Cellular and molecular mechanisms of stress-induced premature senescence (SIPS) of human diploid fibroblasts and melanocytes
暂无分享,去创建一个
[1] A. Balin,et al. Effect of oxygen on the growth of human epidermal keratinocytes. , 1986, The Journal of investigative dermatology.
[2] H. Lin,et al. The 170-kDa glucose-regulated stress protein is an endoplasmic reticulum protein that binds immunoglobulin. , 1993, Molecular biology of the cell.
[3] A. Balin,et al. Effects of oxygen tension on the growth and pigmentation of normal human melanocytes. , 1990, The Journal of investigative dermatology.
[4] G. Hannon,et al. Involvement of the cyclin-dependent kinase inhibitor p16 (INK4a) in replicative senescence of normal human fibroblasts. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[5] Y. Ben-Neriah,et al. The ubiquitous glucose transporter GLUT-1 belongs to the glucose-regulated protein family of stress-inducible proteins. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[6] E. Medrano,et al. Melanin Accumulation Accelerates Melanocyte Senescence by a Mechanism Involving p16INK4a/CDK4/pRB and E2F1 , 2000, Annals of the New York Academy of Sciences.
[7] S. Benchimol,et al. From telomere loss to p53 induction and activation of a DNA-damage pathway at senescence: The telomere loss/DNA damage model of cell aging , 1996, Experimental Gerontology.
[8] D. Woods,et al. Senescence of human fibroblasts induced by oncogenic Raf. , 1998, Genes & development.
[9] J. Remacle,et al. Comparative study of oxygen toxicity in human fibroblasts and endothelial cells , 1990, Journal of cellular physiology.
[10] J. R. Smith,et al. Intraclonal variation in proliferative potential of human diploid fibroblasts: stochastic mechanism for cellular aging. , 1980, Science.
[11] R. Reddel,et al. The telomere lengthening mechanism in telomerase-negative immortal human cells does not involve the telomerase RNA subunit. , 1997, Human molecular genetics.
[12] B. Ames,et al. Oxidative DNA damage and senescence of human diploid fibroblast cells. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[13] S. Baldwin,et al. Regulation of glucose uptake by stressed cells , 1991, Journal of cellular physiology.
[14] J. Remacle,et al. Aging as a multi-step process characterized by a lowering of entropy production leading the cell to a sequence of defined stages. II. Testing some predictions on aging human fibroblasts in culture , 1992, Mechanisms of Ageing and Development.
[15] J. Remacle,et al. Importance of various antioxidant enzymes for cell stability. Confrontation between theoretical and experimental data. , 1992, The Biochemical journal.
[16] A. Koong,et al. Increased cytotoxicity of chronic hypoxic cells by molecular inhibition of GRP78 induction. , 1994, International journal of radiation oncology, biology, physics.
[17] P. Palozza,et al. Different mechanisms of tert-butyl hydroperoxide-induced lethal injury in normal and tumor thymocytes. , 1994, Archives of biochemistry and biophysics.
[18] I. Hickson,et al. Defending genome integrity during DNA replication: a proposed role for RecQ family helicases , 1999, BioEssays : news and reviews in molecular, cellular and developmental biology.
[19] Edward L. Schneider,et al. Handbook of the Biology of Aging , 1990 .
[20] V A Zakian,et al. Telomeres: Beginning to Understand the End , 1995, Science.
[21] J. Remacle,et al. Aging as a multi-step process characterized by a lowering of entropy production leading the cell to a sequence of defined stages , 1991, Mechanisms of Ageing and Development.
[22] A. Goldberg,et al. A set of endoplasmic reticulum proteins possessing properties of molecular chaperones includes Ca(2+)-binding proteins and members of the thioredoxin superfamily. , 1994, The Journal of biological chemistry.
[23] T. Davison,et al. ATM‐dependent telomere loss in aging human diploid fibroblasts and DNA damage lead to the post‐translational activation of p53 protein involving poly(ADP‐ribose) polymerase , 1997, The EMBO journal.
[24] A. Lloyd,et al. Different kinetics of senescence in human fibroblasts and peritoneal mesothelial cells. , 1997, Experimental cell research.
[25] C B Harley,et al. Telomere loss: mitotic clock or genetic time bomb? , 1991, Mutation research.
[26] J. Remacle,et al. Low levels of reactive oxygen species as modulators of cell function. , 1995, Mutation research.
[27] H. Osiewacz,et al. Molecular and cellular gerontology Serpiano, Switzerland, September 18–22, 1999 , 2000, The EMBO journal.
[28] D. Barker,et al. Comparison of the responses of human melanocytes with different melanin contents to ultraviolet B irradiation. , 1995, Cancer research.
[29] T. Zglinicki,et al. Accumulation of single-strand breaks is the major cause of telomere shortening in human fibroblasts. , 2000, Free radical biology & medicine.
[30] M. Matsuo,et al. Shortening of the in vitro lifespan of human diploid fibroblasts exposed to hyperbaric oxygen , 1983, Experimental Gerontology.
[31] J. Remacle,et al. Induction of replicative senescence biomarkers by sublethal oxidative stresses in normal human fibroblast. , 2000, Free radical biology & medicine.
[32] J. R. Smith,et al. Identification of a Gene That Reverses the Immortal Phenotype of a Subset of Cells and Is a Member of a Novel Family of Transcription Factor-Like Genes , 1999, Molecular and Cellular Biology.
[33] G. Saretzki,et al. Telomere Length As a Marker of Oxidative Stress in Primary Human Fibroblast Cultures , 2000, Annals of the New York Academy of Sciences.
[34] B. Ames,et al. Senescence-like growth arrest induced by hydrogen peroxide in human diploid fibroblast F65 cells. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[35] J. Remacle,et al. Effects of modulations of the energetic metabolism on the mortality of cultured cells. , 1994, Biochimica et biophysica acta.
[36] R. G. Allen,et al. Relationship between donor age and the replicative lifespan of human cells in culture: a reevaluation. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[37] C. Schmoor,et al. Accumulation of short telomeres in human fibroblasts prior to replicative senescence. , 2000, Experimental cell research.
[38] Lucio Comai,et al. Functional Interaction between Ku and the Werner Syndrome Protein in DNA End Processing* , 2000, The Journal of Biological Chemistry.
[39] J. Remacle,et al. Stress‐Induced Premature Senescence: Essence of Life, Evolution, Stress, and Aging , 2000, Annals of the New York Academy of Sciences.
[40] J. Langmore,et al. Long G Tails at Both Ends of Human Chromosomes Suggest a C Strand Degradation Mechanism for Telomere Shortening , 1997, Cell.
[41] B. Howard,et al. Human fibroblast commitment to a senescence-like state in response to histone deacetylase inhibitors is cell cycle dependent , 1996, Molecular and cellular biology.
[42] E. Gilson,et al. Interaction between Set1p and checkpoint protein Mec3p in DNA repair and telomere functions , 1999, Nature Genetics.
[43] C. D. Allis,et al. Linking histone acetylation to transcriptional regulation , 1998, Cellular and Molecular Life Sciences CMLS.
[44] T. Kirkwood. Molecular gerontology - Bridging the simple and the complex , 1999 .
[45] J. Sedivy,et al. Expression of catalytically active telomerase does not prevent premature senescence caused by overexpression of oncogenic Ha-Ras in normal human fibroblasts. , 1999, Cancer research.
[46] H. Rodemann. Differential degradation of intracellular proteins in human skin fibroblasts of mitotic and mitomycin-C (MMC)-induced postmitotic differentiation states in vitro. , 1989, Differentiation; research in biological diversity.
[47] R. G. Allen,et al. Is beta-galactosidase staining a marker of senescence in vitro and in vivo? , 2000, Experimental cell research.
[48] T. Hupp,et al. Posttranslational Modifications of p53 in Replicative Senescence Overlapping but Distinct from Those Induced by DNA Damage , 2000, Molecular and Cellular Biology.
[49] G. Calviello,et al. tert-Butyl Hydroperoxide-Induced [Ca2+]i in Thymus and Thymoma Cells , 1993 .
[50] L. Loeb,et al. Functional interaction between the Werner Syndrome protein and DNA polymerase delta. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[51] O. Toussaint,et al. Molecular gerontology : research status and strategies , 1996 .
[52] D. Levine,et al. Haemin enhancement of glucose transport in human lymphocytes: stimulation of protein tyrosine phosphatase and activation of p56lck tyrosine kinase. , 1993, The Biochemical journal.
[53] C. Harley,et al. Extension of life-span by introduction of telomerase into normal human cells. , 1998, Science.
[54] R. Rossi,et al. The time-course of mixed disulfide formation between GSH and proteins in rat blood after oxidative stress with tert-butyl hydroperoxide. , 1994, Biochimica et biophysica acta.
[55] G. Saretzki,et al. Mild hyperoxia shortens telomeres and inhibits proliferation of fibroblasts: a model for senescence? , 1995, Experimental cell research.
[56] D. Shelton,et al. Microarray analysis of replicative senescence , 1999, Current Biology.
[57] C Roskelley,et al. A biomarker that identifies senescent human cells in culture and in aging skin in vivo. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[58] S. M. Saati,et al. Cataloging altered gene expression in young and senescent cells using enhanced differential display. , 1995, Nucleic acids research.
[59] O. Toussaint. Molecular and cellular gerontology , 2000 .
[60] B. Ames,et al. Molecular analysis of H2O2-induced senescent-like growth arrest in normal human fibroblasts: p53 and Rb control G1 arrest but not cell replication. , 1998, The Biochemical journal.
[61] Cynthia Kenyon,et al. Signals from the reproductive system regulate the lifespan of C. elegans , 1999, Nature.
[62] A. Balin,et al. Oxygen modulates growth of human cells at physiologic partial pressures , 1984, The Journal of experimental medicine.
[63] C. Roberts,et al. Failure of senescent human fibroblasts to express the insulin-like growth factor-1 gene. , 1993, The Journal of biological chemistry.
[64] G. Enders,et al. p16INK4a can initiate an autonomous senescence program , 2000, Oncogene.
[65] E. Medrano,et al. Activation of a cAMP pathway and induction of melanogenesis correlate with association of p16(INK4) and p27(KIP1) to CDKs, loss of E2F-binding activity, and premature senescence of human melanocytes. , 1999, Experimental cell research.
[66] M. Gray,et al. The Werner syndrome protein is a DNA helicase , 1997, Nature Genetics.
[67] Qin M. Chen,et al. Human Diploid Fibroblasts Display a Decreased Level of c‐fos mRNA at 72 Hours after Exposure to Sublethal H2O2 Stress , 2000, Annals of the New York Academy of Sciences.
[68] J. Remacle,et al. Cellular aging and the importance of energetic factors , 1995, Experimental Gerontology.
[69] K. Nielsen,et al. Telomere erosion varies during in vitro aging of normal human fibroblasts from young and adult donors. , 2000, Cancer research.
[70] V. Ferrans,et al. Ras Proteins Induce Senescence by Altering the Intracellular Levels of Reactive Oxygen Species* , 1999, The Journal of Biological Chemistry.
[71] J. R. Smith,et al. Genetic analysis of indefinite division in human cells: identification of four complementation groups. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[72] James R. Smith,et al. Intramitotic variation in proliferative potential: Stochastic events in cellular aging , 1985, Mechanisms of Ageing and Development.
[73] R. Weindruch,et al. Oxidative Stress, Caloric Restriction, and Aging , 1996, Science.
[74] K. Haglid,et al. Induction of glucose regulated protein (grp78) and inducible heat shock protein (hsp70) mRNAs in rat brain after kainic acid seizures and focal ischemia , 1993, Neurochemistry International.
[75] T. Kiyono,et al. Both Rb/p16INK4a inactivation and telomerase activity are required to immortalize human epithelial cells , 1998, Nature.
[76] J. Remacle,et al. Appearance of biomarkers of in vitro ageing after successive stimulation of WI‐38 fibroblasts with IL‐1α and TNF‐α: senescence associated β‐galactosidase activity and morphotype transition , 2000 .
[77] K. Dittmann,et al. Selective enrichment and biochemical characterization of seven human skin fibroblasts cell types in vitro. , 1989, Experimental cell research.
[78] L. Hayflick,et al. The serial cultivation of human diploid cell strains. , 1961, Experimental cell research.
[79] B. Herman,et al. Contribution of the mitochondrial permeability transition to lethal injury after exposure of hepatocytes to t-butylhydroperoxide. , 1995, The Biochemical journal.
[80] E. Hiyama,et al. Age-dependent telomere shortening is slowed down by enrichment of intracellular vitamin C via suppression of oxidative stress. , 1998, Life sciences.
[81] S. Lowe,et al. Oncogenic ras Provokes Premature Cell Senescence Associated with Accumulation of p53 and p16INK4a , 1997, Cell.
[82] R. Faragher,et al. The expression of proliferation-dependent antigens during the lifespan of normal and progeroid human fibroblasts in culture. , 1994, Journal of cell science.
[83] C. Richter,et al. Quantification of wild-type mitochondrial DNA and its 4.8-kb deletion in rat organs. , 1997, Biochemical and biophysical research communications.
[84] J. Remacle,et al. Stress and Energy Metabolism in Age-Related Processes , 1996 .
[85] S. Goldstein,et al. Senescence of cultured human fibroblasts: mitotic versus metabolic time. , 1974, Experimental cell research.
[86] A. Koong,et al. The regulation of GRP78 and messenger RNA levels by hypoxia is modulated by protein kinase C activators and inhibitors. , 1994, Radiation research.
[87] T. Zglinicki,et al. Similar gene expression pattern in senescent and hyperoxic-treated fibroblasts. , 1998, The journals of gerontology. Series A, Biological sciences and medical sciences.
[88] S. Petersen,et al. Preferential accumulation of single-stranded regions in telomeres of human fibroblasts. , 1998, Experimental cell research.
[89] G. Dianov,et al. DNA repair and transcription in human premature aging disorders. , 1998, The journal of investigative dermatology. Symposium proceedings.
[90] D. Elder,et al. UVB induces atypical melanocytic lesions and melanoma in human skin. , 1998, The American journal of pathology.
[91] G. Stein. The molecular basis of cell cycle and growth control , 1999 .
[92] J. Griffith,et al. Mammalian Telomeres End in a Large Duplex Loop , 1999, Cell.
[93] A M Olovnikov,et al. A theory of marginotomy. The incomplete copying of template margin in enzymic synthesis of polynucleotides and biological significance of the phenomenon. , 1973, Journal of theoretical biology.
[94] J. Campisi. Cancer, aging and cellular senescence. , 2000, In vivo.
[95] H. C. Lee,et al. Human skin mitochondrial DNA deletions associated with light exposure. , 1994, Archives of biochemistry and biophysics.
[96] R. Wurm,et al. Telomere shortening triggers a p53-dependent cell cycle arrest via accumulation of G-rich single stranded DNA fragments , 1999, Oncogene.
[97] Qin M. Chen,et al. Replicative Senescence and Oxidant‐Induced Premature Senescence: Beyond the Control of Cell Cycle Checkpoints , 2000, Annals of the New York Academy of Sciences.
[98] Y. Eilam,et al. Slowing down aging of cultured embryonal chick chondrocytes by maintenance under lowered oxygen tension , 1988, Mechanisms of Ageing and Development.
[99] G. Saretzki,et al. Accelerated telomere shortening in fibroblasts after extended periods of confluency. , 1998, Free radical biology & medicine.
[100] K. Skorecki,et al. Position effect of human telomeric repeats on replication timing. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[101] R. Holliday. Toward a Biological Understanding of the Ageing Process , 2015, Perspectives in biology and medicine.