Anti‐IL‐33 antibody has a therapeutic effect in a murine model of allergic rhinitis

To cite this article: Kim YH, Yang TY, Park C‐S, Ahn S‐H, Son BK, Kim JH, Lim DH, Jang TY. Anti‐IL‐33 antibody has a therapeutic effect in a murine model of allergic rhinitis. Allergy 2012; 67: 183–190.

[1]  I. McInnes,et al.  Interleukin‐33: a novel mediator with a role in distinct disease pathologies , 2011, Journal of internal medicine.

[2]  C. Lloyd IL-33 family members and asthma - bridging innate and adaptive immune responses. , 2010, Current opinion in immunology.

[3]  Mingcai Li,et al.  Anti-IL-33 antibody treatment inhibits airway inflammation in a murine model of allergic asthma. , 2009, Biochemical and biophysical research communications.

[4]  F. Liew,et al.  The cytokine interleukin-33 mediates anaphylactic shock , 2009, Proceedings of the National Academy of Sciences.

[5]  K. Buckland,et al.  Resolution of allergic inflammation and airway hyperreactivity is dependent upon disruption of the T1/ST2-IL-33 pathway. , 2009, American journal of respiratory and critical care medicine.

[6]  C. Dahinden,et al.  Human basophils and eosinophils are the direct target leukocytes of the novel IL-1 family member IL-33. , 2009, Blood.

[7]  J. Girard,et al.  The IL-1-Like Cytokine IL-33 Is Constitutively Expressed in the Nucleus of Endothelial Cells and Epithelial Cells In Vivo: A Novel ‘Alarmin’? , 2008, PloS one.

[8]  M. Teixeira,et al.  IL-33 Induces Antigen-Specific IL-5+ T Cells and Promotes Allergic-Induced Airway Inflammation Independent of IL-41 , 2008, The Journal of Immunology.

[9]  K. Ohta,et al.  Interleukin-33 enhances adhesion, CD11b expression and survival in human eosinophils , 2008, Laboratory Investigation.

[10]  H. Kita,et al.  A novel IL-1 family cytokine, IL-33, potently activates human eosinophils. , 2008, The Journal of allergy and clinical immunology.

[11]  J. Fujimoto,et al.  Administration of IL-33 induces airway hyperresponsiveness and goblet cell hyperplasia in the lungs in the absence of adaptive immune system. , 2008, International immunology.

[12]  S. Dreskin,et al.  Anti-inflammatory effect of thymoquinone in a mouse model of allergic lung inflammation. , 2006, International immunopharmacology.

[13]  J Fernando Bazan,et al.  IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines. , 2005, Immunity.

[14]  Y. Sugiyama,et al.  Expression and function of the ST2 gene in a murine model of allergic airway inflammation , 2002, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.

[15]  J. Denburg,et al.  Pathogenesis of Murine Experimental Allergic Rhinitis: A Study of Local and Systemic Consequences of IL-5 Deficiency1 , 2002, The Journal of Immunology.

[16]  M. Hayakawa,et al.  Tissue distribution and subcellular localization of a variant form of the human ST2 gene product, ST2V. , 2001, Biochemical and biophysical research communications.

[17]  F. Finkelman,et al.  The other side of the coin: the protective role of the TH2 cytokines. , 2001, The Journal of allergy and clinical immunology.

[18]  C. Meisel,et al.  Crucial Role of the Interleukin 1 Receptor Family Member T1/St2 in T Helper Cell Type 2–Mediated Lung Mucosal Immune Responses , 1999, The Journal of experimental medicine.

[19]  K. Dabbagh,et al.  IL-4 induces mucin gene expression and goblet cell metaplasia in vitro and in vivo. , 1999, Journal of immunology.

[20]  A. Kataura,et al.  Topical antigen provocation increases the number of immunoreactive IL-4-, IL-5- and IL-6-positive cells in the nasal mucosa of patients with perennial allergic rhinitis. , 1997, International archives of allergy and immunology.

[21]  R. Pawankar,et al.  Nasal mast cells in perennial allergic rhinitics exhibit increased expression of the Fc epsilonRI, CD40L, IL-4, and IL-13, and can induce IgE synthesis in B cells. , 1997, The Journal of clinical investigation.

[22]  P. Foster,et al.  Interleukin 5 deficiency abolishes eosinophilia, airways hyperreactivity, and lung damage in a mouse asthma model , 1996, The Journal of experimental medicine.

[23]  P. Howarth,et al.  Immunolocalization of cytokines in the nasal mucosa of normal and perennial rhinitic subjects. The mast cell as a source of IL-4, IL-5, and IL-6 in human allergic mucosal inflammation. , 1993, Journal of immunology.

[24]  R. de Waal Malefyt,et al.  IL-13 induces proliferation and differentiation of human B cells activated by the CD40 ligand. , 1993, International immunology.

[25]  W. Paul,et al.  The presence of interleukin 4 during in vitro priming determines the lymphokine-producing potential of CD4+ T cells from T cell receptor transgenic mice , 1992, The Journal of experimental medicine.

[26]  S. Durham,et al.  Cytokine messenger RNA expression for IL-3, IL-4, IL-5, and granulocyte/macrophage-colony-stimulating factor in the nasal mucosa after local allergen provocation: relationship to tissue eosinophilia. , 1992, Journal of immunology.