Ventricular-arterial and ventricular-ventricular interactions and their relevance to diastolic filling.
暂无分享,去创建一个
[1] Wolfgang A Linke,et al. Myocardial Structure and Function Differ in Systolic and Diastolic Heart Failure , 2006, Circulation.
[2] J. Tyberg,et al. Left Ventricular Pacing Minimizes Diastolic Ventricular Interaction, Allowing Improved Preload-Dependent Systolic Performance , 2004, Circulation.
[3] Yiming Wu,et al. Altered Titin Expression, Myocardial Stiffness, and Left Ventricular Function in Patients With Dilated Cardiomyopathy , 2004, Circulation.
[4] D. Kass,et al. Frequency- and Afterload-Dependent Cardiac Modulation In Vivo by Troponin I With Constitutively Active Protein Kinase A Phosphorylation Sites , 2004, Circulation research.
[5] I. Belenkie,et al. Opening the pericardium during pulmonary artery constriction improves cardiac function. , 2004, Journal of applied physiology.
[6] Marion L Greaser,et al. Titin isoform expression in normal and hypertensive myocardium. , 2003, Cardiovascular research.
[7] K. Schechtman,et al. Peak exercise stroke volume: associations with cardiac structure and diastolic function. , 2003, Journal of applied physiology.
[8] D. Kass,et al. Combined Ventricular Systolic and Arterial Stiffening in Patients With Heart Failure and Preserved Ejection Fraction: Implications for Systolic and Diastolic Reserve Limitations , 2003, Circulation.
[9] Ronglih Liao,et al. Titin Determines the Frank-Starling Relation in Early Diastole , 2003, The Journal of general physiology.
[10] J. Gardin,et al. Outcome of Congestive Heart Failure in Elderly Persons: Influence of Left Ventricular Systolic Function: The Cardiovascular Health Study , 2002, Annals of Internal Medicine.
[11] F. Prinzen,et al. Quantification of interventricular asynchrony during LBBB and ventricular pacing. , 2002, American journal of physiology. Heart and circulatory physiology.
[12] Yiming Wu,et al. Changes in Titin Isoform Expression in Pacing-Induced Cardiac Failure Give Rise to Increased Passive Muscle Stiffness , 2002, Circulation.
[13] R. Allman,et al. Correlates and outcomes of preserved left ventricular systolic function among older adults hospitalized with heart failure. , 2002, American heart journal.
[14] Siegfried Labeit,et al. Cardiac titin: an adjustable multi‐functional spring , 2002, The Journal of physiology.
[15] R. F. Kelly,et al. Utility of history, physical examination, electrocardiogram, and chest radiograph for differentiating normal from decreased systolic function in patients with heart failure. , 2002, The American journal of medicine.
[16] N. Sharma,et al. Clinical presentation, hospital length of stay, and readmission rate in patients with heart failure with preserved and decreased left ventricular systolic function , 2002, Clinical cardiology.
[17] I. Belenkie,et al. Ventricular interaction and external constraint account for decreased stroke work during volume loading in CHF. , 2001, American journal of physiology. Heart and circulatory physiology.
[18] M. Senni,et al. Heart failure with preserved systolic function. A different natural history? , 2001, Journal of the American College of Cardiology.
[19] A. Takeshita,et al. Mortality and readmission of hospitalized patients with congestive heart failure and preserved versus depressed systolic function. , 2001, The American journal of cardiology.
[20] W. Hundley,et al. Cardiac cycle-dependent changes in aortic area and distensibility are reduced in older patients with isolated diastolic heart failure and correlate with exercise intolerance. , 2001, Journal of the American College of Cardiology.
[21] J. Leiden,et al. Phosphorylation of Troponin I by Protein Kinase A Accelerates Relaxation and Crossbridge Cycle Kinetics in Mouse Ventricular Muscle , 2001, Circulation research.
[22] J. Gardin,et al. Importance of heart failure with preserved systolic function in patients ≥65 years of age , 2001 .
[23] E. Botvinick,et al. Ventricular contraction abnormalities in dilated cardiomyopathy: effect of biventricular pacing to correct interventricular dyssynchrony. , 2000, Journal of the American College of Cardiology.
[24] T Centner,et al. Differential expression of cardiac titin isoforms and modulation of cellular stiffness. , 2000, Circulation research.
[25] C. Rest,et al. Use of left ventricular pacing in heart failure: Evaluation by gated blood pool imaging , 1999, Journal of nuclear cardiology : official publication of the American Society of Nuclear Cardiology.
[26] T. Gillebert,et al. Afterload induced changes in myocardial relaxation: a mechanism for diastolic dysfunction. , 1999, Cardiovascular research.
[27] R. Pini,et al. Impact of arterial elastance as a measure of vascular load on left ventricular geometry in hypertension. , 1999, Journal of hypertension.
[28] D. Levy,et al. Congestive heart failure in subjects with normal versus reduced left ventricular ejection fraction: prevalence and mortality in a population-based cohort. , 1999, Journal of the American College of Cardiology.
[29] M. O'Rourke,et al. Isolated systolic hypertension, pulse pressure, and arterial stiffness as risk factors for cardiovascular disease , 1999, Current hypertension reports.
[30] C. H. Chen,et al. Verapamil acutely reduces ventricular-vascular stiffening and improves aerobic exercise performance in elderly individuals. , 1999, Journal of the American College of Cardiology.
[31] C H Chen,et al. Coupled systolic-ventricular and vascular stiffening with age: implications for pressure regulation and cardiac reserve in the elderly. , 1998, Journal of the American College of Cardiology.
[32] I. Belenkie,et al. Diastolic ventricular interaction in chronic heart failure , 1997, The Lancet.
[33] T. Gillebert,et al. Relaxation-systolic pressure relation. A load-independent assessment of left ventricular contractility. , 1997, Circulation.
[34] A. Cohen-Solal,et al. Effects of aging on left ventricular-arterial coupling in man: assessment by means of arterial effective and left ventricular elastances. , 1996, Journal of human hypertension.
[35] D. Kass,et al. Acute cardiovascular effects of OPC-18790 in patients with congestive heart failure. Time- and dose-dependence analysis based on pressure-volume relations. , 1996, Circulation.
[36] M. Frenneaux,et al. Determinants of exercise capacity in patients with coronary artery disease and mild to moderate systolic dysfunction. Role of heart rate and diastolic filling abnormalities. , 1996, European heart journal.
[37] J. Potter,et al. Cardiac troponin I phosphorylation increases the rate of cardiac muscle relaxation. , 1995, Circulation research.
[38] W L Maughan,et al. Contribution of External Forces to Left Ventricular Diastolic Pressure: Implications for the Clinical Use of the Starling Law , 1995, Annals of Internal Medicine.
[39] T. Irving,et al. Passive tension in cardiac muscle: contribution of collagen, titin, microtubules, and intermediate filaments. , 1995, Biophysical journal.
[40] W C Hunter,et al. Ventricular stroke work and efficiency both remain nearly optimal despite altered vascular loading. , 1993, The American journal of physiology.
[41] M. Starling,et al. Left ventricular-arterial coupling relations in the normal human heart. , 1993, American heart journal.
[42] W. Little,et al. Effect of exercise on left ventricular-arterial coupling assessed in the pressure-volume plane. , 1993, The American journal of physiology.
[43] D. Kass,et al. Effect of reduced aortic compliance on cardiac efficiency and contractile function of in situ canine left ventricle. , 1992, Circulation research.
[44] L. Hillis,et al. The natural history of isolated left ventricular diastolic dysfunction. , 1992, The American journal of medicine.
[45] R. Soufer,et al. Long-term outcome in patients with congestive heart failure and intact systolic left ventricular performance. , 1992, The American journal of cardiology.
[46] 達哉 佐々木. Molecular mechanism of regulation of Ca[2+] pump ATPase by phospholamban in cardiac sarcoplasmic reticulum , 1992 .
[47] M. Inui,et al. Molecular mechanism of regulation of Ca2+ pump ATPase by phospholamban in cardiac sarcoplasmic reticulum. Effects of synthetic phospholamban peptides on Ca2+ pump ATPase. , 1992, The Journal of biological chemistry.
[48] W. Little,et al. Left ventricular-arterial coupling in conscious dogs. , 1991, The American journal of physiology.
[49] D. Kitzman,et al. Exercise intolerance in patients with heart failure and preserved left ventricular systolic function: failure of the Frank-Starling mechanism. , 1991, Journal of the American College of Cardiology.
[50] S Sasayama,et al. Ventriculoarterial coupling in normal and failing heart in humans. , 1989, Circulation research.
[51] O. Hess,et al. Diastolic properties of the normal left ventricle during supine exercise. , 1988, British heart journal.
[52] E. Kranias,et al. Phosphorylation of C-protein, troponin I and phospholamban in isolated rabbit hearts. , 1988, The Biochemical journal.
[53] J. Mitchell,et al. The Effect of Pericardiectomy on Maximal Oxygen Consumption and Maximal Cardiac Output in Untrained Dogs , 1986, Circulation research.
[54] R. Magorien,et al. Hemodynamic correlates for timing intervals, ejection rate and filling rate derived from the radionuclide angiographic volume curve. , 1984, The American journal of cardiology.
[55] W L Maughan,et al. Left ventricular interaction with arterial load studied in isolated canine ventricle. , 1983, The American journal of physiology.
[56] A P Avolio,et al. Effects of aging on changing arterial compliance and left ventricular load in a northern Chinese urban community. , 1983, Circulation.
[57] J. Potter,et al. The effect of troponin I phosphorylation on the Ca2+-binding properties of the Ca2+-regulatory site of bovine cardiac troponin. , 1982, The Journal of biological chemistry.
[58] J S Janicki,et al. The pericardium and ventricular interaction, distensibility, and function. , 1980, The American journal of physiology.
[59] J. Weiss,et al. Evidence of incomplete left ventricular relaxation in the dog: prediction from the time constant for isovolumic pressure fall. , 1978, The Journal of clinical investigation.
[60] E. Braunwald,et al. Circulatory Effects of Acute Expansion of Blood Volume: Studies During Maximal Exercise and at Rest , 1966 .
[61] A. Katz. Editorial: The Descending Limb of the Starling Curve and the Failing Heart , 1965, Circulation.
[62] D. Kitzman. Diastolic dysfunction in the elderly. Genesis and diagnostic and therapeutic implications. , 2000, Cardiology clinics.
[63] S. Laurent,et al. Arterial alterations with aging and high blood pressure. A noninvasive study of carotid and femoral arteries. , 1993, Arteriosclerosis and thrombosis : a journal of vascular biology.
[64] J. Ross,et al. Afterload mismatch and preload reserve: a conceptual framework for the analysis of ventricular function. , 1976, Progress in cardiovascular diseases.