Clinical and molecular characterizations of novel POU 3 F 4 mutations reveal that DFN 3 is due to null function of POU 3 F 4 protein
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G. Vriend | H. Venselaar | Myengmo Kang | T. Kwon | J. Bok | U. Kim | M. Song | Jung Tae Lee | W. Lee | Su-Jin Choi | Hee Keun Lee | Kyoung-Ah Kong | Hong‐Joon Park | Kyu‐Yup Lee | Sujin Choi