Rho GTPases Control Polarity, Protrusion, and Adhesion during Cell Movement
暂无分享,去创建一个
[1] X. Q. Chen,et al. The p160 RhoA-binding kinase ROK alpha is a member of a kinase family and is involved in the reorganization of the cytoskeleton , 1996, Molecular and cellular biology.
[2] A. Gotlieb,et al. Microtubule-organizing centers and cell migration: effect of inhibition of migration and microtubule disruption in endothelial cells , 1983, The Journal of cell biology.
[3] S. Narumiya,et al. Molecular Dissection of the Rho-associated Protein Kinase (p160ROCK)-regulated Neurite Remodeling in Neuroblastoma N1E-115 Cells , 1998, The Journal of cell biology.
[4] M. Schliwa,et al. Mechanism of centrosome positioning during the wound response in BSC-1 cells , 1992, The Journal of cell biology.
[5] L. Romer,et al. Inhibition of focal adhesion kinase (FAK) signaling in focal adhesions decreases cell motility and proliferation. , 1996, Molecular biology of the cell.
[6] Anne J. Ridley,et al. The small GTP-binding protein rho regulates the assembly of focal adhesions and actin stress fibers in response to growth factors , 1992, Cell.
[7] John Philip Trinkaus,et al. Cells into Organs: The Forces That Shape the Embryo , 1984 .
[8] D. Lauffenburger,et al. Cell Migration: A Physically Integrated Molecular Process , 1996, Cell.
[9] A. Hall,et al. The assembly of integrin adhesion complexes requires both extracellular matrix and intracellular rho/rac GTPases , 1995, The Journal of cell biology.
[10] M. Moran,et al. Phosphorylation of GAP and GAP-associated proteins by transforming and mitogenic tyrosine kinases , 1990, Nature.
[11] Anne J. Ridley,et al. The small GTP-binding protein rac regulates growth factor-induced membrane ruffling , 1992, Cell.
[12] K. Fujisawa,et al. p160ROCK, a Rho‐associated coiled‐coil forming protein kinase, works downstream of Rho and induces focal adhesions , 1997, FEBS letters.
[13] S. Pawar,et al. Differential gene expression in migrating renal epithelial cells after wounding , 1995, Journal of cellular physiology.
[14] A. Hall,et al. Rho GTPases and the actin cytoskeleton. , 1998, Science.
[15] L. M. May,et al. Distribution of microtubule organizing centers in migrating sheets of endothelial cells , 1981, The Journal of cell biology.
[16] A. Bershadsky,et al. Disruption of the Golgi apparatus by brefeldin A blocks cell polarization and inhibits directed cell migration. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[17] K. Kaibuchi,et al. Formation of Actin Stress Fibers and Focal Adhesions Enhanced by Rho-Kinase , 1997, Science.
[18] E. Hafen,et al. The Drosophila Jun-N-terminal kinase is required for cell morphogenesis but not for DJun-dependent cell fate specification in the eye. , 1996, Genes & development.
[19] M. Ziman,et al. Mutational analysis of CDC42Sc, a Saccharomyces cerevisiae gene that encodes a putative GTP-binding protein involved in the control of cell polarity , 1991, Molecular and cellular biology.
[20] P. Comoglio,et al. Regulation of scatter factor/hepatocyte growth factor responses by Ras, Rac, and Rho in MDCK cells , 1995, Molecular and cellular biology.
[21] L. Lim,et al. The Ras-related protein Cdc42Hs and bradykinin promote formation of peripheral actin microspikes and filopodia in Swiss 3T3 fibroblasts , 1995, Molecular and cellular biology.
[22] R. Weinberg,et al. Association between GTPase activators for Rho and Ras families , 1992, Nature.
[23] C. S. Izzard,et al. Cell-to-substrate contacts in living fibroblasts: an interference reflexion study with an evaluation of the technique. , 1976, Journal of cell science.
[24] S. Noselli. JNK signaling and morphogenesis in Drosophila. , 1998, Trends in genetics : TIG.
[25] T. Mitchison,et al. Actin-Based Cell Motility and Cell Locomotion , 1996, Cell.
[26] A. Ridley,et al. Rho: theme and variations , 1996, Current Biology.
[27] T. Sasaki,et al. Involvement of rho p21 and its inhibitory GDP/GTP exchange protein (rho GDI) in cell motility , 1993, Molecular and cellular biology.
[28] J. Couchman,et al. The behaviour of fibroblasts migrating from chick heart explants: changes in adhesion, locomotion and growth, and in the distribution of actomyosin and fibronectin. , 1979, Journal of cell science.
[29] D. Drubin,et al. Origins of Cell Polarity , 1996, Cell.
[30] S. Singer,et al. Polarization of the Golgi apparatus and the microtubule-organizing center in cultured fibroblasts at the edge of an experimental wound. , 1982, Proceedings of the National Academy of Sciences of the United States of America.
[31] B. Zetter,et al. Platelet-derived Growth Factor and Fibronectin-stimulated Migration Are Differentially Regulated by the Rac and Extracellular Signal-regulated Kinase Pathways* , 1997, The Journal of Biological Chemistry.
[32] L. Van Aelst,et al. Rho GTPases and signaling networks. , 1997, Genes & development.
[33] John G. Collard,et al. A role for Rac in Tiaml-induced membrane ruffling and invasion , 1995, Nature.
[34] J. Chant,et al. Rac and Cdc42 Induce Actin Polymerization and G1 Cell Cycle Progression Independently of p65PAK and the JNK/SAPK MAP Kinase Cascade , 1996, Cell.
[35] Shuh Narumiya,et al. Calcium sensitization of smooth muscle mediated by a Rho-associated protein kinase in hypertension , 1997, Nature.
[36] W. T. Chen. Mechanism of retraction of the trailing edge during fibroblast movement , 1981, The Journal of cell biology.
[37] K. Burridge,et al. Focal adhesions, contractility, and signaling. , 1996, Annual review of cell and developmental biology.
[38] Y. Zheng,et al. Rho proteins play a critical role in cell migration during the early phase of mucosal restitution. , 1997, The Journal of clinical investigation.
[39] Paul Martin,et al. Wound Healing--Aiming for Perfect Skin Regeneration , 1997, Science.
[40] A. Hall,et al. Purification of recombinant Rho/Rac/G25K from Escherichia coli. , 1995, Methods in enzymology.
[41] K. Rottner,et al. Actin and the coordination of protrusion, attachment and retraction in cell crawling , 1996, Bioscience reports.
[42] C. Marshall,et al. rho family GTPase activating proteins p190, bcr and rhoGAP show distinct specificities in vitro and in vivo. , 1993, The EMBO journal.
[43] David A. Cheresh,et al. Regulation of Cell Motility by Mitogen-activated Protein Kinase , 1997, The Journal of cell biology.
[44] R. Bravo,et al. Wounding a fibroblast monolayer results in the rapid induction of the c‐fos proto‐oncogene. , 1986, The EMBO journal.
[45] T. Pawson,et al. The N‐terminal region of GAP regulates cytoskeletal structure and cell adhesion. , 1993, The EMBO journal.
[46] G. Sa,et al. The regulation of endothelial cell motility by p21 ras. , 1994, Oncogene.
[47] B. Gumbiner,et al. Cell Adhesion: The Molecular Basis of Tissue Architecture and Morphogenesis , 1996, Cell.
[48] P. Warne,et al. Role of Phosphoinositide 3-OH Kinase in Cell Transformation and Control of the Actin Cytoskeleton by Ras , 1997, Cell.
[49] J. Chant,et al. Regulation of the polarization of T cells toward antigen-presenting cells by Ras-related GTPase CDC42. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[50] S. Noselli,et al. Coupling of Jun amino-terminal kinase and Decapentaplegic signaling pathways in Drosophila morphogenesis. , 1997, Genes & development.
[51] R. Sosnowski,et al. Interference with endogenous ras function inhibits cellular responses to wounding , 1993, Journal of Cell Biology.
[52] T. Sasaki,et al. Involvement of Rho p21 small GTP-binding protein and its regulator in the HGF-induced cell motility. , 1994, Oncogene.
[53] B. Zetter,et al. The chemotactic response to PDGF-BB: evidence of a role for Ras , 1995, Journal of Cell Biology.
[54] John G. Collard,et al. Identification of an invasion-inducing gene, Tiam-1, that encodes a protein with homology to GDP-GTP exchangers for Rho-like proteins , 1994, Cell.
[55] C. Nobes,et al. An early molecular component of the wound healing response in rat embryos—induction of c-fos protein in cells at the epidermal wound margin , 1992, Mechanisms of Development.
[56] D. Drubin. Development of cell polarity in budding yeast , 1991, Cell.
[57] J. Pringle,et al. CDC42 and CDC43, two additional genes involved in budding and the establishment of cell polarity in the yeast Saccharomyces cerevisiae , 1990, The Journal of cell biology.
[58] E. Scolnick,et al. Monoclonal antibodies to the p21 products of the transforming gene of Harvey murine sarcoma virus and of the cellular ras gene family , 1982, Journal of virology.
[59] S. Aizawa,et al. Reduced cell motility and enhanced focal adhesion contact formation in cells from FAK-deficient mice , 1995, Nature.
[60] C. Nobes,et al. Rho, Rac, and Cdc42 GTPases regulate the assembly of multimolecular focal complexes associated with actin stress fibers, lamellipodia, and filopodia , 1995, Cell.
[61] M. Rothkegel,et al. The molecular architecture of focal adhesions. , 1995, Annual review of cell and developmental biology.
[62] T. Hunter,et al. Integrin signalling and tyrosine phosphorylation: just the FAKs? , 1998, Trends in cell biology.
[63] C. Der,et al. Cdc42 and Rac1 induce integrin-mediated cell motility and invasiveness through PI(3)K , 1997, Nature.
[64] Daniel Zicha,et al. A Role for Cdc42 in Macrophage Chemotaxis , 1998, The Journal of cell biology.
[65] K. Burridge,et al. Focal adhesion assembly. , 1997, Trends in cell biology.
[66] J. Couchman,et al. Controlled induction of focal adhesion disassembly and migration in primary fibroblasts. , 1993, Journal of cell science.
[67] L. F. Ciufo,et al. Nuclear export of yeast signal recognition particle lacking Srp54p by the Xpo1p/Crm1p NES-dependent pathway , 2000, Current Biology.