Aminooxyacetic acid striatal lesions attenuated by 1,3-butanediol and coenzyme Q10

[1]  B. Rosen,et al.  Age‐Dependent Vulnerability of the Striatum to the Mitochondrial Toxin 3‐Nitropropionic Acid , 1993, Journal of neurochemistry.

[2]  F. Nicoletti,et al.  Ubiquinone Protects Cultured Neurons against Spontaneous and Excitotoxin-Induced Degeneration , 1992, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[3]  B. Rosen,et al.  1‐Methyl‐4‐Phenylpyridinium Produces Excitotoxic Lesions in Rat Striatum as a Result of Impairment of Oxidative Metabolism , 1992, Journal of neurochemistry.

[4]  R. Albin,et al.  Alternative excitotoxic hypotheses , 1992, Neurology.

[5]  M. Beal,et al.  Does impairment of energy metabolism result in excitotoxic neuronal death in neurodegenerative illnesses? , 1992, Annals of neurology.

[6]  B. Hyman,et al.  Aminooxyacetic Acid Results in Excitotoxin Lesions by a Novel Indirect Mechanism , 1991, Journal of neurochemistry.

[7]  N. Nishitani,et al.  Marked reduction in CSF lactate and pyruvate levels after CoQ therapy in a patient with mitochondrial myopathy, encephalopathy, lactic acidosis and stroke‐like episodes (MELAS) , 1991, Acta neurologica Scandinavica.

[8]  M. Beal,et al.  Kynurenine Pathway Measurements in Huntington's Disease Striatum: Evidence for Reduced Formation of Kynurenic Acid , 1990, Journal of neurochemistry.

[9]  L. Rochette,et al.  Beneficial effect of 1,3-butanediol on cerebral energy metabolism and edema following brain embolization in rats. , 1990, Stroke.

[10]  B. Ames,et al.  Ubiquinol-10 is an effective lipid-soluble antioxidant at physiological concentrations. , 1990, Proceedings of the National Academy of Sciences of the United States of America.

[11]  S. Kuroda,et al.  Mitochondrial encephalomyopathy (MELAS): Pathological study and successful therapy with coenzyme Q10 and idebenone , 1989, Journal of the Neurological Sciences.

[12]  S Yorifuji,et al.  Long‐term coenzyme Q10 therapy for a mitochondrial encephalomyopathy with cytochrome c oxidase deficiency , 1989, Neurology.

[13]  C. Marie,et al.  Protective Action of 1,3-Butanediol in Cerebral Ischemia. A Neurologic, Histologic, and Metabolic Study , 1987, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[14]  R. Duvoisin,et al.  Dopaminergic toxicity of rotenone and the 1-methyl-4-phenylpyridinium ion after their stereotaxic administration to rats: Implication for the mechanism of 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine toxicity , 1985, Neuroscience Letters.

[15]  G. Zelenock,et al.  Reduction of neurologic deficit by 1,3-butanediol induced ketosis in levine rats. , 1985, Stroke.

[16]  G. Zelenock,et al.  Butanediol Induced Cerebral Protection from Ischemic-Hypoxia in the Instrumented Levine Rat , 1984, Stroke.

[17]  C. Hackenbrock,et al.  Lateral diffusion of ubiquinone during electron transfer in phospholipid- and ubiquinone-enriched mitochondrial membranes. , 1982, The Journal of biological chemistry.

[18]  H. Kanaide,et al.  A protective effect of coenzyme Q10 on ischemia and reperfusion of the isolated perfused rat heart. , 1981, Journal of molecular and cellular cardiology.

[19]  N. Ruderman,et al.  Regulation of glucose and ketone-body metabolism in brain of anaesthetized rats. , 1974, The Biochemical journal.

[20]  G F Cahill,et al.  Brain metabolism during fasting. , 1967, The Journal of clinical investigation.