论文信息 - Transient decrease of serum iron after acute erythropoietin treatment contributes to hepcidin inhibition by ERFE in mice

Transient decrease of serum iron after acute erythropoietin treatment contributes to hepcidin inhibition by ERFE in mice

To coordinate iron supply with hemoglobin (Hb) synthesis, erythropoietin (EPO)-stimulated erythroid precursors release erythroferrone (ERFE), a soluble protein that suppresses the expression of hepcidin ( Hamp ).[1][1] Hepcidin is a short liver peptide that controls iron entry into the circulation

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