Endocytosis machinery is required for beta1-adrenergic receptor-induced hypertrophy in neonatal rat cardiac myocytes.
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S. Vatner | D. Vatner | J. Sadoshima | C. Morisco | C. Marrone | Jonathan Galeotti | D. Shao | Jonathan P. Galeotti
[1] S. Vatner,et al. Inhibition of p38α MAPK rescues cardiomyopathy induced by overexpressed β2-adrenergic receptor, but not β1-adrenergic receptor , 2007 .
[2] R. Lefkowitz,et al. Beta-arrestins and cell signaling. , 2007, Annual review of physiology.
[3] R. Lefkowitz,et al. β-Arrestins and Cell Signaling , 2007 .
[4] S. Vatner,et al. Inhibition of p38 alpha MAPK rescues cardiomyopathy induced by overexpressed beta 2-adrenergic receptor, but not beta 1-adrenergic receptor. , 2007, The Journal of clinical investigation.
[5] R. Lefkowitz,et al. Level of &bgr;-Adrenergic Receptor Kinase 1 Inhibition Determines Degree of Cardiac Dysfunction After Chronic Pressure Overload–Induced Heart Failure , 2005 .
[6] R. Lefkowitz,et al. Level of beta-adrenergic receptor kinase 1 inhibition determines degree of cardiac dysfunction after chronic pressure overload-induced heart failure. , 2005, Circulation.
[7] W. Koch,et al. G Protein–Coupled Receptor Internalization Signaling Is Required for Cardioprotection in Ischemic Preconditioning , 2004, Circulation research.
[8] P. Kang,et al. Akt/Protein Kinase B Promotes Organ Growth in Transgenic Mice , 2002, Molecular and Cellular Biology.
[9] P. Insel,et al. Receptor Number and Caveolar Co-localization Determine Receptor Coupling Efficiency to Adenylyl Cyclase* , 2001, The Journal of Biological Chemistry.
[10] R. Xiao. β-Adrenergic Signaling in the Heart: Dual Coupling of the β2-Adrenergic Receptor to Gs and Gi Proteins , 2001, Science's STKE.
[11] M. Caron,et al. Agonist-dependent Recruitment of Phosphoinositide 3-Kinase to the Membrane by β-Adrenergic Receptor Kinase 1 , 2001, The Journal of Biological Chemistry.
[12] J. Port,et al. Altered beta-adrenergic receptor gene regulation and signaling in chronic heart failure. , 2001, Journal of molecular and cellular cardiology.
[13] S. Vatner,et al. β-adrenergic cardiac hypertrophy is mediated primarily by the β1-subtype in the rat heart , 2001 .
[14] S. Vatner,et al. Beta-adrenergic cardiac hypertrophy is mediated primarily by the beta(1)-subtype in the rat heart. , 2001, Journal of molecular and cellular cardiology.
[15] M. Lisanti,et al. Differential targeting of beta -adrenergic receptor subtypes and adenylyl cyclase to cardiomyocyte caveolae. A mechanism to functionally regulate the cAMP signaling pathway. , 2000, The Journal of biological chemistry.
[16] M. Caron,et al. Differential Affinities of Visual Arrestin, βArrestin1, and βArrestin2 for G Protein-coupled Receptors Delineate Two Major Classes of Receptors* , 2000, The Journal of Biological Chemistry.
[17] G. Condorelli,et al. The Akt-Glycogen Synthase Kinase 3β Pathway Regulates Transcription of Atrial Natriuretic Factor Induced by β-Adrenergic Receptor Stimulation in Cardiac Myocytes* , 2000, The Journal of Biological Chemistry.
[18] R. Lefkowitz,et al. β-Arrestin1 Interacts with the Catalytic Domain of the Tyrosine Kinase c-SRC , 2000, The Journal of Biological Chemistry.
[19] S. Vatner,et al. beta-adrenergic receptor signaling: an acute compensatory adjustment-inappropriate for the chronic stress of heart failure? Insights from Gsalpha overexpression and other genetically engineered animal models. , 2000, Circulation research.
[20] M. Bristow. β-Adrenergic Receptor Blockade in Chronic Heart Failure , 2000 .
[21] M. Caron,et al. Differential affinities of visual arrestin, beta arrestin1, and beta arrestin2 for G protein-coupled receptors delineate two major classes of receptors. , 2000, The Journal of biological chemistry.
[22] R. Lefkowitz,et al. beta-arrestin1 interacts with the catalytic domain of the tyrosine kinase c-SRC. Role of beta-arrestin1-dependent targeting of c-SRC in receptor endocytosis. , 2000, The Journal of biological chemistry.
[23] M. Bristow. beta-adrenergic receptor blockade in chronic heart failure. , 2000, Circulation.
[24] Liaoyuan A. Hu,et al. Identification of the endophilins (SH3p4/p8/p13) as novel binding partners for the beta1-adrenergic receptor. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[25] D. Vaughan,et al. Augmenting β receptors in the heart: Short-term gains offset by long-term pains? , 1999 .
[26] M. Lohse,et al. Progressive hypertrophy and heart failure in beta1-adrenergic receptor transgenic mice. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[27] G. Dorn,et al. Low- and high-level transgenic expression of β2-adrenergic receptors differentially affect cardiac hypertrophy and function in Gαq-overexpressing mice , 1999 .
[28] F. Brodsky,et al. EGF Receptor Signaling Stimulates SRC Kinase Phosphorylation of Clathrin, Influencing Clathrin Redistribution and EGF Uptake , 1999, Cell.
[29] D. Vaughan,et al. Augmenting beta receptors in the heart: short-term gains offset by long-term pains? , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[30] G. Dorn,et al. Low- and high-level transgenic expression of beta2-adrenergic receptors differentially affect cardiac hypertrophy and function in Galphaq-overexpressing mice. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[31] J. Pessin,et al. Expression of a Dominant Interfering Dynamin Mutant in 3T3L1 Adipocytes Inhibits GLUT4 Endocytosis without Affecting Insulin Signaling* , 1998, The Journal of Biological Chemistry.
[32] J. Ross,et al. Expression of a beta-adrenergic receptor kinase 1 inhibitor prevents the development of myocardial failure in gene-targeted mice. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[33] J. Benovic,et al. Modulation of the Arrestin-Clathrin Interaction in Cells , 1997, The Journal of Biological Chemistry.
[34] R. Lefkowitz,et al. Clathrin-mediated Endocytosis of the β-Adrenergic Receptor Is Regulated by Phosphorylation/Dephosphorylation of β-Arrestin1* , 1997, The Journal of Biological Chemistry.
[35] M. Caron,et al. Phosphorylation and desensitization of the human beta 1-adrenergic receptor. Involvement of G protein-coupled receptor kinases and cAMP-dependent protein kinase. , 1995, The Journal of biological chemistry.
[36] R. Lefkowitz,et al. Cardiac function in mice overexpressing the beta-adrenergic receptor kinase or a beta ARK inhibitor. , 1995, Science.
[37] Julie A. Pitcher,et al. Pleckstrin Homology Domain-mediated Membrane Association and Activation of the -Adrenergic Receptor Kinase Requires Coordinate Interaction with G Subunits and Lipid(*) , 1995, The Journal of Biological Chemistry.
[38] R. Lefkowitz,et al. Enhanced myocardial function in transgenic mice overexpressing the beta 2-adrenergic receptor. , 1994, Science.
[39] P. Hawkins,et al. A novel phosphoinositide 3 kinase activity in myeloid-derived cells is activated by G protein βγ subunits , 1994, Cell.
[40] R. Lefkowitz,et al. Cellular expression of the carboxyl terminus of a G protein-coupled receptor kinase attenuates G beta gamma-mediated signaling. , 1994, The Journal of biological chemistry.
[41] P. Hawkins,et al. A novel phosphoinositide 3 kinase activity in myeloid-derived cells is activated by G protein beta gamma subunits. , 1994, Cell.
[42] X. J. Zhou,et al. Alpha- and beta-adrenergic stimulation of protein synthesis in cultured adult ventricular cardiomyocytes. , 1993, Journal of molecular and cellular cardiology.
[43] M. Bouvier,et al. Distinct regulation of beta 1- and beta 2-adrenergic receptors in Chinese hamster fibroblasts. , 1992, Molecular pharmacology.
[44] J. Karliner,et al. Concanavalin A amplifies both beta-adrenergic and muscarinic cholinergic receptor-adenylate cyclase-linked pathways in cardiac myocytes. , 1991, The Journal of clinical investigation.