Absence of 4-1BB reduces obesity-induced atrophic response in skeletal muscle

[1]  D. Guttridge,et al.  MyoD Regulates Skeletal Muscle Oxidative Metabolism Cooperatively with Alternative NF-κB. , 2016, Cell reports.

[2]  J. Long,et al.  Mitochondrial Dysfunction Launches Dexamethasone-Induced Skeletal Muscle Atrophy via AMPK/FOXO3 Signaling. , 2016, Molecular pharmaceutics.

[3]  Jeong Woo Park,et al.  4‐1BBL signaling promotes cell proliferation through reprogramming of glucose metabolism in monocytes/macrophages , 2015, The FEBS journal.

[4]  M. Chong,et al.  Monocyte chemoattractant protein-1: a proinflammatory cytokine elevated in sarcopenic obesity , 2015, Clinical interventions in aging.

[5]  K. Clément,et al.  Human Adipocytes Induce Inflammation and Atrophy in Muscle Cells During Obesity , 2015, Diabetes.

[6]  Mi-Kyung Sung,et al.  Quercetin Protects against Obesity-Induced Skeletal Muscle Inflammation and Atrophy , 2014, Mediators of inflammation.

[7]  S. Bhatnagar,et al.  Regulatory circuitry of TWEAK‐Fn14 system and PGC‐1α in skeletal muscle atrophy program , 2014, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[8]  Ashok Kumar,et al.  TWEAK/Fn14 Signaling Axis Mediates Skeletal Muscle Atrophy and Metabolic Dysfunction , 2014, Front. Immunol..

[9]  J. H. Yoon Park,et al.  Blockade of 4-1BB and 4-1BBL Interaction Reduces Obesity-Induced Skeletal Muscle Inflammation , 2013, Mediators of inflammation.

[10]  M. Sandri.  Protein breakdown in muscle wasting: Role of autophagy-lysosome and ubiquitin-proteasome☆☆☆ , 2013, The international journal of biochemistry & cell biology.

[11]  J. Pessin,et al.  Mechanisms for fiber-type specificity of skeletal muscle atrophy , 2013, Current opinion in clinical nutrition and metabolic care.

[12]  J. Zierath,et al.  Exercise metabolism and the molecular regulation of skeletal muscle adaptation. , 2013, Cell metabolism.

[13]  M. Sandri,et al.  Cellular and molecular mechanisms of muscle atrophy , 2013, Disease Models & Mechanisms.

[14]  R. Yu,et al.  4-1BB/4-1BBL Interaction Promotes Obesity-Induced Adipose Inflammation by Triggering Bidirectional Inflammatory Signaling in Adipocytes/Macrophages , 2012, Mediators of inflammation.

[15]  S. Bhatnagar,et al.  TWEAK and TRAF6 regulate skeletal muscle atrophy , 2012, Current opinion in clinical nutrition and metabolic care.

[16]  B. Lee,et al.  Deficiency for Costimulatory Receptor 4-1BB Protects Against Obesity-Induced Inflammation and Metabolic Disorders , 2011, Diabetes.

[17]  J. Girard,et al.  Increased Mitochondrial Fatty Acid Oxidation Is Sufficient to Protect Skeletal Muscle Cells from Palmitate-induced Apoptosis* , 2010, The Journal of Biological Chemistry.

[18]  A. Goldberg,et al.  Peroxisome Proliferator-activated Receptor γ Coactivator 1α or 1β Overexpression Inhibits Muscle Protein Degradation, Induction of Ubiquitin Ligases, and Disuse Atrophy* , 2010, The Journal of Biological Chemistry.

[19]  S. McGee,et al.  AMPK-mediated regulation of transcription in skeletal muscle. , 2010, Clinical science.

[20]  K. Tang,et al.  TNF‐α‐mediated reduction in PGC‐1α may impair skeletal muscle function after cigarette smoke exposure , 2010, Journal of cellular physiology.

[21]  B. Spiegelman,et al.  Increased muscle PGC-1α expression protects from sarcopenia and metabolic disease during aging , 2009, Proceedings of the National Academy of Sciences.

[22]  C. Peterson,et al.  Muscle inflammatory response and insulin resistance: synergistic interaction between macrophages and fatty acids leads to impaired insulin action. , 2009, American journal of physiology. Endocrinology and metabolism.

[23]  T. Chan,et al.  TNF-alpha reduces PGC-1alpha expression through NF-kappaB and p38 MAPK leading to increased glucose oxidation in a human cardiac cell model. , 2009, Cardiovascular research.

[24]  J. Houmard,et al.  Intramuscular lipid metabolism, insulin action, and obesity , 2009, IUBMB life.

[25]  Ian R. Lanza,et al.  Endurance Exercise as a Countermeasure for Aging , 2008, Diabetes.

[26]  L. Ferrucci,et al.  Sarcopenic obesity: definition, cause and consequences , 2008, Current opinion in clinical nutrition and metabolic care.

[27]  Ashok Kumar,et al.  Nuclear factor-kappa B signaling in skeletal muscle atrophy , 2008, Journal of Molecular Medicine.

[28]  Lieping Chen,et al.  CD137 Is Expressed in Human Atherosclerosis and Promotes Development of Plaque Inflammation in Hypercholesterolemic Mice , 2008, Circulation.

[29]  F. Fontanesi,et al.  Assembly of mitochondrial cytochrome c-oxidase, a complicated and highly regulated cellular process. , 2006, American journal of physiology. Cell physiology.

[30]  David Carling,et al.  Tumor necrosis factor alpha-induced skeletal muscle insulin resistance involves suppression of AMP-kinase signaling. , 2006, Cell metabolism.

[31]  B. Spiegelman,et al.  Peroxisome proliferator-activated receptor gamma coactivator 1 coactivators, energy homeostasis, and metabolism. , 2006, Endocrine reviews.

[32]  J. McLaurin,et al.  PPAR-α Expression Inversely Correlates with Inflammatory Cytokines IL-1β and TNF-α in Aging Rats , 2005, Neurochemical Research.

[33]  D. Glass,et al.  Skeletal muscle hypertrophy and atrophy signaling pathways. , 2005, The international journal of biochemistry & cell biology.

[34]  J. Hawley,et al.  Open access, freely available online Primer Skeletal Muscle Fiber Type: Influence on Contractile and Metabolic Properties , 2022 .

[35]  S. Kandarian,et al.  The molecular basis of skeletal muscle atrophy. , 2004, American journal of physiology. Cell physiology.

[36]  Genhong Cheng,et al.  The signaling adaptors and pathways activated by TNF superfamily. , 2003, Cytokine & growth factor reviews.

[37]  Z. Lee,et al.  Immune Responses in 4-1BB (CD137)-Deficient Mice1 , 2002, The Journal of Immunology.

[38]  G. Dohm,et al.  Muscle fiber type is associated with obesity and weight loss. , 2002, American journal of physiology. Endocrinology and metabolism.

[39]  Rena R Wing,et al.  Skeletal muscle fatty acid metabolism in association with insulin resistance, obesity, and weight loss. , 1999, American journal of physiology. Endocrinology and metabolism.

[40]  N. Halaihel,et al.  Heterogeneity of pig intestinald-glucose transport systems. , 1999, American journal of physiology. Cell physiology.

[41]  C. Takahashi,et al.  Cutting edge: 4-1BB is a bona fide CD8 T cell survival signal. , 1999, Journal of immunology.

[42]  B. Kwon,et al.  Role of 4-1BB in immune responses. , 1998, Seminars in immunology.

[43]  Z. Lee,et al.  Potential role of 4-1BB in T cell activation. Comparison with the costimulatory molecule CD28. , 1995, Journal of immunology.

[44]  In-kyu Lee,et al.  Hemin improves insulin sensitivity in skeletal muscle in high fat-fed mice. , 2014, Journal of pharmacological sciences.

[45]  Zhen Yan,et al.  Regulation of exercise-induced fiber type transformation, mitochondrial biogenesis, and angiogenesis in skeletal muscle. , 2011, Journal of applied physiology.

[46]  J. McLaurin,et al.  PPAR-alpha expression inversely correlates with inflammatory cytokines IL-1beta and TNF-alpha in aging rats. , 2005, Neurochemical research.