin in aspirin-sensitive asthma.

[1]  J. Oates,et al.  Eicosanoids in bronchoalveolar lavage fluid of aspirin-intolerant patients with asthma after aspirin challenge. , 1994, American journal of respiratory and critical care medicine.

[2]  E. Israel,et al.  The pivotal role of 5-lipoxygenase products in the reaction of aspirin-sensitive asthmatics to aspirin. , 1993, The American review of respiratory disease.

[3]  S. Dahlén,et al.  The leukotriene-receptor antagonist MK-0679 blocks airway obstruction induced by inhaled lysine-aspirin in aspirin-sensitive asthmatics. , 1993, The European respiratory journal.

[4]  K. Sladek,et al.  Cysteinyl leukotrienes overproduction and mast cell activation in aspirin-provoked bronchospasm in asthma. , 1993, The European respiratory journal.

[5]  M. Kowalski,et al.  Nasal secretions in response to acetylsalicylic acid. , 1993, The Journal of allergy and clinical immunology.

[6]  A. Ford-hutchinson,et al.  Urinary leukotriene E4 after lysine-aspirin inhalation in asthmatic subjects. , 1992, The American review of respiratory disease.

[7]  S. Dahlén,et al.  Urinary excretion of leukotriene E4 and 11-dehydro-thromboxane B2 in response to bronchial provocations with allergen, aspirin, leukotriene D4, and histamine in asthmatics. , 1992, The American review of respiratory disease.

[8]  J. Shelhamer,et al.  Human neutrophil-derived histamine-releasing activity (HRA-N) causes the release of serotonin but not arachidonic acid metabolites from rat basophilic leukemia cells. , 1992, The Journal of allergy and clinical immunology.

[9]  L. Schwartz Cellular inflammation in asthma: neutral proteases of mast cells. , 1992, The American review of respiratory disease.

[10]  J. Montserrat,et al.  Release of peptide leukotriene into nasal secretions after local instillation of aspirin in aspirin-sensitive asthmatic patients. , 1992, The American review of respiratory disease.

[11]  G. FitzGerald,et al.  Increased excretion of leukotriene E4 during aspirin-induced asthma. , 1992, The Journal of laboratory and clinical medicine.

[12]  J. Denburg,et al.  Histochemical and immunohistochemical characteristics of mast cells in nasal polyps. , 1992, American journal of respiratory cell and molecular biology.

[13]  D. Stevenson,et al.  Tryptase and histamine release during aspirin-induced respiratory reactions. , 1991, The Journal of allergy and clinical immunology.

[14]  T. Lee,et al.  The potent and selective sulfidopeptide leukotriene antagonist, SK&F 104353, inhibits aspirin-induced asthma. , 1991, The American review of respiratory disease.

[15]  A. Ford-hutchinson,et al.  Urinary leukotriene E4 concentrations increase after aspirin challenge in aspirin-sensitive asthmatic subjects. , 1991, The American review of respiratory disease.

[16]  R. D. Dyer,et al.  5-lipoxygenase inhibitory activity of zileuton. , 1991, The Journal of pharmacology and experimental therapeutics.

[17]  L. Dubé,et al.  Pharmacokinetics, safety, and ability to diminish leukotriene synthesis by zileuton, an inhibitor of 5-lipoxygenase. , 1991, Agents and actions. Supplements.

[18]  H. Knapp,et al.  Reduced allergen-induced nasal congestion and leukotriene synthesis with an orally active 5-lipoxygenase inhibitor. , 1990, The New England journal of medicine.

[19]  A. Kagey‐Sobotka,et al.  Disassociation of the release of histamine and arachidonic acid metabolites from osmotically activated basophils and human lung mast cells. , 1990, The American review of respiratory disease.

[20]  T. Yamashita,et al.  Etiology of nasal polyps associated with aspirin-sensitive asthma. , 1989, Rhinology. Supplement.

[21]  L. Schwartz,et al.  Tryptase levels in nasal-lavage fluid as an indicator of the immediate allergic response. , 1988, The Journal of allergy and clinical immunology.

[22]  D. Stevenson,et al.  Release of leukotrienes, prostaglandins, and histamine into nasal secretions of aspirin-sensitive asthmatics during reaction to aspirin. , 1988, The American review of respiratory disease.

[23]  K. Hozawa,et al.  Mast cell degranulation in nasal polyps. , 1986, Acta oto-laryngologica. Supplementum.

[24]  A. Togias,et al.  Inflammatory mediators in late antigen-induced rhinitis. , 1985, The New England journal of medicine.

[25]  L. Schwartz Monoclonal antibodies against human mast cell tryptase demonstrate shared antigenic sites on subunits of tryptase and selective localization of the enzyme to mast cells. , 1985, Journal of immunology.

[26]  R. Naclerio,et al.  Peptide leukotriene release after antigen challenge in patients sensitive to ragweed. , 1984, The New England journal of medicine.

[27]  E. Corey,et al.  Leukotriene B4 action on endothelium mediates augmented neutrophil/endothelial adhesion. , 1984, Proceedings of the National Academy of Sciences of the United States of America.

[28]  J. Nowak Eicosanoids and the lungs. , 1984, Annals of clinical research.

[29]  J. Delaney The effect of ketotifen on aspirin‐induced asthmatic reactions , 1983, Clinical allergy.

[30]  J. Shelhamer,et al.  Slow-reacting substances, leukotrienes C4 and D4, increase the release of mucus from human airways in vitro. , 2015, The American review of respiratory disease.

[31]  S. Dahlén,et al.  Leukotrienes promote plasma leakage and leukocyte adhesion in postcapillary venules: in vivo effects with relevance to the acute inflammatory response. , 1981, Proceedings of the National Academy of Sciences of the United States of America.

[32]  A. Szczeklik,et al.  Inhibition by Ketotifen of Idiosyncratic Reactions to Aspirin , 1980, Allergy.

[33]  E. Corey,et al.  Comparative airway and vascular activities of leukotrienes C-1 and D in vivo and in vitro. , 1980, Proceedings of the National Academy of Sciences of the United States of America.

[34]  A. Szczeklik,et al.  Inhibition of idiosyncratic reactions to aspirin in asthmatic patients by clemastine. , 1979, Thorax.

[35]  I. Olsson,et al.  Arginine-rich cationic proteins of human eosinophil granules: comparison of the constituents of eosinophilic and neutrophilic leukocytes. , 1977, Laboratory investigation; a journal of technical methods and pathology.

[36]  E. Tan,et al.  Oral aspirin challenges in asthmatic patients: a study of plasma histamine , 1976, Clinical allergy.

[37]  A. Szczeklik,et al.  Relationship of inhibition of prostaglandin biosynthesis by analgesics to asthma attacks in aspirin-sensitive patients. , 1975, British medical journal.