Palmitoylethanolamide exerts neuroprotective effects in mixed neuroglial cultures and organotypic hippocampal slices via peroxisome proliferator-activated receptor-α
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L. Steardo | G. Esposito | M. Valenza | C. Scuderi | M. Carratù | C. Stecca
[1] Yen-Ching Chen,et al. Palmitoylethanolamide exerts neuroprotective effects in mixed neuroglial cultures and organotypic hippocampal slices via peroxisome proliferator-activated receptor-α , 2012, Journal of Neuroinflammation.
[2] V. Marzo,et al. Palmitoylethanolamide counteracts reactive astrogliosis induced by β-amyloid peptide , 2011, Journal of cellular and molecular medicine.
[3] D. Selkoe. Alzheimer's disease. , 2011, Cold Spring Harbor perspectives in biology.
[4] M. O’Banion,et al. Neuroinflammatory processes in Alzheimer’s disease , 2010, Journal of Neural Transmission.
[5] H. Hansen. Palmitoylethanolamide and other anandamide congeners. Proposed role in the diseased brain , 2010, Experimental Neurology.
[6] Fred H. Gage,et al. Mechanisms Underlying Inflammation in Neurodegeneration , 2010, Cell.
[7] S. Cuzzocrea,et al. Selective N-acylethanolamine-hydrolyzing acid amidase inhibition reveals a key role for endogenous palmitoylethanolamide in inflammation , 2009, Proceedings of the National Academy of Sciences.
[8] V. Marzo,et al. Anti‐inflammatory Effect of Palmitoylethanolamide on Human Adipocytes , 2009, Obesity.
[9] K. Fuxe,et al. Neurotensin receptor involvement in the rise of extracellular glutamate levels and apoptotic nerve cell death in primary cortical cultures after oxygen and glucose deprivation. , 2008, Cerebral cortex.
[10] J. Bright,et al. PPAR Regulation of Inflammatory Signaling in CNS Diseases , 2008, PPAR research.
[11] E. Mazzon,et al. Effects of Palmitoylethanolamide on Signaling Pathways Implicated in the Development of Spinal Cord Injury , 2008, Journal of Pharmacology and Experimental Therapeutics.
[12] N. Stella,et al. Microglia produce and hydrolyze palmitoylethanolamide , 2008, Neuropharmacology.
[13] G. Landreth,et al. PPARs in the brain. , 2007, Biochimica et biophysica acta.
[14] G. Landreth. Therapeutic use of agonists of the nuclear receptor PPARgamma in Alzheimer's disease. , 2007, Current Alzheimer research.
[15] G. Landreth. Therapeutic use of agonists of the nuclear receptor PPARgamma in Alzheimer's disease. , 2007, Current Alzheimer research.
[16] Tony Wyss-Coray,et al. Inflammation in Alzheimer disease: driving force, bystander or beneficial response? , 2006, Nature Medicine.
[17] Roberto Russo,et al. The search for the palmitoylethanolamide receptor. , 2005, Life sciences.
[18] M. Block,et al. Microglia and inflammation-mediated neurodegeneration: Multiple triggers with a common mechanism , 2005, Progress in Neurobiology.
[19] D. Piomelli,et al. The Nuclear Receptor Peroxisome Proliferator-Activated Receptor-α Mediates the Anti-Inflammatory Actions of Palmitoylethanolamide , 2005, Molecular Pharmacology.
[20] D. Greenberg,et al. Palmitoylethanolamide Increases after Focal Cerebral Ischemia and Potentiates Microglial Cell Motility , 2003, The Journal of Neuroscience.
[21] G. Scambia,et al. Erythropoietin exerts anti‐apoptotic effects on rat microglial cells in vitro , 2002, The European journal of neuroscience.
[22] T. Möller,et al. Astrocytes in Culture Produce Anandamide and Other Acylethanolamides* , 2002, The Journal of Biological Chemistry.
[23] R. Mrak,et al. Interleukin-1, neuroinflammation, and Alzheimer’s disease , 2001, Neurobiology of Aging.
[24] H. Hansen,et al. N-Acylethanolamines and precursor phospholipids - relation to cell injury. , 2000, Chemistry and physics of lipids.
[25] E. Meli,et al. 1‐Aminoindan‐1,5‐dicarboxylic acid and (S)‐(+)‐2‐(3′‐carboxybicyclo[1.1.1] pentyl)‐glycine, two mGlu1 receptor‐preferring antagonists, reduce neuronal death in in vitro and in vivo models of cerebral ischaemia , 1999, The European journal of neuroscience.
[26] T. Willson,et al. Ligand binding and co-activator assembly of the peroxisome proliferator-activated receptor-γ , 1998, Nature.
[27] A. Buriani,et al. The ALIAmide palmitoylethanolamide and cannabinoids, but not anandamide, are protective in a delayed postglutamate paradigm of excitotoxic death in cerebellar granule neurons. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[28] K. Umesono,et al. Convergence of 9-cis retinoic acid and peroxisome proliferator signalling pathways through heterodimer formation of their receptors , 1992, Nature.