GSTM1 and APE1 genotypes affect arsenic-induced oxidative stress: a repeated measures study

[1]  C. Tseng,et al.  Arsenic Methylation, Urinary Arsenic Metabolites and Human Diseases: Current Perspective , 2007, Journal of environmental science and health. Part C, Environmental carcinogenesis & ecotoxicology reviews.

[2]  M. Bates,et al.  Please Scroll down for Article Journal of Toxicology and Environmental Health, Part A , 2022 .

[3]  K. Hemminki,et al.  Association of DNA repair polymorphisms with DNA repair functional outcomes in healthy human subjects. , 2006, Carcinogenesis.

[4]  Louis W. Chang,et al.  Effects of arsenic exposure among semiconductor workers: a cautionary note on urinary 8-oxo-7,8-dihydro-2'-deoxyguanosine. , 2006, Free radical biology & medicine.

[5]  L. Hudson,et al.  Inorganic arsenic compounds cause oxidative damage to DNA and protein by inducing ROS and RNS generation in human keratinocytes , 2005, Molecular and Cellular Biochemistry.

[6]  Thomas J. Smith,et al.  Toenail Arsenic Concentrations, GSTT1 Gene Polymorphisms, and Arsenic Exposure from Drinking Water , 2005, Cancer Epidemiology Biomarkers & Prevention.

[7]  S. Okada,et al.  The role of trivalent dimethylated arsenic in dimethylarsinic acid-promoted skin and lung tumorigenesis in mice: tumor-promoting action through the induction of oxidative stress. , 2005, Toxicology letters.

[8]  Xinhua Liu,et al.  Urinary Creatinine and Arsenic Metabolism , 2005, Environmental health perspectives.

[9]  M. Evans,et al.  DNA repair is responsible for the presence of oxidatively damaged DNA lesions in urine. , 2005, Mutation research.

[10]  F. Parl.  Glutathione S-transferase genotypes and cancer risk. , 2005, Cancer letters.

[11]  T. Yoshimura,et al.  Chronic arsenic exposure and urinary 8-Hydroxy-2′-deoxyguanosine in an arsenic-affected area in Inner Mongolia, China , 2005, Journal of Exposure Analysis and Environmental Epidemiology.

[12]  Zhongwen Wang,et al.  Immunohistochemical analysis of oxidative DNA damage in arsenic-related human skin samples from arsenic-contaminated area of China. , 2004, Cancer letters.

[13]  M. Carless,et al.  The GSTM1 null genotype confers an increased risk for solar keratosis development in an Australian Caucasian population. , 2002, The Journal of investigative dermatology.

[14]  H. Rüdiger,et al.  Urinary excretion of 8-hydroxy-2'-deoxyguanosine measured by high-performance liquid chromatography with electrochemical detection. , 2002, Journal of chromatography. B, Analytical technologies in the biomedical and life sciences.

[15]  M. Evans,et al.  Urinary 8-oxo-2'-deoxyguanosine: redox regulation of DNA repair in vivo? , 2002, Free radical biology & medicine.

[16]  F. Stagnitti,et al.  Arsenic contamination in Bangladesh groundwater: A major environmental and social disaster , 2002, International journal of environmental health research.

[17]  S. Toyokuni,et al.  Comparison between high-performance liquid chromatography and enzyme-linked immunosorbent assay for the determination of 8-hydroxy-2'-deoxyguanosine in human urine. , 2002, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.

[18]  C. Hopenhayn-Rich,et al.  Evidence for induction of oxidative stress caused by chronic exposure of Chinese residents to arsenic contained in drinking water. , 2002, Environmental health perspectives.

[19]  D. Christiani,et al.  Differential association of the codon 72 p53 and GSTM1 polymorphisms on histological subtype of non-small cell lung carcinoma. , 2001, Cancer research.

[20]  Y. Hsueh,et al.  Association of blood arsenic levels with increased reactive oxidants and decreased antioxidant capacity in a human population of northeastern Taiwan. , 2001, Environmental health perspectives.

[21]  T. Rossman,et al.  Arsenite is a cocarcinogen with solar ultraviolet radiation for mouse skin: an animal model for arsenic carcinogenesis. , 2001, Toxicology and applied pharmacology.

[22]  A. Mutti,et al.  Polymorphism of quinone-metabolizing enzymes and susceptibility to ozone-induced acute effects. , 2001, American journal of respiratory and critical care medicine.

[23]  A. Collins,et al.  Glutathione S-transferase polymorphisms influence the level of oxidative DNA damage and antioxidant protection in humans. , 2001, Mutation research.

[24]  Alexander Pilger,et al.  Longitudinal study of urinary 8-hydroxy-2′-deoxyguanosine excretion in healthy adults , 2001, Free radical research.

[25]  R. Strange,et al.  Detoxifying enzyme genotypes and susceptibility to cutaneous malignancy , 2000, The British journal of dermatology.

[26]  S. Flora.  ARSENIC‐INDUCED OXIDATIVE STRESS AND ITS REVERSIBILITY FOLLOWING COMBINED ADMINISTRATION OF N‐ACETYLCYSTEINE AND MESO 2,3–DIMERCAPTOSUCCINIC ACID IN RATS , 1999, Clinical and experimental pharmacology & physiology.

[27]  D. Christiani,et al.  Determination of total arsenic concentrations in nails by inductively coupled plasma mass spectrometry , 1999, Biological Trace Element Research.

[28]  Y. Hsueh,et al.  Urinary levels of inorganic and organic arsenic metabolites among residents in an arseniasis-hyperendemic area in Taiwan. , 1998, Journal of toxicology and environmental health. Part A.

[29]  R. Sram.  Effect of glutathione S-transferase M1 polymorphisms on biomarkers of exposure and effects. , 1998, Environmental health perspectives.

[30]  T. Rebbeck.  Molecular epidemiology of the human glutathione S-transferase genotypes GSTM1 and GSTT1 in cancer susceptibility. , 1997, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.

[31]  Y. Hsueh,et al.  Arsenic methylation capacity, body retention, and null genotypes of glutathione S-transferase M1 and T1 among current arsenic-exposed residents in Taiwan. , 1997, Mutation research.

[32]  H. Ochi,et al.  Quantitative immunohistochemical determination of 8-hydroxy-2'-deoxyguanosine by a monoclonal antibody N45.1: its application to ferric nitrilotriacetate-induced renal carcinogenesis model. , 1997, Laboratory investigation; a journal of technical methods and pathology.

[33]  A. Collins,et al.  Oxidative DNA damage in human cells: the influence of antioxidants and DNA repair. , 1997, Biochemical Society transactions.

[34]  F. Díaz-Barriga,et al.  Arsenic increased lipid peroxidation in rat tissues by a mechanism independent of glutathione levels. , 1995, Environmental health perspectives.

[35]  A. Grollman,et al.  Insertion of specific bases during DNA synthesis past the oxidation-damaged base 8-oxodG , 1991, Nature.

[36]  H. Pardue,et al.  Kinetic study of the Jaffé reaction for quantifying creatinine in serum: 2. Evaluation of buffered reagent and comparison of different data-processing options. , 1989, Clinical chemistry.

[37]  Xianglin Shi,et al.  Oxidative mechanism of arsenic toxicity and carcinogenesis , 2004, Molecular and Cellular Biochemistry.

[38]  Xianglin Shi,et al.  Molecular mechanisms of arsenic carcinogenesis , 2004, Molecular and Cellular Biochemistry.

[39]  A. Barchowsky,et al.  Arsenic induces oxidant stress and NF-kappa B activation in cultured aortic endothelial cells. , 1996, Free radical biology & medicine.

[40]  J. Tobin.  Estimation of Relationships for Limited Dependent Variables , 1958 .