Thymic selection by a single MHC/peptide ligand: autoreactive T cells are low-affinity cells.
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J. Sprent | C. Surh | B. Ernst | D. Lee | C. Ahn | C. Ahn | Dong-sup Lee | Bettina Ernst
[1] K. Oshimi,et al. Differential Effects of Anti-Fas Ligand and Anti-Tumor Necrosis Factor α Antibodies on Acute Graft-Versus-Host Disease Pathologies , 1998 .
[2] M. Bevan,et al. T-cell selection. , 1998, Current opinion in immunology.
[3] H. Waldmann,et al. How do monoclonal antibodies induce tolerance? A role for infectious tolerance? , 1998, Annual review of immunology.
[4] L. Picker,et al. Independent regulation of cutaneous lymphocyte-associated antigen expression and cytokine synthesis phenotype during human CD4+ memory T cell differentiation. , 1997, Journal of immunology.
[5] C. Janeway,et al. The imprint of intrathymic self-peptides on the mature T cell receptor repertoire. , 1997, Immunity.
[6] A. Rudensky,et al. Deficient positive selection of CD4 T cells in mice displaying altered repertoires of MHC class II-bound self-peptides. , 1997, Immunity.
[7] C. Benoist,et al. Selection of a broad repertoire of CD4+ T cells in H-2Ma0/0 mice. , 1997, Immunity.
[8] J. Sprent,et al. Thymic selection by a single MHC/peptide ligand produces a semidiverse repertoire of CD4+ T cells. , 1997, Immunity.
[9] M. Bevan,et al. In thymic selection, peptide diversity gives and takes away. , 1997, Immunity.
[10] P. Marrack,et al. Positive selection of thymocytes bearing αβ T cell receptors , 1997 .
[11] M. Utsuyama,et al. Positive and negative CD4+ thymocyte selection by a single MHC class II/peptide ligand affected by its expression level in the thymus. , 1997, Immunity.
[12] C. Benoist,et al. Positive selection of T cells: fastidious or promiscuous? , 1997, Current opinion in immunology.
[13] E. Podack,et al. Graft-versus-host-disease-associated lymphoid hypoplasia and B cell dysfunction is dependent upon donor T cell-mediated Fas-ligand function, but not perforin function. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[14] P. A. Peterson,et al. Antigen Presentation and T Cell Development in H2-M-Deficient Mice , 1996, Science.
[15] C. Benoist,et al. Mice Lacking H2-M Complexes, Enigmatic Elements of the MHC Class II Peptide-Loading Pathway , 1996, Cell.
[16] H. Ruley,et al. H2-M Mutant Mice Are Defective in the Peptide Loading of Class II Molecules, Antigen Presentation, and T Cell Repertoire Selection , 1996, Cell.
[17] P. Marrack,et al. The Repertoire of T Cells Shaped by a Single MHC/Peptide Ligand , 1996, Cell.
[18] J. Tschopp,et al. Cytotoxic T cells deficient in both functional fas ligand and perforin show residual cytolytic activity yet lose their capacity to induce lethal acute graft-versus-host disease , 1996, The Journal of experimental medicine.
[19] B. Fowlkes,et al. Positive selection of T cells. , 1995, Current opinion in immunology.
[20] P. Fink,et al. Positive selection of thymocytes. , 1995, Annual review of immunology.
[21] J. Sprent,et al. Profound atrophy of the bone marrow reflecting major histocompatibility complex class II-restricted destruction of stem cells by CD4+ cells , 1994, The Journal of experimental medicine.
[22] Kristin A. Hogquist,et al. T cell receptor antagonist peptides induce positive selection , 1994, Cell.
[23] J. Sprent,et al. Tolerance of CD8+ T cells developing in parent-->F1 chimeras prepared with supralethal irradiation: step-wise induction of tolerance in the intrathymic and extrathymic environments , 1993, The Journal of experimental medicine.
[24] J. Sprent,et al. Two subsets of epithelial cells in the thymic medulla , 1992, The Journal of experimental medicine.
[25] A. Singer,et al. Cellular basis of skin allograft rejection: an in vivo model of immune-mediated tissue destruction. , 1992, Annual review of immunology.
[26] E. Berg,et al. The cutaneous lymphocyte antigen is a skin lymphocyte homing receptor for the vascular lectin endothelial cell-leukocyte adhesion molecule 1 , 1991, The Journal of experimental medicine.
[27] A. Singer,et al. Specific prolongation of MHC class II disparate skin allografts by in vivo administration of anti-IFN-gamma monoclonal antibody. , 1990, Journal of immunology.
[28] J. Sprent,et al. Role of T cell subsets in lethal graft-versus-host disease (GVHD) directed to class I versus class II H-2 differences. II. Protective effects of L3T4+ cells in anti-class II GVHD. , 1990, Journal of immunology.
[29] J. Sprent,et al. Strong T cell tolerance in parent----F1 bone marrow chimeras prepared with supralethal irradiation. Evidence for clonal deletion and anergy , 1990, The Journal of experimental medicine.
[30] H. Boehmer,et al. Developmental biology of T cells in T cell-receptor transgenic mice. , 1990 .
[31] A. Singer,et al. Rejection of skin allografts by CD4+ T cells is antigen-specific and requires expression of target alloantigen on Ia- epidermal cells. , 1989, Journal of immunology.
[32] J. Sprent,et al. T Cell Selection in the Thymus , 1988, Immunological reviews.
[33] P. Vassalli,et al. Tumor necrosis factor/cachectin is an effector of skin and gut lesions of the acute phase of graft-vs.-host disease , 1987, The Journal of experimental medicine.
[34] R. Palmiter,et al. Transgenic mice selectively lacking MHC class II (I-E) antigen expression on B cells: An in vivo approach to investigate la gene function , 1987, Cell.
[35] A. Singer,et al. Phenotype, specificity, and function of T cell subsets and T cell interactions involved in skin allograft rejection , 1987, The Journal of experimental medicine.
[36] P. Marrack,et al. T cell tolerance by clonal elimination in the thymus , 1987, Cell.
[37] B. B. Mishell,et al. Selected Methods in Cellular Immunology , 1980 .
[38] M. Bevan. In a radiation chimaera, host H–2 antigens determine immune responsiveness of donor cytotoxic cells , 1977, Nature.
[39] W. Lapp,et al. Graft-versus-host induced immunosuppression: depressed T cell helper function in vitro. , 1976, Cellular immunology.
[40] M. Simonsen. Graft versus host reactions. Their natural history, and applicability as tools of research. , 1962, Progress in allergy.