Last Word on Viewpoint: Why predominantly neurological decompression sickness in breath-hold divers?

to the editor: We appreciate the insightful comments provided by all authors (see Ref. [3][1]) who commented on our Viewpoint ([4][2]) on the hypothesis that the hypoxia of breath-hold diving may recruit pulmonary shunts and lead to the arterialization of venous gas emboli (VGE). Because of the

[1]  K. Tetzlaff,et al.  Why predominantly neurological decompression sickness in breath-hold divers? , 2016, Journal of applied physiology.

[2]  Jonathan E. Elliott,et al.  Commentaries on Viewpoint: Why predominantly neurological DCS in breath-hold divers? , 2016, Journal of applied physiology.

[3]  P. Sheard.  Ocular tear film bubble formation after breath-hold diving. , 2008, Undersea & hyperbaric medicine : journal of the Undersea and Hyperbaric Medical Society, Inc.

[4]  A. Boussuges,et al.  Bubbles in the left cardiac cavities after diving , 2007, Heart.

[5]  A. Diedrich,et al.  Central chemoreflex sensitivity and sympathetic neural outflow in elite breath-hold divers. , 2008, Journal of applied physiology.