Molecular and Subcellular Cardiology

ION CHANNELS AND PuMPS

[1]  R Fischmeister,et al.  Nitric oxide regulates cardiac Ca2+ current. Involvement of cGMP-inhibited and cGMP-stimulated phosphodiesterases through guanylyl cyclase activation. , 1993, The Journal of biological chemistry.

[2]  A. Fabiato,et al.  Time and calcium dependence of activation and inactivation of calcium- induced release of calcium from the sarcoplasmic reticulum of a skinned canine cardiac Purkinje cell , 1985, The Journal of general physiology.

[3]  F. Hofmann,et al.  cGMP‐dependent protein kinase decreases calcium sensitivity of skinned cardiac fibers , 1982, FEBS letters.

[4]  P. D. de Tombe,et al.  An internal viscous element limits unloaded velocity of sarcomere shortening in rat myocardium. , 1992 .

[5]  C W Balke,et al.  Local, stochastic release of Ca2+ in voltage‐clamped rat heart cells: visualization with confocal microscopy. , 1994, The Journal of physiology.

[6]  J. Wang,et al.  Tracer-diffusion in Liquids. IV. Self-diffusion of Calcium Ion and Chloride Ion in Aqueous Calcium Chloride Solutions1 , 1953 .

[7]  L. Xu,et al.  The ryanodine receptor-Ca2+ release channel complex of skeletal muscle sarcoplasmic reticulum. Evidence for a cooperatively coupled, negatively charged homotetramer. , 1989, The Journal of biological chemistry.

[8]  A. J. Brady,et al.  Mechanical properties of isolated cardiac myocytes. , 1991, Physiological reviews.

[9]  W. Wier,et al.  Intracellular calcium transients underlying the short‐term force‐interval relationship in ferret ventricular myocardium. , 1986, The Journal of physiology.

[10]  John M. Murray,et al.  Manifestations of Cooperative Behavior in the Regulated Actin Filament during Actin-Activated ATP Hydrolysis in the Presence of Calcium , 1973 .

[11]  L. Abbott,et al.  Modeling state-dependent inactivation of membrane currents. , 1994, Biophysical journal.

[12]  A. Huxley,et al.  Tension transients during steady shortening of frog muscle fibres. , 1985, The Journal of physiology.

[13]  R Fischmeister,et al.  Ca2+ current is regulated by cyclic GMP-dependent protein kinase in mammalian cardiac myocytes. , 1991, Proceedings of the National Academy of Sciences of the United States of America.

[14]  G. Meissner,et al.  Ryanodine activation and inhibition of the Ca2+ release channel of sarcoplasmic reticulum. , 1986, The Journal of biological chemistry.

[15]  A. Huxley.  Muscle structure and theories of contraction. , 1957, Progress in biophysics and biophysical chemistry.

[16]  K. Ono,et al.  Potentiation by cyclic GMP of beta‐adrenergic effect on Ca2+ current in guinea‐pig ventricular cells. , 1991, The Journal of physiology.

[17]  K. Edman,et al.  The contractile state of rabbit papillary muscle in relation to stimulation frequency. , 1976, The Journal of physiology.

[18]  A. Hodgkin,et al.  A quantitative description of membrane current and its application to conduction and excitation in nerve , 1990 .

[19]  L. E. Ford,et al.  Velocity transients and viscoelastic resistance to active shortening in cat papillary muscle. , 1982, Biophysical journal.

[20]  A. Badran,et al.  On the ultrastructure of cardiac muscle. , 1969, The Journal of the Egyptian Medical Association.

[21]  D. Brutsaert,et al.  Myocardial performance is modulated by interaction of cardiac endothelium derived nitric oxide and prostaglandins. , 1995, Cardiovascular research.

[22]  A Mebazaa,et al.  Right and left ventricular cultured endocardial endothelium produces prostacyclin and PGE2. , 1993, Journal of molecular and cellular cardiology.

[23]  Shaomeng Wang,et al.  Characterization of junctional and longitudinal sarcoplasmic reticulum from heart muscle. , 1988, The Journal of biological chemistry.

[24]  A. Huxley,et al.  The relation between stiffness and filament overlap in stimulated frog muscle fibres. , 1981, The Journal of physiology.

[25]  G. Meissner,et al.  Rapid calcium release from cardiac sarcoplasmic reticulum vesicles is dependent on Ca2+ and is modulated by Mg2+, adenine nucleotide, and calmodulin. , 1987, The Journal of biological chemistry.

[26]  S. Marom,et al.  State-dependent inactivation of the Kv3 potassium channel. , 1994, Biophysical journal.

[27]  E. Eisenberg,et al.  Evidence for cross-bridge attachment in relaxed muscle at low ionic strength. , 1982, Proceedings of the National Academy of Sciences of the United States of America.

[28]  H. Erickson,et al.  Structural and functional characterization of the purified cardiac ryanodine receptor-Ca2+ release channel complex. , 1989, The Journal of biological chemistry.

[29]  U Walter,et al.  Physiological role of cGMP and cGMP-dependent protein kinase in the cardiovascular system. , 1989, Reviews of physiology, biochemistry and pharmacology.

[30]  K. Wang,et al.  Architecture of the sarcomere matrix of skeletal muscle: immunoelectron microscopic evidence that suggests a set of parallel inextensible nebulin filaments anchored at the Z line , 1988, The Journal of cell biology.

[31]  J S Janicki,et al.  Ventricular remodeling in heart failure: the role of myocardial collagen. , 1995, Advances in experimental medicine and biology.

[32]  Donald M. Bers,et al.  Excitation-Contraction Coupling and Cardiac Contractile Force , 1993, Developments in Cardiovascular Medicine.

[33]  M. Stern,et al.  Theory of excitation-contraction coupling in cardiac muscle. , 1992, Biophysical journal.

[34]  A Mebazaa,et al.  Paracrine effects of endocardial endothelial cells on myocyte contraction mediated via endothelin. , 1993, The American journal of physiology.

[35]  W. Y. Cheung,et al.  Calmodulin plays a pivotal role in cellular regulation. , 1980, Science.

[36]  D M Bers,et al.  Intracellular Ca2+ transients during rapid cooling contractures in guinea‐pig ventricular myocytes. , 1989, The Journal of physiology.

[37]  Rodolphe Fischmeister,et al.  Cyclic GMP regulates the Ca-channel current in guinea pig ventricular myocytes , 1989, Pflügers Archiv - European Journal of Physiology.

[38]  M. Noble,et al.  The Diastolic Viscous Properties of Cat Papillary Muscle , 1977, Circulation research.

[39]  A. Huxley,et al.  Tension responses to sudden length change in stimulated frog muscle fibres near slack length , 1977, The Journal of physiology.

[40]  D L Morgan,et al.  Variation of muscle stiffness with tension during tension transients and constant velocity shortening in the frog. , 1981, The Journal of physiology.

[41]  F W Prinzen,et al.  Asymmetrical changes in ventricular wall mass by asynchronous electrical activation of the heart. , 1993, Advances in experimental medicine and biology.

[42]  J S Shiner,et al.  Calcium Requirements for Cardiac Myofibrillar Activation , 1974, Circulation research.

[43]  J. Beavo,et al.  Primary sequence of cyclic nucleotide phosphodiesterase isozymes and the design of selective inhibitors. , 1990, Trends in pharmacological sciences.

[44]  P Haugen,et al.  The stiffness under isotonic releases during a twitch of a frog muscle fibre. , 1988, Advances in experimental medicine and biology.

[45]  D M Bers,et al.  SR Ca loading in cardiac muscle preparations based on rapid-cooling contractures. , 1989, The American journal of physiology.

[46]  M. Simionescu,et al.  Calf cardiac valvular endothelial cells in culture: production of glycosaminoglycans, prostacyclin and fibronectin. , 1988, Journal of molecular and cellular cardiology.