Vessels in benign prostatic hyperplasia contain more binding sites for endostatin than vessels in normal prostate tissue.
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U. Engelmann | W. Bloch | A. Schmidt | K. Addicks | F. Sommer | T. Klotz | E. Ozgür
[1] R. Timpl,et al. The angiogenesis inhibitor endostatin impairs blood vessel maturation during wound healing , 2000, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[2] K. Kinzler,et al. Genes expressed in human tumor endothelium. , 2000, Science.
[3] R. Timpl,et al. Structural basis and potential role of heparin/heparan sulfate binding to the angiogenesis inhibitor endostatin , 1999, The EMBO journal.
[4] V. Sukhatme,et al. Endostatin Induces Endothelial Cell Apoptosis* , 1999, The Journal of Biological Chemistry.
[5] A. Friedl,et al. Differential binding of fibroblast growth factor-2 and -7 to basement membrane heparan sulfate: comparison of normal and abnormal human tissues. , 1997, The American journal of pathology.
[6] William Arbuthnot Sir Lane,et al. Endostatin: An Endogenous Inhibitor of Angiogenesis and Tumor Growth , 1997, Cell.
[7] J. Oesterling,et al. The prevalence of prostatism: a population-based survey of urinary symptoms. , 1993, The Journal of urology.
[8] M. Becich,et al. The relative proportion of stromal and epithelial hyperplasia is related to the development of symptomatic benign prostate hyperplasia. , 1992, The Journal of urology.
[9] P. Walsh,et al. The development of human benign prostatic hyperplasia with age. , 1984, The Journal of urology.