Hippocampal CA1 βCaMKII mediates neuroinflammatory responses via COX-2/PGE2 signaling pathways in depression
暂无分享,去创建一个
[1] N. Gulyaeva. Functional Neurochemistry of the Ventral and Dorsal Hippocampus: Stress, Depression, Dementia and Remote Hippocampal Damage , 2018, Neurochemical Research.
[2] G. Ateş,et al. Genetic deletion of xCT attenuates peripheral and central inflammation and mitigates LPS‐induced sickness and depressive‐like behavior in mice , 2018, Glia.
[3] Wen-juan Lin,et al. Fibroblast Growth Factor 2 Modulates Hippocampal Microglia Activation in a Neuroinflammation Induced Model of Depression , 2018, Front. Cell. Neurosci..
[4] F. Klamt,et al. Alzheimer’s disease master regulators analysis: search for potential molecular targets and drug repositioning candidates , 2018, Alzheimer's Research & Therapy.
[5] Y. Chau,et al. GluN2B/CaMKII mediates CFA-induced hyperalgesia via HDAC4-modified spinal COX2 transcription , 2018, Neuropharmacology.
[6] Xuan Luo,et al. Galantamine improves cognition, hippocampal inflammation, and synaptic plasticity impairments induced by lipopolysaccharide in mice , 2018, Journal of neuroinflammation.
[7] A. Carvalho,et al. Deep brain stimulation for treatment-resistant depression: an integrative review of preclinical and clinical findings and translational implications , 2018, Molecular Psychiatry.
[8] Hailan Hu,et al. Astroglial Kir4.1 in the lateral habenula drives neuronal bursts in depression , 2018, Nature.
[9] B. Ebert,et al. Agonistic targeting of TLR1/TLR2 induces p38 MAPK-dependent apoptosis and NFκB-dependent differentiation of AML cells. , 2017, Blood advances.
[10] Sakulrat Mankhong,et al. Involvement of p38 MAPK and ATF‐2 signaling pathway in anti‐inflammatory effect of a novel compound bis[(5‐methyl)2‐furyl](4‐nitrophenyl)methane on lipopolysaccharide‐stimulated macrophages , 2017, International immunopharmacology.
[11] M. Kaster,et al. NLRP3 inflammasome-driven pathways in depression: Clinical and preclinical findings , 2017, Brain, Behavior, and Immunity.
[12] C. Pariante. Why are depressed patients inflamed? A reflection on 20 years of research on depression, glucocorticoid resistance and inflammation , 2017, European Neuropsychopharmacology.
[13] Z. Ronai,et al. ATF2, a paradigm of the multifaceted regulation of transcription factors in biology and disease , 2017, Pharmacological research.
[14] Arijit Ghosh,et al. Hippocampal CysLT1R knockdown or blockade represses LPS-induced depressive behaviors and neuroinflammatory response in mice , 2017, Acta Pharmacologica Sinica.
[15] Derk-Jan Dijk,et al. The Functional and Clinical Significance of the 24-Hour Rhythm of Circulating Glucocorticoids , 2016, Endocrine reviews.
[16] T. Suslow,et al. Pro- and anti-inflammatory cytokines, but not CRP, are inversely correlated with severity and symptoms of major depression , 2016, Psychiatry Research.
[17] H. Klein,et al. Electroconvulsive seizures (ECS) do not prevent LPS-induced behavioral alterations and microglial activation , 2015, Journal of Neuroinflammation.
[18] R. Yirmiya,et al. Depression as a Microglial Disease , 2015, Trends in Neurosciences.
[19] M. Adzic,et al. The contribution of hypothalamic neuroendocrine, neuroplastic and neuroinflammatory processes to lipopolysaccharide-induced depressive-like behaviour in female and male rats: Involvement of glucocorticoid receptor and C/EBP-β , 2015, Behavioural Brain Research.
[20] D. Ionescu,et al. Towards new mechanisms: an update on therapeutics for treatment-resistant major depressive disorder , 2015, Molecular Psychiatry.
[21] F. Su,et al. Fluoxetine and S-citalopram inhibit M1 activation and promote M2 activation of microglia in vitro , 2015, Neuroscience.
[22] E. Choi,et al. Compromised MAPK signaling in human diseases: an update , 2015, Archives of Toxicology.
[23] Qing-Yu Zhang,et al. Microglial NLRP3 inflammasome activation mediates IL-1β-related inflammation in prefrontal cortex of depressive rats , 2014, Brain, Behavior, and Immunity.
[24] G. Rosoklija,et al. Microglia of Prefrontal White Matter in Suicide , 2014, Journal of neuropathology and experimental neurology.
[25] H. Hsieh,et al. Upregulation of COX-2/PGE2 by ET-1 Mediated Through Ca2+-Dependent Signals in Mouse Brain Microvascular Endothelial Cells , 2014, Molecular Neurobiology.
[26] L. Edvinsson,et al. CaMKII and MEK1/2 inhibition time-dependently modify inflammatory signaling in rat cerebral arteries during organ culture , 2014, Journal of Neuroinflammation.
[27] A. Pudełko,et al. Imipramine and fluoxetine inhibit LPS-induced activation and affect morphology of microglial cells in the rat glial culture , 2014, Pharmacological reports : PR.
[28] Yasuo Watanabe,et al. Nitric oxide enhances increase in cytosolic Ca(2+) and promotes nicotine-triggered MAPK pathway in PC12 cells. , 2013, Nitric oxide : biology and chemistry.
[29] R. Malinow,et al. βCaMKII in Lateral Habenula Mediates Core Symptoms of Depression , 2013, Science.
[30] A. Malik,et al. Store-operated Ca2+ Entry (SOCE) Induced by Protease-activated Receptor-1 Mediates STIM1 Protein Phosphorylation to Inhibit SOCE in Endothelial Cells through AMP-activated Protein Kinase and p38β Mitogen-activated Protein Kinase* , 2013, The Journal of Biological Chemistry.
[31] Eric Lau,et al. ATF2 – at the crossroad of nuclear and cytosolic functions , 2012, Journal of Cell Science.
[32] H. Son,et al. Neuropathological abnormalities of astrocytes, GABAergic neurons, and pyramidal neurons in the dorsolateral prefrontal cortices of patients with major depressive disorder , 2012, European Neuropsychopharmacology.
[33] N. Herrmann,et al. A Meta-Analysis of Cytokines in Major Depression , 2010, Biological Psychiatry.
[34] M. Sofroniew,et al. Astrocytes: biology and pathology , 2009, Acta Neuropathologica.
[35] K. Ko,et al. Peony glycosides produce antidepressant-like action in mice exposed to chronic unpredictable mild stress: Effects on hypothalamic-pituitary-adrenal function and brain-derived neurotrophic factor , 2009, Progress in Neuro-Psychopharmacology and Biological Psychiatry.
[36] Charles L. Raison,et al. Inflammation and Its Discontents: The Role of Cytokines in the Pathophysiology of Major Depression , 2009, Biological Psychiatry.
[37] Stafford L. Lightman,et al. The HPA axis in major depression: classical theories and new developments , 2008, Trends in Neurosciences.
[38] B. Cuevas,et al. Role of mitogen-activated protein kinase kinase kinases in signal integration , 2007, Oncogene.
[39] R. Duman,et al. A Role for MAP Kinase Signaling in Behavioral Models of Depression and Antidepressant Treatment , 2007, Biological Psychiatry.
[40] P. Seeman,et al. Increased expression of calcium/calmodulin‐dependent protein kinase IIβ in frontal cortex in schizophrenia and depression , 2006, Synapse.
[41] M. Dragunow,et al. Activating transcription factor 2 expression in the adult human brain: Association with both neurodegeneration and neurogenesis , 2005, Neuroscience.
[42] S. Yamawaki,et al. Influence of immobilization stress on the levels of CaMKII and phospho-CaMKII in the rat hippocampus. , 2004, The international journal of neuropsychopharmacology.
[43] H. Manji,et al. Focus on CaMKII: a molecular switch in the pathophysiology and treatment of mood and anxiety disorders. , 2004, The international journal of neuropsychopharmacology.
[44] R. Post,et al. Decreased prefrontal CaMKII α mRNA in bipolar illness , 2002 .
[45] H. Li,et al. Retinoblastoma protein interacts with ATF2 and JNK/p38 in stimulating the transforming growth factor-beta2 promoter. , 2001, Archives of biochemistry and biophysics.
[46] J. Seckl,et al. Glucocorticoids and the ageing hippocampus , 2000, Journal of anatomy.
[47] H. Vedder,et al. Cytokine Production and Treatment Response in Major Depressive Disorder , 2000, Neuropsychopharmacology.
[48] Tobias Meyer,et al. CaMKIIβ Functions As an F-Actin Targeting Module that Localizes CaMKIIα/β Heterooligomers to Dendritic Spines , 1998, Neuron.
[49] B. Dérijard,et al. Transcription factor ATF2 regulation by the JNK signal transduction pathway , 1995, Science.
[50] S. Russo,et al. Immune and Neuroendocrine Mechanisms of Stress Vulnerability and Resilience , 2017, Neuropsychopharmacology.
[51] B. Bogerts,et al. Immunological aspects in the neurobiology of suicide: elevated microglial density in schizophrenia and depression is associated with suicide. , 2008, Journal of psychiatric research.
[52] R. Post,et al. Decreased prefrontal CaMKII alpha mRNA in bipolar illness. , 2002, Neuroreport.
[53] K. Shen,et al. CaMKIIbeta functions as an F-actin targeting module that localizes CaMKIIalpha/beta heterooligomers to dendritic spines. , 1998, Neuron.
[54] H. Schulman,et al. The multifunctional calcium/calmodulin-dependent protein kinase: from form to function. , 1995, Annual review of physiology.