Diabetic endothelial dysfunction: the role of poly(ADP-ribose) polymerase activation
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K. Murthy | É. Szabó | C. Szabó | L. Liaudet | A. Salzman | L. Virág | G. Southan | P. Jagtap | J. Mabley | A. Marton | F. Soriano | D. Hoyt
[1] C. Szab,et al. Poly(ADP-ribose) polymerase activation and the pathogenesis of circulatory shock , 2000 .
[2] Zhao-Qi Wang,et al. Characterization of sPARP-1 , 2000, The Journal of Biological Chemistry.
[3] Y. Kaneda,et al. Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage , 2000, Nature.
[4] K Miyamoto,et al. Integrin-mediated neutrophil adhesion and retinal leukostasis in diabetes. , 2000, Investigative ophthalmology & visual science.
[5] Ç. Karasu. Time course of changes in endothelium-dependent and -independent relaxation of chronically diabetic aorta: role of reactive oxygen species. , 2000, European journal of pharmacology.
[6] C. Ong,et al. Detection of oxidative DNA damage in human sperm and its association with sperm function and male infertility. , 2000, Free radical biology & medicine.
[7] M. O’connor,et al. Requirement of intracellular calcium mobilization for peroxynitrite-induced poly(ADP-ribose) synthetase activation and cytotoxicity. , 1999, Molecular pharmacology.
[8] Xueliang Du,et al. Generation of reactive oxygen intermediates, activation of NF-κB, and induction of apoptosis in human endothelial cells by glucose: role of nitric oxide synthase? , 1999 .
[9] J. Tooke,et al. Vascular function in Type 2 diabetes mellitus and pre‐diabetes: the case for intrinsic endotheliopathy , 1999, Diabetic medicine : a journal of the British Diabetic Association.
[10] T. Wascher,et al. Increased superoxide anion formation in endothelial cells during hyperglycemia: an adaptive response or initial step of vascular dysfunction? , 1999, Diabetes research and clinical practice.
[11] R. Andriantsitohaina,et al. Resistance to endotoxic shock as a consequence of defective NF‐κB activation in poly (ADP‐ribose) polymerase‐1 deficient mice , 1999, The EMBO journal.
[12] Z. Naito,et al. Ultrastructural changes and immunohistochemical localization of nitric oxide synthase, advanced glycation end products and NF-kappa B in aorta of streptozotocin treated Mongolian gerbils. , 1999, Nihon Ika Daigaku zasshi.
[13] K. Kamata,et al. Effect of insulin treatment on smooth muscle contractility and endothelium‐dependent relaxation in rat aortae from established STZ‐induced diabetes , 1999, British journal of pharmacology.
[14] M. Laakso,et al. Reducing the burden of diabetes: managing cardiovascular disease † , 1999, Diabetes/metabolism research and reviews.
[15] S. Snyder,et al. Poly (ADP-ribose) polymerase, nitric oxide and cell death. , 1999, Trends in pharmacological sciences.
[16] S. Snyder,et al. Poly(ADP-ribose) polymerase-deficient mice are protected from streptozotocin-induced diabetes. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[17] H. Kolb,et al. Mice lacking the poly(ADP-ribose) polymerase gene are resistant to pancreatic beta-cell destruction and diabetes development induced by streptozocin , 1999, Nature Medicine.
[18] T. Lange,et al. Tankyrase, a poly(ADP-ribose) polymerase at human telomeres. , 1998, Science.
[19] B. Zingarelli,et al. Genetic disruption of poly (ADP-ribose) synthetase inhibits the expression of P-selectin and intercellular adhesion molecule-1 in myocardial ischemia/reperfusion injury. , 1998, Circulation research.
[20] H. Wong,et al. Melatonin inhibits expression of the inducible isoform of nitric oxide synthase in murine macrophages: role of inhibition of NFκB activation , 1998, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[21] G. Remuzzi,et al. Leukocyte-endothelial interaction is augmented by high glucose concentrations and hyperglycemia in a NF-kB-dependent fashion. , 1998, The Journal of clinical investigation.
[22] L. Rodríguez-Mañas,et al. Endothelial dysfunction and metabolic control in streptozotocin‐induced diabetic rats , 1998, British journal of pharmacology.
[23] S. Cuzzocrea,et al. Protection against peroxynitrite-induced fibroblast injury and arthritis development by inhibition of poly(ADP-ribose) synthase. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[24] Barry Halliwell,et al. Formation of nitric oxide-derived inflammatory oxidants by myeloperoxidase in neutrophils , 1998, Nature.
[25] S. Snyder,et al. Poly(ADP-ribose) polymerase gene disruption renders mice resistant to cerebral ischemia , 1997, Nature Medicine.
[26] Riaz-ul-Haq,et al. Activation of nuclear factor-kappaB in cultured endothelial cells by increased glucose concentration: prevention by calphostin C. , 1997, Journal of cardiovascular pharmacology.
[27] S. Cuzzocrea,et al. Endothelial dysfunction in a rat model of endotoxic shock. Importance of the activation of poly (ADP-ribose) synthetase by peroxynitrite. , 1997, The Journal of clinical investigation.
[28] B. Halliwell. What nitrates tyrosine? Is nitrotyrosine specific as a biomarker of peroxynitrite formation in vivo? , 1997, FEBS letters.
[29] T. Lüscher,et al. High glucose increases nitric oxide synthase expression and superoxide anion generation in human aortic endothelial cells. , 1997, Circulation.
[30] C. Rosenfeld,et al. Estrogen acutely stimulates nitric oxide synthase activity in fetal pulmonary artery endothelium. , 1997, The American journal of physiology.
[31] G. Pieper,et al. Diabetic-induced endothelial dysfunction in rat aorta: role of hydroxyl radicals. , 1997, Cardiovascular research.
[32] J. Coyle,et al. DNA strand breaks induced by sustained glutamate excitotoxicity in primary neuronal cultures , 1996, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[33] M. O’connor,et al. DNA strand breakage, activation of poly (ADP-ribose) synthetase, and cellular energy depletion are involved in the cytotoxicity of macrophages and smooth muscle cells exposed to peroxynitrite. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[34] G. Pieper,et al. Evaluation of the mechanism of endothelial dysfunction in the genetically-diabetic BB rat. , 1996, Life sciences.
[35] G. Pieper,et al. Syngeneic Pancreatic Islet Transplantation Reverses Endothelial Dysfunction in Experimental Diabetes , 1995, Diabetes.
[36] F. Luscinskas,et al. Activation‐Dependent Isolation and Culture of Murine Pulmonary Microvascular Endothelium , 1995, Microcirculation.
[37] J. Adams,et al. High-performance liquid chromatography analysis of oxidized and reduced pyridine dinucleotides in specific brain regions. , 1995, Analytical biochemistry.
[38] E. Wagner,et al. Mice lacking ADPRT and poly(ADP-ribosyl)ation develop normally but are susceptible to skin disease. , 1995, Genes & development.
[39] J S Beckman,et al. Peroxynitrite-mediated tyrosine nitration catalyzed by superoxide dismutase. , 1992, Archives of biochemistry and biophysics.
[40] K. Ueda,et al. Specific inhibitors of poly(ADP-ribose) synthetase and mono(ADP-ribosyl)transferase. , 1992, The Journal of biological chemistry.
[41] B. Brenner,et al. Nitric oxide synthesis in endothelial cells: evidence for a pathway inducible by TNF-alpha. , 1991, The American journal of physiology.
[42] R. Furchgott. The 1989 Ulf von Euler lecture. Studies on endothelium-dependent vasodilation and the endothelium-derived relaxing factor. , 1990, Acta physiologica Scandinavica.
[43] W. Earnshaw,et al. Detection of DNA cleavage in apoptotic cells. , 2000, Methods in enzymology.
[44] J. Zhang. Use of biotinylated NAD to label and purify ADP-ribosylated proteins. , 1997, Methods in enzymology.