Guidelines for bronchoprovocation on the investigation of occupational asthma. Report of the Subcommittee on Bronchoprovocation for Occupational Asthma.
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J. Malo | L. Fabbri | I. Bernstein | F. Hargreave | P. Burge | J. Cohn | J. Salvaggio | A. Cartier | André Cartier | Jean-Luc Malo | R. McKay | Leonardo M. Fabbri | Roy T. McKay
[1] J. Pepys,et al. Bronchial provocation tests in etiologic diagnosis and analysis of asthma. , 2015, The American review of respiratory disease.
[2] J. Malo,et al. Acute exposure to sawdust does not alter airway calibre and responsiveness to histamine in asthmatic subjects. , 1988, European Respiratory Journal.
[3] J. Malo,et al. Occupational asthma in workers of a pharmaceutical company processing spiramycin. , 1988, Thorax.
[4] L. Fabbri,et al. Protective effect of antiasthma drugs on late asthmatic reactions and increased airway responsiveness induced by toluene diisocyanate in sensitized subjects. , 1987, The American review of respiratory disease.
[5] J. Malo,et al. Occupational asthma and IgE sensitization in a pharmaceutical company processing psyllium. , 1987, The American review of respiratory disease.
[6] J. Malo,et al. Exposure to a sensitizing occupational agent can cause a long-lasting increase in bronchial responsiveness to histamine in the absence of significant changes in airway caliber. , 1986, The Journal of allergy and clinical immunology.
[7] L. Fabbri,et al. Late, but not early, asthmatic reactions induced by toluene-diisocyanate are associated with increased airway responsiveness to methacholine. , 1986, European journal of respiratory diseases.
[8] M. Chan-yeung,et al. Occupational asthma caused by eastern white cedar (Thuja occidentalis) with demonstration that plicatic acid is present in this wood dust and is the causal agent. , 1986, The Journal of allergy and clinical immunology.
[9] B. Butcher,et al. Absence of hyperresponsiveness to methacholine in a worker with methylene diphenyl diisocyanate (MDI)-induced asthma. , 1986, Chest.
[10] H. Prince,et al. Antibody to human T-lymphotropic virus type III in wives of hemophiliacs. Evidence for heterosexual transmission. , 1985, Annals of internal medicine.
[11] J. Malo,et al. Occupational asthma in snow crab-processing workers. , 1984, The Journal of allergy and clinical immunology.
[12] J. Malo,et al. Monitoring of maximum expiratory peak flow rates and histamine inhalation tests in the investigation of occupational asthma , 1984, Clinical allergy.
[13] K. Venables,et al. 1 Clinical and Epidemiological Methods in Investigating Occupational Asthma , 1984, Clinics in Immunology and Allergy.
[14] D. Cockcroft,et al. Recurrent nocturnal asthma after bronchoprovocation with Western Red Cedar sawdust: association with acute increase in non‐allergic bronchial responsiveness , 1984, Clinical allergy.
[15] M. Chan-yeung,et al. Asthma caused by diphenylmethane diisocyanate in foundry workers. Clinical, bronchial provocation, and immunologic studies. , 1983, The American review of respiratory disease.
[16] J. Salvaggio,et al. Development and loss of toluene diisocyanate reactivity: immunologic, pharmacologic, and provocative challenge studies. , 1982, The Journal of allergy and clinical immunology.
[17] N. Thomson,et al. Bronchial responsiveness to histamine or methacholine in asthma: measurement and clinical significance. , 1981, The Journal of allergy and clinical immunology.
[18] J. Pepys. Challenge procedures in occupational asthma. , 1981, Bulletin of the New York Academy of Medicine.
[19] I. Bernstein,et al. Absence of airway hyperreactivity to methacholine in a worker sensitized to toluene diisocyanate (TDI). , 1980, Journal of occupational medicine. : official publication of the Industrial Medical Association.
[20] Laurie J. Smith,et al. Guidelines for the methodology of exercise challenge testing of asthmatics. Study Group on Exercise Challenge, Bronchoprovocation Committee, American Academy of Allergy. , 1979, The Journal of allergy and clinical immunology.
[21] H. Schwartz,et al. A proposed standardized method for bronchoprovocation tests in toluene diisocyanate--induced asthma. , 1979, The Journal of allergy and clinical immunology.
[22] B. Butcher. Inhalation challenge testing with toluene diisocyanate. , 1979, The Journal of allergy and clinical immunology.
[23] P. Burge,et al. Peak flow rate records in the diagnosis of occupational asthma due to isocyanates. , 1979, Thorax.
[24] H. Schwartz,et al. Patterns of airway reactivity to asthma produced by exposure to toluene di-isocyanate. , 1979, Chest.
[25] R. Davies,et al. Recurrent nocturnal asthma after exposure to grain dust. , 1976, The American review of respiratory disease.
[26] J. Salvaggio,et al. Toluene diisocyanate (TDI) pulmonary disease: immunologic and inhalation challenge studies. , 1976, The Journal of allergy and clinical immunology.
[27] J. Malo,et al. 305 Significant changes in non-specific bronchial responsiveness after isolated immediate, but not after late bronchospastic reactions , 1988 .
[28] R. McKay. Bronchoprovocation Challenge Testing in Occupational Airways Disorders , 1986 .
[29] J. Malo,et al. 263 Changes in airway responsiveness to histamine preceding fluctuations of bronchomotor tone in occupational asthma , 1985 .
[30] J. Malo,et al. 264 Occupational asthma due to azobisformamide , 1985 .
[31] N. Eiser,et al. Committee Report: Guidelines for Standardization of Bronchial Challenges with (Nonspecific) Bronchoconstricting Agents , 1985 .
[32] Burge Ps. Single and serial measurements of lung function in the diagnosis of occupational asthma. , 1982 .