FAM19A5, a brain-specific chemokine, inhibits RANKL-induced osteoclast formation through formyl peptide receptor 2

[1]  Y. Bae,et al.  Formyl Peptide Receptors in Cellular Differentiation and Inflammatory Diseases , 2017, Journal of cellular biochemistry.

[2]  C. Tsatsanis,et al.  Akt Signaling Pathway in Macrophage Activation and M1/M2 Polarization , 2017, The Journal of Immunology.

[3]  Xinjian Peng,et al.  Identification of FAM3D as a new endogenous chemotaxis agonist for the formyl peptide receptors , 2016, Journal of Cell Science.

[4]  Xinjian Peng,et al.  Identification of FAM 3 D as a novel endogenous chemotaxis agonist for the FPRs ( formyl peptide receptors ) , 2016 .

[5]  Hak Jung Kim,et al.  Serum amyloid A inhibits RANKL-induced osteoclast formation , 2015, Experimental & Molecular Medicine.

[6]  G. Landreth,et al.  ERK Signaling Is Essential for Macrophage Development , 2015, PloS one.

[7]  K. Dhaliwal,et al.  The role of formylated peptides and formyl peptide receptor 1 in governing neutrophil function during acute inflammation. , 2015, The American journal of pathology.

[8]  R. Tohtong,et al.  Novel Serum Biomarkers to Differentiate Cholangiocarcinoma from Benign Biliary Tract Diseases Using a Proteomic Approach , 2015, Disease markers.

[9]  C. Eberhart,et al.  MicroRNA Profiling in Intraocular Medulloepitheliomas , 2015, PloS one.

[10]  E. Mellins,et al.  Alternative pathways of osteoclastogenesis in inflammatory arthritis , 2015, Nature Reviews Rheumatology.

[11]  Yuehui Wang,et al.  FAM19A3, a novel secreted protein, modulates the microglia/macrophage polarization dynamics and ameliorates cerebral ischemia , 2015, FEBS letters.

[12]  Jung Ha Kim,et al.  MicroRNA-26a Regulates RANKL-Induced Osteoclast Formation , 2014, Molecules and cells.

[13]  Tingting Li,et al.  FAM19A4 is a novel cytokine ligand of formyl peptide receptor 1 (FPR1) and is able to promote the migration and phagocytosis of macrophages , 2014, Cellular and Molecular Immunology.

[14]  D. Ma,et al.  Low Concentration of Quercetin Antagonizes the Cytotoxic Effects of Anti-Neoplastic Drugs in Ovarian Cancer , 2014, PloS one.

[15]  J. Seo,et al.  Oxidized low-density lipoprotein-induced foam cell formation is mediated by formyl peptide receptor 2. , 2014, Biochemical and biophysical research communications.

[16]  Rebecca Seal,et al.  TAFA4, a chemokine-like protein, modulates injury-induced mechanical and chemical pain hypersensitivity in mice. , 2013, Cell reports.

[17]  Joon-Hyuk Choi,et al.  Role of formyl peptide receptor 2 on the serum amyloid A-induced macrophage foam cell formation. , 2013, Biochemical and biophysical research communications.

[18]  Han-Woong Lee,et al.  Knockout mice created by TALEN-mediated gene targeting , 2013, Nature Biotechnology.

[19]  T. Miyamoto Regulation of Osteoclast Differentiation and Bone Homeostasis , 2012 .

[20]  D. Ye,et al.  Pleiotropic regulation of macrophage polarization and tumorigenesis by formyl peptide receptor-2 , 2011, Oncogene.

[21]  Jinhu Xiong,et al.  Matrix-embedded cells control osteoclast formation , 2011, Nature Medicine.

[22]  Hak Jung Kim,et al.  Sphingosylphosphorylcholine Stimulates CCL2 Production from Human Umbilical Vein Endothelial Cells , 2011, The Journal of Immunology.

[23]  G. Tomar,et al.  IL-3 Inhibits Human Osteoclastogenesis and Bone Resorption through Downregulation of c-Fms and Diverts the Cells to Dendritic Cell Lineage , 2010, The Journal of Immunology.

[24]  H. Takayanagi,et al.  Interferon regulatory factor-8 regulates bone metabolism by suppressing osteoclastogenesis , 2009, Nature Medicine.

[25]  Marc Parmentier,et al.  International Union of Basic and Clinical Pharmacology. LXXIII. Nomenclature for the Formyl Peptide Receptor (FPR) Family , 2009, Pharmacological Reviews.

[26]  M. Birnbaum,et al.  Osteoclast stimulatory transmembrane protein (OC‐STAMP), a novel protein induced by RANKL that promotes osteoclast differentiation , 2008, Journal of cellular physiology.

[27]  M. Ishii,et al.  Osteoclast cell fusion: mechanisms and molecules , 2008, Modern rheumatology.

[28]  B. Lamothe,et al.  TRAF6 ubiquitin ligase is essential for RANKL signaling and osteoclast differentiation. , 2007, Biochemical and biophysical research communications.

[29]  Soo Young Lee,et al.  MafB negatively regulates RANKL-mediated osteoclast differentiation. , 2007, Blood.

[30]  Marc Parmentier,et al.  Formyl peptide receptors: a promiscuous subfamily of G protein-coupled receptors controlling immune responses. , 2006, Cytokine & growth factor reviews.

[31]  Teiji Wada,et al.  RANKL-RANK signaling in osteoclastogenesis and bone disease. , 2006, Trends in molecular medicine.

[32]  Y. Toyama,et al.  DC-STAMP is essential for cell–cell fusion in osteoclasts and foreign body giant cells , 2005, The Journal of experimental medicine.

[33]  Yeong‐Min Park,et al.  The Synthetic Peptide Trp-Lys-Tyr-Met-Val-D-Met Inhibits Human Monocyte-Derived Dendritic Cell Maturation via Formyl Peptide Receptor and Formyl Peptide Receptor-Like 21 , 2005, The Journal of Immunology.

[34]  Xu Feng RANKing Intracellular Signaling in Osteoclasts , 2005, IUBMB life.

[35]  J. Kwak,et al.  Identification of Peptides That Antagonize Formyl Peptide Receptor-Like 1-Mediated Signaling1 , 2004, The Journal of Immunology.

[36]  T. Taniguchi,et al.  Costimulatory signals mediated by the ITAM motif cooperate with RANKL for bone homeostasis , 2004, Nature.

[37]  W. Funk,et al.  TAFA: a novel secreted family with conserved cysteine residues and restricted expression in the brain. , 2004, Genomics.

[38]  David L. Lacey,et al.  Osteoclast differentiation and activation , 2003, Nature.

[39]  E. Wagner,et al.  Reaching a genetic and molecular understanding of skeletal development. , 2002, Developmental cell.

[40]  S. Ryu,et al.  Identification of novel chemoattractant peptides for human leukocytes. , 2001, Blood.

[41]  Ji Ming Wang,et al.  The Synthetic Peptide Trp-Lys-Tyr-Met-Val-d-Met Is a Potent Chemotactic Agonist for Mouse Formyl Peptide Receptor1 , 2000, The Journal of Immunology.

[42]  H. Yasuda,et al.  RANK is the essential signaling receptor for osteoclast differentiation factor in osteoclastogenesis. , 1998, Biochemical and biophysical research communications.