Carnosic acid attenuates apoptosis induced by amyloid-β 1–42 or 1–43 in SH-SY5Y human neuroblastoma cells
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K. Itoh | T. Matsumiya | F. Xing | Ryo Hayakari | T. Imaizumi | H. Yoshida | K. Tanji | I. Takahashi | J. Mimura | S. Kawaguchi | Kunio Kosaka | Hiroshi Tanaka | K. Tsuruga | Liang Wang | Pengfei Meng
[1] K. Itoh,et al. Carnosic acid suppresses the production of amyloid-β 1-42 and 1-43 by inducing an α-secretase TACE/ADAM17 in U373MG human astrocytoma cells , 2014, Neuroscience Research.
[2] S. Ovsepian,et al. Drain of the brain: low-affinity p75 neurotrophin receptor affords a molecular sink for clearance of cortical amyloid β by the cholinergic modulator system , 2013, Neurobiology of Aging.
[3] Lan Ma,et al. Modeling heterogeneous responsiveness of intrinsic apoptosis pathway , 2013, BMC Systems Biology.
[4] H. Rupasinghe,et al. Polyphenols: Multipotent Therapeutic Agents in Neurodegenerative Diseases , 2013, Oxidative medicine and cellular longevity.
[5] Bhaswati Banerjee,et al. Caspases: A Potential Therapeutic Targets in the Treatment of Alzheimer's Disease , 2013 .
[6] B. Winblad,et al. The Pathogenic Aβ43 Is Enriched in Familial and Sporadic Alzheimer Disease , 2013, PloS one.
[7] K. Itoh,et al. Carnosic acid suppresses the production of amyloid-β 1–42 by inducing the metalloprotease gene TACE/ADAM17 in SH-SY5Y human neuroblastoma cells , 2013, Neuroscience Research.
[8] O. Arancio,et al. Caspase-9 mediates synaptic plasticity and memory deficits of Danish dementia knock-in mice: caspase-9 inhibition provides therapeutic protection , 2012, Molecular Neurodegeneration.
[9] W. Klein. Synaptotoxic amyloid-β oligomers: a molecular basis for the cause, diagnosis, and treatment of Alzheimer's disease? , 2012, Journal of Alzheimer's disease : JAD.
[10] F. Cecconi,et al. Caspase-3 in the central nervous system: beyond apoptosis , 2012, Trends in Neurosciences.
[11] A. Kakita,et al. p62/sequestosome 1 binds to TDP‐43 in brains with frontotemporal lobar degeneration with TDP‐43 inclusions , 2012, Journal of neuroscience research.
[12] Rajesh S. Omtri,et al. Differences in the cellular uptake and intracellular itineraries of amyloid beta proteins 40 and 42: ramifications for the Alzheimer's drug discovery. , 2012, Molecular pharmaceutics.
[13] Morgan Sheng,et al. Caspases in synaptic plasticity , 2012, Molecular Brain.
[14] V. de Laurenzi,et al. Role of Apoptosis in disease , 2012, Aging.
[15] W. Klein,et al. Different β-amyloid oligomer assemblies in Alzheimer brains correlate with age of disease onset and impaired cholinergic activity , 2012, Neurobiology of Aging.
[16] W. Klein,et al. The Aβ oligomer hypothesis for synapse failure and memory loss in Alzheimer’s disease , 2011, Neurobiology of Learning and Memory.
[17] M. Modo,et al. Targeting the Nrf2–Keap1 antioxidant defence pathway for neurovascular protection in stroke , 2011, The Journal of physiology.
[18] C. Broeckhoven,et al. Potent amyloidogenicity and pathogenicity of Aβ43 , 2011, Nature Neuroscience.
[19] K. Itoh,et al. Nrf2 regulates NGF mRNA induction by carnosic acid in T98G glioblastoma cells and normal human astrocytes. , 2011, Journal of biochemistry.
[20] T. Dawson,et al. Poly(ADP-Ribose) (PAR) Binding to Apoptosis-Inducing Factor Is Critical for PAR Polymerase-1–Dependent Cell Death (Parthanatos) , 2011, Science Signaling.
[21] K. Itoh,et al. Edaravone and carnosic acid synergistically enhance the expression of nerve growth factor in human astrocytes under hypoxia/reoxygenation , 2011, Neuroscience Research.
[22] J. McLaurin,et al. Mechanisms of Amyloid-Beta Peptide Uptake by Neurons: The Role of Lipid Rafts and Lipid Raft-Associated Proteins , 2010, International journal of Alzheimer's disease.
[23] D. Linseman,et al. Nutraceutical Antioxidants as Novel Neuroprotective Agents , 2010, Molecules.
[24] Gopal Thinakaran,et al. Membrane rafts in Alzheimer's disease beta-amyloid production. , 2010, Biochimica et biophysica acta.
[25] C. Schengrund. Lipid rafts: Keys to neurodegeneration , 2010, Brain Research Bulletin.
[26] W. Klein,et al. A Mouse Model of Amyloid β Oligomers: Their Contribution to Synaptic Alteration, Abnormal Tau Phosphorylation, Glial Activation, and Neuronal Loss In Vivo , 2010, The Journal of Neuroscience.
[27] T. Rohn. The role of caspases in Alzheimer’s disease; potential novel therapeutic opportunities , 2010, Apoptosis.
[28] B. Winblad,et al. Aβ43 is more frequent than Aβ40 in amyloid plaque cores from Alzheimer disease brains , 2009, Journal of neurochemistry.
[29] H. Tanila,et al. Nuclear factor erythroid 2-related factor 2 protects against beta amyloid , 2008, Molecular and Cellular Neuroscience.
[30] F. Baas,et al. Oligomer-specific Abeta toxicity in cell models is mediated by selective uptake. , 2008, Biochimica et biophysica acta.
[31] H. Chun,et al. Beneficial effects of carnosic acid on dieldrin-induced dopaminergic neuronal cell death , 2008, Neuroreport.
[32] R. Resende,et al. Neurotoxic effect of oligomeric and fibrillar species of amyloid-beta peptide 1-42: Involvement of endoplasmic reticulum calcium release in oligomer-induced cell death , 2008, Neuroscience.
[33] T. Terasaki,et al. Cerebral clearance of human amyloid‐β peptide (1–40) across the blood–brain barrier is reduced by self‐aggregation and formation of low‐density lipoprotein receptor‐related protein‐1 ligand complexes , 2007, Journal of neurochemistry.
[34] T. Iwatsubo,et al. Immunoreactivity of phage library-derived human single-chain antibodies to amyloid beta conformers in vitro. , 2007, Journal of biochemistry.
[35] Kim N. Green,et al. Intracellular amyloid-β in Alzheimer's disease , 2007, Nature Reviews Neuroscience.
[36] S. Elmore. Apoptosis: A Review of Programmed Cell Death , 2007, Toxicologic pathology.
[37] D. Dickson,et al. Filling the Gaps in the Aβ Cascade Hypothesis of Alzheimers Disease , 2006 .
[38] A. Yoshimura,et al. ER stress-induced apoptosis and caspase-12 activation occurs downstream of mitochondrial apoptosis involving Apaf-1 , 2006, Journal of Cell Science.
[39] G. Bu,et al. Efficient transfer of receptor-associated protein (RAP) across the blood-brain barrier , 2004, Journal of Cell Science.
[40] D. Dickson. Apoptotic mechanisms in Alzheimer neurofibrillary degeneration: cause or effect? , 2004, The Journal of clinical investigation.
[41] T. Kudo,et al. Involvement of caspase-4 in endoplasmic reticulum stress-induced apoptosis and Aβ-induced cell death , 2004, The Journal of cell biology.
[42] M. Kirkitadze,et al. Amyloid β-protein (Aβ) assembly: Aβ40 and Aβ42 oligomerize through distinct pathways , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[43] David S. Park,et al. Apoptosis-inducing factor is involved in the regulation of caspase-independent neuronal cell death , 2002, The Journal of cell biology.
[44] F. Checler,et al. The caspase‐derived C‐terminal fragment of βAPP induces caspase‐independent toxicity and triggers selective increase of Aβ42 in mammalian cells , 2001, Journal of neurochemistry.
[45] D. Selkoe. Alzheimer's disease: genes, proteins, and therapy. , 2001, Physiological reviews.
[46] Mark P. Mattson,et al. Apoptosis in neurodegenerative disorders , 2000, Nature Reviews Molecular Cell Biology.
[47] Junying Yuan,et al. Apoptosis in the nervous system , 2000, Nature.
[48] Junying Yuan,et al. Caspase-12 mediates endoplasmic-reticulum-specific apoptosis and cytotoxicity by amyloid-β , 2000, Nature.
[49] C. Cotman,et al. Neuronal Apoptosis Induced by β-Amyloid Is Mediated by Caspase-8 , 1999, Neurobiology of Disease.
[50] P. Rakic,et al. The role of cell death in regulating the size and shape of the mammalian forebrain. , 1999, Cerebral cortex.
[51] David Smith,et al. Involvement of Caspases in Proteolytic Cleavage of Alzheimer’s Amyloid-β Precursor Protein and Amyloidogenic Aβ Peptide Formation , 1999, Cell.
[52] J. Morris,et al. Tangles and plaques in nondemented aging and “preclinical” Alzheimer's disease , 1999, Annals of neurology.
[53] G. de Murcia,et al. Importance of Poly(ADP-ribose) Polymerase and Its Cleavage in Apoptosis , 1998, The Journal of Biological Chemistry.
[54] E. Mandelkow,et al. Tau in Alzheimer's disease. , 1998, Trends in cell biology.
[55] Y. Lazebnik,et al. Caspases: enemies within. , 1998, Science.
[56] B. Margolis,et al. The X11α Protein Slows Cellular Amyloid Precursor Protein Processing and Reduces Aβ40 and Aβ42 Secretion* , 1998, The Journal of Biological Chemistry.
[57] Keisuke Kuida,et al. Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice , 1996, Nature.
[58] Shai Shaham,et al. The C. elegans cell death gene ced-3 encodes a protein similar to mammalian interleukin-1β-converting enzyme , 1993, Cell.
[59] Masahiko S. Satoh,et al. Role of poly(ADP-ribose) formation in DNA repair , 1992, Nature.
[60] K. Itoh,et al. Role of Nrf2 and p62/ZIP in the neurite outgrowth by carnosic acid in PC12h cells. , 2010, Journal of biochemistry.
[61] M. Kirkitadze,et al. Molecular mechanisms initiating amyloid beta-fibril formation in Alzheimer's disease. , 2005, Acta biochimica Polonica.
[62] D. Butterfield. Amyloid beta-peptide (1-42)-induced oxidative stress and neurotoxicity: implications for neurodegeneration in Alzheimer's disease brain. A review. , 2002, Free radical research.