Human papillomavirus type 56 polymorphism in Canadian women with and without cervical lesions.

[1]  E. Franco,et al.  Human papillomavirus type 52 polymorphism and high‐grade lesions of the uterine cervix , 2013, International journal of cancer.

[2]  K. Kapila,et al.  Phylogenetic analysis of partial L1 gene of 10 human papillomavirus types isolated most commonly from women with normal and abnormal cervical cytology in Kuwait , 2013, Archives of Virology.

[3]  S. Franceschi,et al.  HPV16 genetic variation and the development of cervical cancer worldwide , 2012, British Journal of Cancer.

[4]  Chien-Jen Chen,et al.  Persistence of type-specific human papillomavirus infection and increased long-term risk of cervical cancer. , 2011, Journal of the National Cancer Institute.

[5]  Chien-Jen Chen,et al.  Unique variants of human papillomavirus genotypes 52 and 58 and risk of cervical neoplasia , 2011, International journal of cancer.

[6]  E. Franco,et al.  Distribution of human papillomavirus genotypes in cervical intraepithelial neoplasia and invasive cervical cancer in Canada , 2011, Journal of medical virology.

[7]  E. Franco,et al.  Determinants of human papillomavirus infection among Inuit women of northern Quebec, Canada. , 2010, Sexually transmitted diseases.

[8]  R. DeSalle,et al.  A population-based prospective study of carcinogenic human papillomavirus variant lineages, viral persistence, and cervical neoplasia. , 2010, Cancer research.

[9]  K. Straif,et al.  A review of human carcinogens--Part B: biological agents. , 2009, The Lancet. Oncology.

[10]  F. Thierry.  Transcriptional regulation of the papillomavirus oncogenes by cellular and viral transcription factors in cervical carcinoma. , 2009, Virology.

[11]  E. Franco,et al.  Prevalence and Age Distribution of Human Papillomavirus Infection in a Population of Inuit Women in Nunavik, Quebec , 2008, Cancer Epidemiology Biomarkers & Prevention.

[12]  L. Turek,et al.  The E8∧E2 Gene Product of Human Papillomavirus Type 16 Represses Early Transcription and Replication but Is Dispensable for Viral Plasmid Persistence in Keratinocytes , 2008, Journal of Virology.

[13]  S. Franceschi,et al.  Human papillomavirus type distribution in invasive cervical cancer and high‐grade cervical lesions: A meta‐analysis update , 2007, International journal of cancer.

[14]  G. Matlashewski,et al.  Human papillomavirus type 33 polymorphisms and high-grade squamous intraepithelial lesions of the uterine cervix. , 2006, The Journal of infectious diseases.

[15]  L. Laimins,et al.  Human papillomaviruses: basic mechanisms of pathogenesis and oncogenicity , 2006, Reviews in medical virology.

[16]  H. Bernard,et al.  Genome variation of human papillomavirus types: Phylogenetic and medical implications , 2006, International journal of cancer.

[17]  F. X. Bosch,et al.  Worldwide genomic diversity of the human papillomaviruses-53, 56, and 66, a group of high-risk HPVs unrelated to HPV-16 and HPV-18. , 2005, Virology.

[18]  R. DeSalle,et al.  Diversifying Selection in Human Papillomavirus Type 16 Lineages Based on Complete Genome Analyses , 2005, Journal of Virology.

[19]  W. G. Hubert.  Variant Upstream Regulatory Region Sequences Differentially Regulate Human Papillomavirus Type 16 DNA Replication throughout the Viral Life Cycle , 2005, Journal of Virology.

[20]  G. Matlashewski,et al.  The p53 codon 72 polymorphism and risk of high-grade cervical intraepithelial neoplasia. , 2005, Cancer detection and prevention.

[21]  C. Tremblay,et al.  Viral polymorphism in human papillomavirus types 33 and 35 and persistent and transient infection in the genital tract of women. , 2004, The Journal of infectious diseases.

[22]  C. Tremblay,et al.  Genomic polymorphism of human papillomavirus type 52 predisposes toward persistent infection in sexually active women. , 2004, The Journal of infectious diseases.

[23]  Claude Fauquet,et al.  Classification of papillomaviruses. , 2004, Virology.

[24]  Michelle S. Longworth,et al.  Pathogenesis of Human Papillomaviruses in Differentiating Epithelia , 2004, Microbiology and Molecular Biology Reviews.

[25]  Michal Abrahamowicz,et al.  The natural history of type-specific human papillomavirus infections in female university students. , 2003, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.

[26]  Allan Hildesheim,et al.  Chapter 5: Viral and host factors in human papillomavirus persistence and progression. , 2003, Journal of the National Cancer Institute. Monographs.

[27]  C. Wheeler,et al.  Herpes simplex virus type II is not a cofactor to human papillomavirus in cancer of the uterine cervix. , 2003, American journal of obstetrics and gynecology.

[28]  E. Franco,et al.  Persistent human papillomavirus infection and cervical neoplasia. , 2002, The Lancet. Oncology.

[29]  V. Fontaine,et al.  Inhibition of human papillomavirus‐16 long control region activity by interferon‐gamma overcome by p300 overexpression , 2001, Molecular carcinogenesis.

[30]  B. Rose,et al.  Variable Oncogene Promoter Activity of Human Papillomavirus Type 16 Cervical Cancer Isolates from Australia , 2001, Journal of Clinical Microbiology.

[31]  A. Giannoudis,et al.  Human papillomavirus variants and squamous neoplasia of the cervix , 2001, The Journal of pathology.

[32]  F. Coutlée,et al.  Prevalence of risk factors associated with human papillomavirus infection in women living with HIV. Canadian Women's HIV Study Group. , 1999, CMAJ : Canadian Medical Association journal = journal de l'Association medicale canadienne.

[33]  T. Heinemeyer,et al.  Databases on transcriptional regulation : TRANSFAC , TRRD and COMPEL , 1997 .

[34]  F. Coutlée,et al.  Comparison between vaginal tampon and cervicovaginal lavage specimen collection for detection of human papillomavirus DNA by the polymerase chain reaction , 1997, Journal of medical virology.

[35]  C. Desaintes,et al.  Control of papillomavirus DNA replication and transcription. , 1996, Seminars in cancer biology.

[36]  M. Yaniv,et al.  Control of HPV 18 DNA replication by cellular and viral transcription factors. , 1995, Nucleic acids research.

[37]  J. Thompson,et al.  CLUSTAL W: improving the sensitivity of progressive multiple sequence alignment through sequence weighting, position-specific gap penalties and weight matrix choice. , 1994, Nucleic acids research.

[38]  D. Pim,et al.  Characterization of the human papillomavirus E2 protein: evidence of trans‐activation and trans‐repression in cervical keratinocytes. , 1994, The EMBO journal.

[39]  H. Bernard,et al.  The human papillomavirus type 16 E2 transcription factor binds with low cooperativity to two flanking sites and represses the E6 promoter through displacement of Sp1 and TFIID , 1994, Journal of virology.

[40]  H. Delius,et al.  Primer-directed sequencing of human papillomavirus types. , 1994, Current topics in microbiology and immunology.