Lack of association between PAX6/SOSTDC1/FAM20B gene polymorphisms and mesiodens
暂无分享,去创建一个
Jincheng Xu | Yongfeng Chen | S. Liu | Kai Zhang | Rongxiu Zhang | Jian-cheng Li | S. Liao | R. Tian
[1] Noriko Osumi,et al. The role of Pax6 in brain development and its impact on pathogenesis of autism spectrum disorder , 2019, Brain Research.
[2] P. Jagodziński,et al. GREM2 nucleotide variants and the risk of tooth agenesis. , 2018, Oral diseases.
[3] J. Dixon,et al. Structure and evolution of the Fam20 kinases , 2018, Nature Communications.
[4] Zhan-shan Zhang. Association between COX2 -765G/C polymorphism and periodontitis in Chinese population: a meta-analysis , 2018, BMC oral health.
[5] Jonghyun Shin,et al. Effects of mesiodens on adjacent permanent teeth: a retrospective study in Korean children based on cone‐beam computed tomography , 2018, International journal of paediatric dentistry.
[6] Tieliu Shi,et al. A functional SNP in the 3′‐UTR of TAP2 gene interacts with microRNA hsa‐miR‐1270 to suppress the gene expression , 2018, Environmental and molecular mutagenesis.
[7] A. Saâd,et al. Genetic study of non‐syndromic tooth agenesis through the screening of paired box 9, msh homeobox 1, axin 2, and Wnt family member 10A genes: a case‐series , 2018, European journal of oral sciences.
[8] H. Kim,et al. Genetic alterations in mesiodens as revealed by targeted next-generation sequencing and gene co-occurrence network analysis. , 2017, Oral diseases.
[9] H. Lim,et al. PAX6 aniridia syndrome: clinics, genetics, and therapeutics , 2017, Current opinion in ophthalmology.
[10] Shangfeng Liu,et al. The epidemiology of supernumerary teeth and the associated molecular mechanism , 2017, Organogenesis.
[11] G. Rutter,et al. The transcription factor Pax6 is required for pancreatic β cell identity, glucose-regulated ATP synthesis, and Ca2+ dynamics in adult mice , 2017, The Journal of Biological Chemistry.
[12] M. Nowicki,et al. Association of ENAM gene single nucleotide polymorphisms with dental caries in Polish children , 2016, Clinical Oral Investigations.
[13] D. Bai,et al. Inactivation of Fam20B in the dental epithelium of mice leads to supernumerary incisors. , 2015, European journal of oral sciences.
[14] T. Ho,et al. BMP-SHH signaling network controls epithelial stem cell fate via regulation of its niche in the developing tooth. , 2015, Developmental cell.
[15] D. Kennedy,et al. Clinical recommendations for management of mesiodens and unerupted permanent maxillary central incisors , 2014, European archives of paediatric dentistry : official journal of the European Academy of Paediatric Dentistry.
[16] A. Acevedo,et al. Dental anomalies in primary dentition and their corresponding permanent teeth , 2014, Clinical Oral Investigations.
[17] T. Çolak,et al. Investigation of prevalence and characteristics of mesiodens in a non-syndromic 11256 dental outpatients. , 2013, European review for medical and pharmacological sciences.
[18] Mervyn G. Thomas,et al. Autosomal-dominant nystagmus, foveal hypoplasia and presenile cataract associated with a novel PAX6 mutation , 2013, European Journal of Human Genetics.
[19] Sue Povey,et al. Nephrocalcinosis (Enamel Renal Syndrome) Caused by Autosomal Recessive FAM20A Mutations , 2013, Nephron Physiology.
[20] Z. Baqain,et al. FAM20A Mutations Can Cause Enamel-Renal Syndrome (ERS) , 2013, PLoS genetics.
[21] Hai-hua Lei,et al. PAX6 polymorphisms in 20 Chinese children with supernumerary teeth in the maxillary incisor area. , 2011, International journal of paediatric dentistry.
[22] Jeffrey C Murray,et al. Whole-Exome sequencing identifies FAM20A mutations as a cause of amelogenesis imperfecta and gingival hyperplasia syndrome. , 2011, American journal of human genetics.
[23] S. Juo,et al. A functional polymorphism at 3'UTR of the PAX6 gene may confer risk for extreme myopia in the Chinese. , 2011, Investigative ophthalmology & visual science.
[24] B. Sreedevi,et al. Survey of mesiodens and its characteristics in 2500 children of Davangere city, India. , 2010, European journal of paediatric dentistry.
[25] R. Krumlauf,et al. Inhibition of Wnt signaling by Wise (Sostdc1) and negative feedback from Shh controls tooth number and patterning , 2010, Development.
[26] G. Meighani,et al. Diagnosis and Management of Supernumerary (Mesiodens): A Review of the Literature , 2010, Journal of dentistry.
[27] Yusuke Nakamura,et al. Associations of functional NLRP3 polymorphisms with susceptibility to food-induced anaphylaxis and aspirin-induced asthma. , 2009, The Journal of allergy and clinical immunology.
[28] E. Järvinen,et al. Tinkering with the inductive mesenchyme: Sostdc1 uncovers the role of dental mesenchyme in limiting tooth induction , 2009, Development.
[29] K. Gündüz,et al. Mesiodens: a radiographic study in children. , 2008, Journal of oral science.
[30] R. Mesquita,et al. A survey of 460 supernumerary teeth in Brazilian children and adolescents. , 2008, International journal of paediatric dentistry.
[31] S. Iseki,et al. Study of Pax6 mutant rat revealed the association between upper incisor formation and midface formation , 2006, Developmental dynamics : an official publication of the American Association of Anatomists.
[32] Nobuyuki Itoh,et al. Regulation of Mammalian Tooth Cusp Patterning by Ectodin , 2005, Science.
[33] K. Eriksson,et al. A common variant upstream of the PAX6 gene influences islet function in man , 2011, Diabetologia.