Guidelines for bronchoprovocation on the investigation of occupational asthma. Report of the Subcommittee on Bronchoprovocation for Occupational Asthma.

[1]  J. Pepys,et al.  Bronchial provocation tests in etiologic diagnosis and analysis of asthma. , 2015, The American review of respiratory disease.

[2]  J. Malo,et al.  Acute exposure to sawdust does not alter airway calibre and responsiveness to histamine in asthmatic subjects. , 1988, European Respiratory Journal.

[3]  J. Malo,et al.  Occupational asthma in workers of a pharmaceutical company processing spiramycin. , 1988, Thorax.

[4]  L. Fabbri,et al.  Protective effect of antiasthma drugs on late asthmatic reactions and increased airway responsiveness induced by toluene diisocyanate in sensitized subjects. , 1987, The American review of respiratory disease.

[5]  J. Malo,et al.  Occupational asthma and IgE sensitization in a pharmaceutical company processing psyllium. , 1987, The American review of respiratory disease.

[6]  J. Malo,et al.  Exposure to a sensitizing occupational agent can cause a long-lasting increase in bronchial responsiveness to histamine in the absence of significant changes in airway caliber. , 1986, The Journal of allergy and clinical immunology.

[7]  L. Fabbri,et al.  Late, but not early, asthmatic reactions induced by toluene-diisocyanate are associated with increased airway responsiveness to methacholine. , 1986, European journal of respiratory diseases.

[8]  M. Chan-yeung,et al.  Occupational asthma caused by eastern white cedar (Thuja occidentalis) with demonstration that plicatic acid is present in this wood dust and is the causal agent. , 1986, The Journal of allergy and clinical immunology.

[9]  B. Butcher,et al.  Absence of hyperresponsiveness to methacholine in a worker with methylene diphenyl diisocyanate (MDI)-induced asthma. , 1986, Chest.

[10]  H. Prince,et al.  Antibody to human T-lymphotropic virus type III in wives of hemophiliacs. Evidence for heterosexual transmission. , 1985, Annals of internal medicine.

[11]  J. Malo,et al.  Occupational asthma in snow crab-processing workers. , 1984, The Journal of allergy and clinical immunology.

[12]  J. Malo,et al.  Monitoring of maximum expiratory peak flow rates and histamine inhalation tests in the investigation of occupational asthma , 1984, Clinical allergy.

[13]  K. Venables,et al.  1 Clinical and Epidemiological Methods in Investigating Occupational Asthma , 1984, Clinics in Immunology and Allergy.

[14]  D. Cockcroft,et al.  Recurrent nocturnal asthma after bronchoprovocation with Western Red Cedar sawdust: association with acute increase in non‐allergic bronchial responsiveness , 1984, Clinical allergy.

[15]  M. Chan-yeung,et al.  Asthma caused by diphenylmethane diisocyanate in foundry workers. Clinical, bronchial provocation, and immunologic studies. , 1983, The American review of respiratory disease.

[16]  J. Salvaggio,et al.  Development and loss of toluene diisocyanate reactivity: immunologic, pharmacologic, and provocative challenge studies. , 1982, The Journal of allergy and clinical immunology.

[17]  N. Thomson,et al.  Bronchial responsiveness to histamine or methacholine in asthma: measurement and clinical significance. , 1981, The Journal of allergy and clinical immunology.

[18]  J. Pepys Challenge procedures in occupational asthma. , 1981, Bulletin of the New York Academy of Medicine.

[19]  I. Bernstein,et al.  Absence of airway hyperreactivity to methacholine in a worker sensitized to toluene diisocyanate (TDI). , 1980, Journal of occupational medicine. : official publication of the Industrial Medical Association.

[20]  Laurie J. Smith,et al.  Guidelines for the methodology of exercise challenge testing of asthmatics. Study Group on Exercise Challenge, Bronchoprovocation Committee, American Academy of Allergy. , 1979, The Journal of allergy and clinical immunology.

[21]  H. Schwartz,et al.  A proposed standardized method for bronchoprovocation tests in toluene diisocyanate--induced asthma. , 1979, The Journal of allergy and clinical immunology.

[22]  B. Butcher Inhalation challenge testing with toluene diisocyanate. , 1979, The Journal of allergy and clinical immunology.

[23]  P. Burge,et al.  Peak flow rate records in the diagnosis of occupational asthma due to isocyanates. , 1979, Thorax.

[24]  H. Schwartz,et al.  Patterns of airway reactivity to asthma produced by exposure to toluene di-isocyanate. , 1979, Chest.

[25]  R. Davies,et al.  Recurrent nocturnal asthma after exposure to grain dust. , 1976, The American review of respiratory disease.

[26]  J. Salvaggio,et al.  Toluene diisocyanate (TDI) pulmonary disease: immunologic and inhalation challenge studies. , 1976, The Journal of allergy and clinical immunology.

[27]  J. Malo,et al.  305 Significant changes in non-specific bronchial responsiveness after isolated immediate, but not after late bronchospastic reactions , 1988 .

[28]  R. McKay Bronchoprovocation Challenge Testing in Occupational Airways Disorders , 1986 .

[29]  J. Malo,et al.  263 Changes in airway responsiveness to histamine preceding fluctuations of bronchomotor tone in occupational asthma , 1985 .

[30]  J. Malo,et al.  264 Occupational asthma due to azobisformamide , 1985 .

[31]  N. Eiser,et al.  Committee Report: Guidelines for Standardization of Bronchial Challenges with (Nonspecific) Bronchoconstricting Agents , 1985 .

[32]  Burge Ps Single and serial measurements of lung function in the diagnosis of occupational asthma. , 1982 .