Heterogeneity of channel density in inositol-1,4,5-trisphosphate-sensitive Ca2+ stores
暂无分享,去创建一个
[1] H. Kasai,et al. Spatial dynamics of second messengers: IP3 and cAMP as long-range and associative messengers , 1994, Trends in Neurosciences.
[2] M. F. Schneider,et al. Inositol 1,4,5-trisphosphate-mediated quantal Ca2+ release measured by high resolution imaging of Ca2+ within organelles. , 1993, The Journal of biological chemistry.
[3] L. Missiaen,et al. Loading dependence of inositol 1,4,5-trisphosphate-induced Ca2+ release in the clonal cell line A7r5. Implications for the mechanism of quantal Ca2+ release. , 1993, The Journal of biological chemistry.
[4] G. Hajnóczky,et al. Imaging of inositol 1,4,5-trisphosphate-induced Ca2+ fluxes in single permeabilized hepatocytes. Demonstration of both quantal and nonquantal patterns of Ca2+ release. , 1993, The Journal of biological chemistry.
[5] M. Endo,et al. Caffeine Inhibits Ca2+-Mediated Potentiation of Inositol 1,4,5-Trisphosphate-Induced Ca2+ Release in Permeabilized Vascular Smooth Muscle Cells , 1993 .
[6] T. Machen,et al. Technique for in situ measurement of calcium in intracellular inositol 1,4,5-trisphosphate-sensitive stores using the fluorescent indicator mag-fura-2. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[7] A. Dawson,et al. A steady-state mechanism can account for the properties of inositol 2,4,5-trisphosphate-stimulated Ca2+ release from permeabilized L1210 cells. , 1993, The Biochemical journal.
[8] M. Berridge. Inositol trisphosphate and calcium signalling , 1993, Nature.
[9] Masamitsu Iino,et al. Calcium-dependent immediate feedback control of inositol 1,4,5-trisphosphate-induced Ca2+ release , 1992, Nature.
[10] L. Missiaen,et al. Ca2+ release induced by inositol 1,4,5-trisphosphate is a steady-state phenomenon controlled by luminal Ca2+ in permeabilized cells , 1992, Nature.
[11] Y. Imaizumi,et al. Effects of cyclopiazonic acid, a novel Ca2+‐ATPase inhibitor, on contractile responses in skinned ileal smooth muscle , 1992, British journal of pharmacology.
[12] M. Berridge,et al. All‐or‐nothing Ca2+ mobilization from the intracellular stores of single histamine‐stimulated HeLa cells. , 1992, The Journal of physiology.
[13] R. Huganir,et al. Quantal calcium release by purified reconstituted inositol 1,4,5-trisphosphate receptors , 1992, Nature.
[14] C. Taylor,et al. Quantal Ca2+ mobilization stimulated by inositol 1,4,5-trisphosphate in permeabilized hepatocytes. , 1991, The Biochemical journal.
[15] M. Berridge,et al. Spontaneous calcium release from inositol trisphosphate-sensitive calcium stores , 1991, Nature.
[16] James Watras,et al. Bell-shaped calcium-response curves of lns(l,4,5)P3- and calcium-gated channels from endoplasmic reticulum of cerebellum , 1991, Nature.
[17] S. M. Goldin,et al. Calcium as a coagonist of inositol 1,4,5-trisphosphate-induced calcium release. , 1991, Science.
[18] I. Parker,et al. Localized all-or-none calcium liberation by inositol trisphosphate. , 1990, Science.
[19] M. Iino,et al. Biphasic Ca2+ dependence of inositol 1,4,5-trisphosphate-induced Ca release in smooth muscle cells of the guinea pig taenia caeci , 1990, The Journal of general physiology.
[20] L. Stryer,et al. Transient calcium release induced by successive increments of inositol 1,4,5-trisphosphate. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[21] R. Irvine. ‘Quanta’ Ca2+ release and the control of Ca2+ entry by inositol phosphates ‐ a possible mechanism , 1990, FEBS letters.
[22] M. Iino. Calcium-induced calcium release mechanism in guinea pig taenia caeci , 1989, The Journal of general physiology.
[23] J. Mullaney,et al. GTP-activated communication between distinct inositol 1,4,5-trisphosphate-sensitive and -insensitive calcium pools , 1989, Nature.
[24] R. London,et al. A fluorescent indicator for measuring cytosolic free magnesium. , 1989, The American journal of physiology.
[25] S. Muallem,et al. Hormone-evoked calcium release from intracellular stores is a quantal process. , 1989, The Journal of biological chemistry.
[26] S. Kobayashi,et al. Heparin inhibits the inositol 1,4,5-trisphosphate-dependent, but not the independent, calcium release induced by guanine nucleotide in vascular smooth muscle. , 1988, Biochemical and biophysical research communications.
[27] G. Ahnert-Hilger,et al. Controlled manipulation of the cell interior by pore-forming proteins. , 1988, Trends in pharmacological sciences.
[28] F. Plum. Handbook of Physiology. , 1960 .