Genetic polymorphisms of antioxidant enzymes CAT and SOD affect the outcome of clinical, biochemical, and anthropometric variables in people with obesity under a dietary intervention

[1]  P. De Feo,et al.  An intensive lifestyle intervention reduces circulating oxidised low-density lipoprotein and increases human paraoxonase activity in obese subjects. , 2016, Obesity research & clinical practice.

[2]  O. Tabatabaei-Malazy,et al.  Influencing of antioxidants' gene variants on risk of diabetes mellitus and its complications: a systematic review. , 2019, Minerva endocrinologica.

[3]  C. Hernández-Guerrero,et al.  Genetic Polymorphisms in SOD (rs2070424, rs7880) and CAT (rs7943316, rs1001179) Enzymes Are Associated with Increased Body Fat Percentage and Visceral Fat in an Obese Population from Central Mexico. , 2016, Archives of medical research.

[4]  Khyber Saify.  Genetic polymorphisms in the promoter region of catalase gene, creates new potential PAX-6 and STAT4 response elements , 2016, Molecular biology research communications.

[5]  M. Saadat,et al.  Influence of A-21T and C-262T genetic polymorphisms at the promoter region of the catalase (CAT) on gene expression , 2016, Environmental Health and Preventive Medicine.

[6]  J. Mu,et al.  Genetic variants in adiponectin and blood pressure responses to dietary sodium or potassium interventions: a family-based association study , 2016, Journal of Human Hypertension.

[7]  M. Duarte,et al.  The effects of rosuvastatin on lipid-lowering, inflammatory, antioxidant and fibrinolytics blood biomarkers are influenced by Val16Ala superoxide dismutase manganese-dependent gene polymorphism , 2016, The Pharmacogenomics Journal.

[8]  Saify Khyber.  GENETIC POLYMORPHISMS IN THE PROMOTER REGION OF CATALASE GENE, CREATES NEW POTENTIAL PAX-6 AND STAT4 RESPONSE ELEMENTS (SHORT COMMUNICATION) , 2016 .

[9]  A. Holmgren,et al.  Paradoxical Roles of Antioxidant Enzymes: Basic Mechanisms and Health Implications. , 2016, Physiological reviews.

[10]  C. Champagne,et al.  Position of the Academy of Nutrition and Dietetics: Interventions for the Treatment of Overweight and Obesity in Adults. , 2016, Journal of the Academy of Nutrition and Dietetics.

[11]  Prasenjit Manna,et al.  Obesity, Oxidative Stress, Adipose Tissue Dysfunction, and the Associated Health Risks: Causes and Therapeutic Strategies. , 2015, Metabolic syndrome and related disorders.

[12]  V. Lánská,et al.  Body Composition Changes in Adult Females after Lifestyle Intervention Are Influenced by the NYD-SP18 Variant. , 2015, Central European journal of public health.

[13]  César Angel Hernández Guerrero,et al.  GLUTATHIONE PEROXIDASE-1 PRO200LEU POLYMORPHISM (RS1050450) IS ASSOCIATED WITH MORBID OBESITY INDEPENDENTLY OF THE PRESENCE OF PREDIABETES OR DIABETES IN WOMEN FROM CENTRAL MEXICO. , 2015 .

[14]  E. Becer,et al.  Association of the Ala16Val MnSOD gene polymorphism with plasma leptin levels and oxidative stress biomarkers in obese patients. , 2015, Gene.

[15]  F. Hu,et al.  Advances in the Science, Treatment, and Prevention of the Disease of Obesity: Reflections From a Diabetes Care Editors’ Expert Forum , 2015, Diabetes Care.

[16]  G. Vargas-Alarcón,et al.  Adipose Tissue in Metabolic Syndrome: Onset and Progression of Atherosclerosis. , 2015, Archives of medical research.

[17]  Herbert F. Jelinek,et al.  Identifying Common Genetic Risk Factors of Diabetic Neuropathies , 2015, Front. Endocrinol..

[18]  L. Santos,et al.  Direct Healthcare Cost of Obesity in Brazil: An Application of the Cost-of-Illness Method from the Perspective of the Public Health System in 2011 , 2015, PloS one.

[19]  T. Buchanan,et al.  Variation in PPARG is associated with longitudinal change in insulin resistance in Mexican Americans at risk for type 2 diabetes. , 2015, The Journal of clinical endocrinology and metabolism.

[20]  C. H. Hernández Guerrero,et al.  GLUTATHIONE PEROXIDASE-1 PRO200LEU POLYMORPHISM (RS1050450) IS ASSOCIATED WITH MORBID OBESITY INDEPENDENTLY OF THE PRESENCE OF PREDIABETES OR DIABETES IN WOMEN FROM CENTRAL MEXICO. , 2015, Nutricion hospitalaria.

[21]  Angelo J. Canty,et al.  The association of previously reported polymorphisms for microvascular complications in a meta-analysis of diabetic retinopathy , 2014, Human Genetics.

[22]  Angelo J. Canty,et al.  The association of previously reported polymorphisms for microvascular complications in a meta-analysis of diabetic retinopathy , 2014, Human Genetics.

[23]  Niels Wacher-Rodarte,et al.  Triggering factors of primary care costs in the years following type 2 diabetes diagnosis in Mexico. , 2014, Archives of medical research.

[24]  W. Isabel,et al.  Prevalence of Catalase (-21 A/T) Gene Variant in South Indian (Tamil) Population , 2014, BioMed research international.

[25]  A. Bombak.  The “Obesity Epidemic”: Evolving Science, Unchanging Etiology , 2014 .

[26]  C. Aguilera,et al.  Genetics of Oxidative Stress in Obesity , 2014, International journal of molecular sciences.

[27]  R. Leis,et al.  Are catalase -844A/G polymorphism and activity associated with childhood obesity? , 2013, Antioxidants & redox signaling.

[28]  E. Parra,et al.  SOD2 gene Val16Ala polymorphism is associated with macroalbuminuria in Mexican Type 2 Diabetes patients: a comparative study and meta-analysis , 2013, BMC Medical Genetics.

[29]  A. Astrup,et al.  Influence of SNPs in nutrient-sensitive candidate genes and gene-diet interactions on blood lipids: the DiOGenes study. , 2013, The British journal of nutrition.

[30]  J. González‐Gallego,et al.  The MnSOD Ala16Val SNP: Relevance to human diseases and interaction with environmental factors , 2013, Free radical research.

[31]  O. Melander,et al.  Genetic susceptibility to obesity and diet intakes: association and interaction analyses in the Malmö Diet and Cancer Study , 2013, Genes & Nutrition.

[32]  S. Bijlsma,et al.  Nutrigenomics approach elucidates health-promoting effects of high vegetable intake in lean and obese men , 2013, Genes & Nutrition.

[33]  L. Quintana-Murci,et al.  The impact of natural selection on health and disease: uses of the population genetics approach in humans , 2013, Evolutionary applications.

[34]  J. Fletcher,et al.  Association of copper-zinc superoxide dismutase (SOD1) and manganese superoxide dismutase (SOD2) genes with nonsyndromic myelomeningocele. , 2012, Birth defects research. Part A, Clinical and molecular teratology.

[35]  L. Góth,et al.  Effects of rs769217 and rs1001179 polymorphisms of catalase gene on blood catalase, carbohydrate and lipid biomarkers in diabetes mellitus , 2012, Free radical research.

[36]  J. Coombes,et al.  Relationships between single nucleotide polymorphisms of antioxidant enzymes and disease. , 2012, Gene.

[37]  K. Xiang,et al.  Polymorphic variations in manganese superoxide dismutase (MnSOD), glutathione peroxidase-1 (GPX1), and catalase (CAT) contribute to elevated plasma triglyceride levels in Chinese patients with type 2 diabetes or diabetic cardiovascular disease , 2012, Molecular and Cellular Biochemistry.

[38]  J. Loscalzo,et al.  Glutathione peroxidase-1 in health and disease: from molecular mechanisms to therapeutic opportunities. , 2011, Antioxidants & redox signaling.

[39]  ZhiSheng Li,et al.  Genetic polymorphisms of superoxide dismutases, catalase, and glutathione peroxidase in age-related cataract , 2011, Molecular vision.

[40]  S. Humphries,et al.  Association between the rs4880 superoxide dismutase 2 (C>T) gene variant and coronary heart disease in diabetes mellitus. , 2010, Diabetes research and clinical practice.

[41]  B. Jaber,et al.  NADPH oxidase p22phox and catalase gene variants are associated with biomarkers of oxidative stress and adverse outcomes in acute renal failure. , 2007, Journal of the American Society of Nephrology : JASN.

[42]  H. Vincent,et al.  Biomarkers and potential mechanisms of obesity-induced oxidant stress in humans , 2006, International Journal of Obesity.

[43]  P. Stover.  Influence of human genetic variation on nutritional requirements. , 2006, The American journal of clinical nutrition.

[44]  B. Ainsworth,et al.  International physical activity questionnaire: 12-country reliability and validity. , 2003, Medicine and science in sports and exercise.

[45]  M. Martín,et al.  Optimización de un método para la determinación de la peroxidación lipídica en suero humano , 2001 .

[46]  I. Romieu,et al.  Validity and reproducibility of a food frequency questionnaire to assess dietary intake of women living in Mexico City. , 1998, Salud publica de Mexico.

[47]  M. Irigoyen-Camacho.  Caries dental en escolares del Distrito Federal , 1997 .

[48]  The validity of 24-hour dietary recalls. , 1976, Nutrition reviews.