MICA and NLRP3 gene polymorphisms interact synergistically affecting the risk of ankylosing spondylitis.
暂无分享,去创建一个
[1] R. Sánchez-Sánchez,et al. The interplay between HLA-B and NLRP3 polymorphisms may be associated with the genetic susceptibility of gout , 2022, Molecular Biology Reports.
[2] Xiangyu Wang,et al. Relationship Between IL1 Gene Polymorphism and Susceptibility to Ankylosing Spondylitis: An Updated and Supplemented Meta-Analysis , 2021, Biochemical Genetics.
[3] R. Sánchez-Sánchez,et al. A proposed HLA-B*27 screening method for ankylosing spondylitis detection based on tag-single nucleotide polymorphisms: a preliminary study , 2021, Molecular Biology Reports.
[4] Wangen Li,et al. NLRP3 Inflammasome: Checkpoint Connecting Innate and Adaptive Immunity in Autoimmune Diseases , 2021, Frontiers in Immunology.
[5] Joohee Jung,et al. NLRP3 Deficiency in Hepatocellular Carcinoma Enhances Surveillance of NK-92 through a Modulation of MICA/B , 2021, International journal of molecular sciences.
[6] Douglas J. Matthies,et al. Major Histocompatibility Complex Class I-Related Chain A (MICA) Allelic Variants Associate With Susceptibility and Prognosis of Gastric Cancer , 2021, Frontiers in Immunology.
[7] J. Rosenbaum,et al. Putative Pathobionts in HLA-B27-Associated Spondyloarthropathy , 2021, Frontiers in Immunology.
[8] M. Brown,et al. Inflammasome Activation in Ankylosing Spondylitis Is Associated With Gut Dysbiosis , 2021, Arthritis & rheumatology.
[9] I. Peláez-Ballestas,et al. Prevalence, demographics, and clinical characteristics of Latin American patients with spondyloarthritis , 2021, Advances in Rheumatology.
[10] L. Bi,et al. Role of the NLRP3 inflammasome in autoimmune diseases. , 2020, Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie.
[11] K. Bogunia-Kubik,et al. Association of MICA-129Met/Val polymorphism with clinical outcome of anti-TNF therapy and MICA serum levels in patients with rheumatoid arthritis , 2020, The Pharmacogenomics Journal.
[12] P. Emery,et al. Recent advances in ankylosing spondylitis: understanding the disease and management , 2018, F1000Research.
[13] Chen Zhao,et al. The association of NLRP3 and TNFRSF1A polymorphisms with risk of ankylosing spondylitis and treatment efficacy of etanercept , 2017, Journal of clinical laboratory analysis.
[14] M. Bicalho,et al. MICA and NKG2D: Is There an Impact on Kidney Transplant Outcome? , 2017, Front. Immunol..
[15] F. Eren,et al. Association of ERAP1, IL23R and PTGER4 Polymorphisms with Radiographic Severity of Ankylosing Spondylitis , 2017, The open rheumatology journal.
[16] Xiaodong Zhou,et al. Associations of MICA Polymorphisms with Inflammatory Rheumatic Diseases , 2015, The open rheumatology journal.
[17] U. Vogel,et al. Genetic Variations in Pattern Recognition Receptor Loci Are Associated with Anti-TNF Response in Patients with Rheumatoid Arthritis , 2015, PloS one.
[18] Jian-hua Xu,et al. Associations between ERAP1 polymorphisms and ankylosing spondylitis susceptibility: An updated meta-analysis , 2015, Modern rheumatology.
[19] Y. H. Lee,et al. Meta-analysis of the association between functional MICA-TM polymorphisms and systemic lupus erythematosus, rheumatoid arthritis and ankylosing spondylitis , 2015, Zeitschrift für Rheumatologie.
[20] J. Reveille. An update on the contribution of the MHC to as susceptibility , 2014, Clinical Rheumatology.
[21] P. Söderkvist,et al. Genetic variants in CARD8 but not in NLRP3 are associated with ankylosing spondylitis , 2013, Scandinavian journal of rheumatology.
[22] T. Merriman,et al. Prevalence of HLA-B27 in the New Zealand population: effect of age and ethnicity , 2013, Arthritis Research & Therapy.
[23] P. Scheet,et al. MICA, a gene contributing strong susceptibility to ankylosing spondylitis , 2013, Annals of the rheumatic diseases.
[24] Y. Lee,et al. The associations between interleukin-1 polymorphisms and susceptibility to ankylosing spondylitis: a meta-analysis. , 2012, Joint, bone, spine : revue du rhumatisme.
[25] M. Brown,et al. Is there a higher genetic load of susceptibility loci in familial ankylosing spondylitis? , 2012, Arthritis care & research.
[26] Paul Weston,et al. Interaction between ERAP1 and HLA-B27 in ankylosing spondylitis implicates peptide handling in the mechanism for HLA-B27 in disease susceptibility , 2011, Nature Genetics.
[27] T. Merriman,et al. Evidence of interaction of CARD8 rs2043211 with NALP3 rs35829419 in Crohn's disease , 2010, Genes and Immunity.
[28] M. Phipps,et al. MICA polymorphism: biology and importance in immunity and disease. , 2010, Trends in molecular medicine.
[29] Michael M. Ward,et al. Genome-wide association study of ankylosing spondylitis identifies non-MHC susceptibility loci , 2010, Nature Genetics.
[30] P. Donnelly,et al. Association scan of 14,500 nonsynonymous SNPs in four diseases identifies autoimmunity variants , 2007, Nature Genetics.
[31] Jason H. Moore,et al. Power of multifactor dimensionality reduction for detecting gene‐gene interactions in the presence of genotyping error, missing data, phenocopy, and genetic heterogeneity , 2003, Genetic epidemiology.
[32] C. Yee,et al. Tumour-derived soluble MIC ligands impair expression of NKG2D and T-cell activation , 2002, Nature.
[33] F. Martinon,et al. The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta. , 2002, Molecular cell.
[34] A. Cats,et al. Evaluation of diagnostic criteria for ankylosing spondylitis. A proposal for modification of the New York criteria. , 1984, Arthritis and rheumatism.
[35] Judith A. Smith. Update on Ankylosing Spondylitis: Current Concepts in Pathogenesis , 2014, Current Allergy and Asthma Reports.
[36] R. Elston,et al. Odds ratio based multifactor-dimensionality reduction method for detecting gene-gene interactions , 2007, Bioinform..