Cytotoxic T cells specific for glutamic acid decarboxylase in autoimmune diabetes
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P. van Endert | F. Sinigaglia | R. Lang | A. Felix | P. Panina-Bordignon | E. Benazzi | G. A. Spinas | R. Pastore | Francesco Sinigaglia | G. A. Spinas | Paola Panina-Bordignon | Rosmarie Lang | Peter M. van Endert | Elena Benazzi | Arthur M. Felix | Rocco M. Pastore
[1] A. Mortara,et al. Interleukin 12 administration induces T helper type 1 cells and accelerates autoimmune diabetes in NOD mice , 1995, The Journal of experimental medicine.
[2] H. Mcdevitt,et al. Th1 and Th2 CD4+ T cells in the pathogenesis of organ-specific autoimmune diseases. , 1995, Immunology today.
[3] R. Tampé,et al. A sequential model for peptide binding and transport by the transporters associated with antigen processing. , 1994, Immunity.
[4] R. Tosi,et al. HLA-A2-binding peptides cross-react not only within the A2 subgroup but also with other HLA-A-locus allelic products. , 1994, Human immunology.
[5] T. Utsugi,et al. Evidence for the role of CD8+ cytotoxic T cells in the destruction of pancreatic beta-cells in nonobese diabetic mice. , 1994, Journal of immunology.
[6] A. Cooke. Autoimmune Disease: Gadding around the beta cell , 1994, Current Biology.
[7] C. Benoist,et al. Major histocompatibility complex class I molecules are required for the development of insulitis in non‐obese diabetic mice , 1993, European journal of immunology.
[8] R. Tisch,et al. Immune response to glutamic acid decarboxylase correlates with insulitis in non-obese diabetic mice , 1993, Nature.
[9] A. Tobin,et al. Spontaneous loss of T-cell tolerance to glutamic acid decarboxylase in murine insulin-dependent diabetes , 1993, Nature.
[10] C. Benoist,et al. Following a diabetogenic T cell from genesis through pathogenesis , 1993, Cell.
[11] J. Sidney,et al. Prominent role of secondary anchor residues in peptide binding to HLA-A2.1 molecules , 1993, Cell.
[12] S. H. van der Burg,et al. Identification of peptide sequences that potentially trigger HLA‐A2.1‐restricted cytotoxic T lymphocytes , 1993, European journal of immunology.
[13] S. Y. Yang,et al. Isoelectric focusing subtypes of HLA-A can be defined by oligonucleotide typing. , 1993, Tissue antigens.
[14] J. Miyazaki,et al. Prevention of autoimmune insulitis in nonobese diabetic mice by expression of major histocompatibility complex class I Ld molecules. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[15] M. Matsumoto,et al. Analysis of the roles of CD4+ and CD8+ T cells in autoimmune diabetes of NOD mice using transfer to NOD athymic nude mice , 1992, European journal of immunology.
[16] R. Henderson,et al. Characterization of peptides bound to the class I MHC molecule HLA-A2.1 by mass spectrometry. , 1992, Science.
[17] M. Erlander,et al. Response of peripheral-blood mononuclear cells to glutamate decarboxylase in insulin-dependent diabetes , 1992, The Lancet.
[18] H. Kikutani,et al. The murine autoimmune diabetes model: NOD and related strains. , 1992, Advances in immunology.
[19] Y. Fu,et al. Linkage of faulty major histocompatibility complex class I to autoimmune diabetes. , 1991, Science.
[20] W. Ogawa,et al. Prevention of Cyclophosphamide-Induced and Spontaneous Diabetes in NOD/Shi/Kbe Mice by Anti-MHC Class I Kd Monoclonal Antibody , 1991, Diabetes.
[21] H. Rammensee,et al. Allele-specific motifs revealed by sequencing of self-peptides eluted from MHC molecules , 1991, Nature.
[22] P. Bedossa,et al. Prevention of diabetes in NOD mice treated with antibody to murine IFN gamma. , 1991, Journal of autoimmunity.
[23] K. Haskins,et al. Acceleration of diabetes in young NOD mice with a CD4+ islet-specific T cell clone. , 1990, Science.
[24] S. Baekkeskov,et al. Identification of the 64K autoantigen in insulin-dependent diabetes as the GABA-synthesizing enzyme glutamic acid decarboxylase , 1990, Nature.
[25] N. Sarvetnick,et al. Loss of pancreatic islet tolerance induced by β-cell expression of interferon-γ , 1990, Nature.
[26] T. Elliott,et al. Assembly of MHC class I molecules analyzed in vitro , 1990, Cell.
[27] A. McMichael,et al. Class I cross-restricted T cells reveal low responder allele due to processing of viral antigen , 1989, Nature.
[28] J. Todd,et al. HLA-DQβ gene contributes to susceptibility and resistance to insulin-dependent diabetes mellitus , 1987, Nature.
[29] C. Boitard,et al. Syngeneic transfer of autoimmune diabetes from diabetic NOD mice to healthy neonates. Requirement for both L3T4+ and Lyt-2+ T cells , 1987, The Journal of experimental medicine.
[30] J. Schmidt,et al. A simple, rapid and large capacity ELISA for biologically active native and recombinant human IFN gamma. , 1987, Journal of biological regulators and homeostatic agents.
[31] D. R. Gamble,et al. In situ characterization of autoimmune phenomena and expression of HLA molecules in the pancreas in diabetic insulitis. , 1985, The New England journal of medicine.
[32] P. Terasaki,et al. Microdroplet testing for HLA-A, -B, -C, and -D antigens. The Phillip Levine Award Lecture. , 1978, American journal of clinical pathology.