Lack of association between PAX6/SOSTDC1/FAM20B gene polymorphisms and mesiodens

[1]  Noriko Osumi,et al.  The role of Pax6 in brain development and its impact on pathogenesis of autism spectrum disorder , 2019, Brain Research.

[2]  P. Jagodziński,et al.  GREM2 nucleotide variants and the risk of tooth agenesis. , 2018, Oral diseases.

[3]  J. Dixon,et al.  Structure and evolution of the Fam20 kinases , 2018, Nature Communications.

[4]  Zhan-shan Zhang Association between COX2 -765G/C polymorphism and periodontitis in Chinese population: a meta-analysis , 2018, BMC oral health.

[5]  Jonghyun Shin,et al.  Effects of mesiodens on adjacent permanent teeth: a retrospective study in Korean children based on cone‐beam computed tomography , 2018, International journal of paediatric dentistry.

[6]  Tieliu Shi,et al.  A functional SNP in the 3′‐UTR of TAP2 gene interacts with microRNA hsa‐miR‐1270 to suppress the gene expression , 2018, Environmental and molecular mutagenesis.

[7]  A. Saâd,et al.  Genetic study of non‐syndromic tooth agenesis through the screening of paired box 9, msh homeobox 1, axin 2, and Wnt family member 10A genes: a case‐series , 2018, European journal of oral sciences.

[8]  H. Kim,et al.  Genetic alterations in mesiodens as revealed by targeted next-generation sequencing and gene co-occurrence network analysis. , 2017, Oral diseases.

[9]  H. Lim,et al.  PAX6 aniridia syndrome: clinics, genetics, and therapeutics , 2017, Current opinion in ophthalmology.

[10]  Shangfeng Liu,et al.  The epidemiology of supernumerary teeth and the associated molecular mechanism , 2017, Organogenesis.

[11]  G. Rutter,et al.  The transcription factor Pax6 is required for pancreatic β cell identity, glucose-regulated ATP synthesis, and Ca2+ dynamics in adult mice , 2017, The Journal of Biological Chemistry.

[12]  M. Nowicki,et al.  Association of ENAM gene single nucleotide polymorphisms with dental caries in Polish children , 2016, Clinical Oral Investigations.

[13]  D. Bai,et al.  Inactivation of Fam20B in the dental epithelium of mice leads to supernumerary incisors. , 2015, European journal of oral sciences.

[14]  T. Ho,et al.  BMP-SHH signaling network controls epithelial stem cell fate via regulation of its niche in the developing tooth. , 2015, Developmental cell.

[15]  D. Kennedy,et al.  Clinical recommendations for management of mesiodens and unerupted permanent maxillary central incisors , 2014, European archives of paediatric dentistry : official journal of the European Academy of Paediatric Dentistry.

[16]  A. Acevedo,et al.  Dental anomalies in primary dentition and their corresponding permanent teeth , 2014, Clinical Oral Investigations.

[17]  T. Çolak,et al.  Investigation of prevalence and characteristics of mesiodens in a non-syndromic 11256 dental outpatients. , 2013, European review for medical and pharmacological sciences.

[18]  Mervyn G. Thomas,et al.  Autosomal-dominant nystagmus, foveal hypoplasia and presenile cataract associated with a novel PAX6 mutation , 2013, European Journal of Human Genetics.

[19]  Sue Povey,et al.  Nephrocalcinosis (Enamel Renal Syndrome) Caused by Autosomal Recessive FAM20A Mutations , 2013, Nephron Physiology.

[20]  Z. Baqain,et al.  FAM20A Mutations Can Cause Enamel-Renal Syndrome (ERS) , 2013, PLoS genetics.

[21]  Hai-hua Lei,et al.  PAX6 polymorphisms in 20 Chinese children with supernumerary teeth in the maxillary incisor area. , 2011, International journal of paediatric dentistry.

[22]  Jeffrey C Murray,et al.  Whole-Exome sequencing identifies FAM20A mutations as a cause of amelogenesis imperfecta and gingival hyperplasia syndrome. , 2011, American journal of human genetics.

[23]  S. Juo,et al.  A functional polymorphism at 3'UTR of the PAX6 gene may confer risk for extreme myopia in the Chinese. , 2011, Investigative ophthalmology & visual science.

[24]  B. Sreedevi,et al.  Survey of mesiodens and its characteristics in 2500 children of Davangere city, India. , 2010, European journal of paediatric dentistry.

[25]  R. Krumlauf,et al.  Inhibition of Wnt signaling by Wise (Sostdc1) and negative feedback from Shh controls tooth number and patterning , 2010, Development.

[26]  G. Meighani,et al.  Diagnosis and Management of Supernumerary (Mesiodens): A Review of the Literature , 2010, Journal of dentistry.

[27]  Yusuke Nakamura,et al.  Associations of functional NLRP3 polymorphisms with susceptibility to food-induced anaphylaxis and aspirin-induced asthma. , 2009, The Journal of allergy and clinical immunology.

[28]  E. Järvinen,et al.  Tinkering with the inductive mesenchyme: Sostdc1 uncovers the role of dental mesenchyme in limiting tooth induction , 2009, Development.

[29]  K. Gündüz,et al.  Mesiodens: a radiographic study in children. , 2008, Journal of oral science.

[30]  R. Mesquita,et al.  A survey of 460 supernumerary teeth in Brazilian children and adolescents. , 2008, International journal of paediatric dentistry.

[31]  S. Iseki,et al.  Study of Pax6 mutant rat revealed the association between upper incisor formation and midface formation , 2006, Developmental dynamics : an official publication of the American Association of Anatomists.

[32]  Nobuyuki Itoh,et al.  Regulation of Mammalian Tooth Cusp Patterning by Ectodin , 2005, Science.

[33]  K. Eriksson,et al.  A common variant upstream of the PAX6 gene influences islet function in man , 2011, Diabetologia.