Variability in Serum Sodium Concentration and Prognostic Significance in Severe Traumatic Brain Injury: A Multicenter Observational Study

[1]  R. Bellomo,et al.  Serum sodium and intracranial pressure changes after desmopressin therapy in severe traumatic brain injury patients: a multi-centre cohort study , 2019, Annals of Intensive Care.

[2]  J. Anstey,et al.  Diabetes Insipidus and Syndrome of Inappropriate Antidiuretic Hormone in Critically Ill Patients. , 2019, Critical care clinics.

[3]  R. Bellomo,et al.  Early osmotherapy in severe traumatic brain injury: an international multicentre study. , 2019, Journal of neurotrauma.

[4]  Yuedong Wang,et al.  Increased Mortality Associated with Higher Pre-Dialysis Serum Sodium Variability: Results of the International MONitoring Dialysis Outcome Initiative , 2018, American Journal of Nephrology.

[5]  Md Hamidul Huque,et al.  A comparison of multiple imputation methods for missing data in longitudinal studies , 2018, BMC Medical Research Methodology.

[6]  B. Plaud,et al.  Association between continuous hyperosmolar therapy and survival in patients with traumatic brain injury – a multicentre prospective cohort study and systematic review , 2017, Critical Care.

[7]  D. Greenhalgh,et al.  Sodium variability is associated with increased mortality in severe burn injury , 2017, Burns & Trauma.

[8]  C. Robertson,et al.  Morbidity and mortality associated with hypernatremia in patients with severe traumatic brain injury. , 2017, Neurosurgical focus.

[9]  G. Brophy,et al.  Treatment of Hyponatremia in Patients with Acute Neurological Injury , 2017, Neurocritical Care.

[10]  J. Verbalis,et al.  Hyponatremia in Neurotrauma: The Role of Vasopressin. , 2016, Journal of neurotrauma.

[11]  Xiaofeng Meng,et al.  Traumatic Brain Injury Patients With a Glasgow Coma Scale Score of ⩽8, Cerebral Edema, and/or a Basal Skull Fracture are More Susceptible to Developing Hyponatremia , 2016, Journal of neurosurgical anesthesiology.

[12]  T. Yumoto,et al.  Prevalence, risk factors, and short-term consequences of traumatic brain injury-associated hyponatremia. , 2015, Acta medica Okayama.

[13]  R. Sterns Disorders of plasma sodium--causes, consequences, and correction. , 2015, The New England journal of medicine.

[14]  M. Li,et al.  Hypernatremia severity and the risk of death after traumatic brain injury. , 2013, Injury.

[15]  W. Tormey,et al.  Acute glucocorticoid deficiency and diabetes insipidus are common after acute traumatic brain injury and predict mortality. , 2013, The Journal of clinical endocrinology and metabolism.

[16]  R. Bellomo,et al.  Albumin resuscitation for traumatic brain injury: is intracranial hypertension the cause of increased mortality? , 2013, Journal of neurotrauma.

[17]  K. Reinhart,et al.  Fluctuations in serum sodium level are associated with an increased risk of death in surgical ICU patients. , 2013 .

[18]  T. Fabian,et al.  The relationship between serum sodium and intracranial pressure when using hypertonic saline to target mild hypernatremia in patients with head trauma , 2012, Critical Care.

[19]  S. Lohani,et al.  Hyponatremia in patients with traumatic brain injury: etiology, incidence, and severity correlation. , 2011, World neurosurgery.

[20]  J. Verbalis Brain volume regulation in response to changes in osmolality , 2010, Neuroscience.

[21]  D. Zygun,et al.  Characterization of intensive care unit acquired hyponatremia and hypernatremia following cardiac surgery , 2010, Canadian journal of anaesthesia = Journal canadien d'anesthesie.

[22]  G. Brabant,et al.  Following brain trauma, copeptin, a stable peptide derived from the AVP precusor, does not reflect osmoregulation but correlates with injury severity. , 2010, Acta neurochirurgica. Supplement.

[23]  Svetlana K. Eden,et al.  Immortal time bias in critical care research: Application of time-varying Cox regression for observational cohort studies* , 2009, Critical care medicine.

[24]  U. Maggiore,et al.  The relation between the incidence of hypernatremia and mortality in patients with severe traumatic brain injury , 2009, Critical care.

[25]  D. Zygun,et al.  The epidemiology of intensive care unit-acquired hyponatraemia and hypernatraemia in medical-surgical intensive care units , 2008, Critical care.

[26]  Y. Katayama,et al.  Hyponatremia in patients with traumatic brain injury: incidence, mechanism, and response to sodium supplementation or retention therapy with hydrocortisone. , 2007, Surgical neurology.

[27]  F. Prêteux,et al.  Opposed effects of hypertonic saline on contusions and noncontused brain tissue in patients with severe traumatic brain injury* , 2006, Critical care medicine.

[28]  E. Deibert,et al.  Hypernatremia in the neurologic intensive care unit: how high is too high? , 2006, Journal of critical care.

[29]  J. Phillips,et al.  Posterior pituitary dysfunction after traumatic brain injury. , 2004, The Journal of clinical endocrinology and metabolism.

[30]  C. Patlak,et al.  Volume regulatory loss of Na, Cl, and K from rat brain during acute hyponatremia. , 1987, The American journal of physiology.