chronic myelogenous leukemia New type of Bcr/Abl junction in Philadelphia chromosome-positive

[1]  G. Saglio,et al.  A new hereditary persistence of fetal hemoglobin deletion has the breakpoint within the 3' beta-globin gene enhancer. , 1990, Blood.

[2]  C. Croce,et al.  Nucleotide sequence analysis of human abl and bcr-abl cDNAs. , 1989, Oncogene.

[3]  C. Denny,et al.  Localization of preferential sites of rearrangement within the BCR gene in Philadelphia chromosome-positive acute lymphoblastic leukemia. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[4]  S. Thibodeau,et al.  The bcr Gene in Philadelphia Chromosome Positive Acute Lymphoblastic Leukemia , 1989 .

[5]  C. Westbrook,et al.  Molecular Diagnosis of the Philadelphia Chromosome in Acute Lymphoblastic Leukemia. , 1989, Leukemia & lymphoma.

[6]  J U Gutterman,et al.  The molecular genetics of Philadelphia chromosome-positive leukemias. , 1988, The New England journal of medicine.

[7]  K. Mills,et al.  The site of the breakpoint within the bcr is a prognostic factor in Philadelphia-positive CML patients. , 1988, Blood.

[8]  R. Berger,et al.  Molecular cloning of a 5' segment of the genomic phl gene defines a new breakpoint cluster region (bcr2) in Philadelphia-positive acute leukemias. , 1988, Leukemia.

[9]  G. Saglio,et al.  Variability of the molecular defects corresponding to the presence of a Philadelphia chromosome in human hematologic malignancies. , 1988, Blood.

[10]  Collins Sj.  Direct sequencing of amplified genomic fragments documents N-ras point mutations in myeloid leukemia. , 1988 .

[11]  R. Kurzrock,et al.  Identification of molecular variants of p210bcr-abl in chronic myelogenous leukemia , 1987 .

[12]  G. Mcmahon,et al.  Characterization of c-Ki-ras oncogene alleles by direct sequencing of enzymatically amplified DNA from carcinogen-induced tumors. , 1987, Proceedings of the National Academy of Sciences of the United States of America.

[13]  A. Berrebi,et al.  bcr-abl RNA in patients with chronic myelogenous leukemia. , 1987, Blood.

[14]  R. Kurzrock,et al.  A novel c-abl protein product in Philadelphia-positive acute lymphoblastic leukaemia , 1987, Nature.

[15]  O. Witte,et al.  Unique forms of the abl tyrosine kinase distinguish Ph1-positive CML from Ph1-positive ALL. , 1987, Science.

[16]  S. Smith,et al.  A new fused transcript in Philadelphia chromosome positive acute lymphocytic leukaemia , 1987, Nature.

[17]  S. Rodenhuis,et al.  Distinguishing the Philadelphia chromosome of acute lymphoblastic leukemia from its counterpart in chronic myelogenous leukemia. , 1985, The New England journal of medicine.

[18]  E. Canaani,et al.  Fused transcript of abl and bcr genes in chronic myelogenous leukaemia , 1985, Nature.

[19]  Kees Stam,et al.  Structural organization of the bcr gene and its role in the Ph′ translocation , 1985, Nature.

[20]  Susan M. Watanabe,et al.  An alteration of the human c-abl protein in K562 leukemia cells unmasks associated tyrosine kinase activity , 1984, Cell.

[21]  J. Stephenson,et al.  Philadelphia chromosomal breakpoints are clustered within a limited region, bcr, on chromosome 22 , 1984, Cell.

[22]  J. Sambrook,et al.  Molecular Cloning: A Laboratory Manual , 2001 .