Lifetime history of alcohol consumption and K‐ras mutations in pancreatic ductal adenocarcinoma
暂无分享,去创建一个
N. Malats | A. Carrato | F. Real | M. Crous-Bou | M. Porta | L. Guarner | Tomás López | Eva Morales | M. Jariod | J. Rifà
[1] A. Carrato,et al. Past medical conditions and K-ras mutations in pancreatic ductal adenocarcinoma: a hypothesis-generating study , 2009, Cancer Causes & Control.
[2] F. Real,et al. Pancreatic cancer development and progression: remodeling the model. , 2008, Gastroenterology.
[3] M. Omary,et al. Epidemiology of alcohol-related liver and pancreatic disease in the United States. , 2008, Archives of internal medicine.
[4] Manal M. Hassan,et al. Risk Factors for Pancreatic Cancer: Case-Control Study , 2007, The American Journal of Gastroenterology.
[5] F. Stickel,et al. “Allgemeine und systematische Pharmakologie und Toxikologie” WS 18/19 Seminarthema: Chemische Kanzerogenese , 2017 .
[6] J. Abbruzzese,et al. Lifestyle, Occupational, and Reproductive Factors in Relation to Pancreatic Cancer Risk , 2007, Pancreas.
[7] N. Malats,et al. Lifetime History of Tobacco Consumption and K-ras Mutations in Exocrine Pancreatic Cancer , 2007, Pancreas.
[8] N. Malats,et al. Food and nutrient intakes and K-ras mutations in exocrine pancreatic cancer , 2007, Journal of Epidemiology and Community Health.
[9] M. Otsuki,et al. 4. Chronic pancreatitis and pancreatic cancer, lifestyle-related diseases. , 2007, Internal medicine.
[10] H. Friess,et al. Update on pancreatic cancer and alcohol‐associated risk , 2006, Journal of gastroenterology and hepatology.
[11] Qing Zhang,et al. Differing molecular pathology of pancreatic adenocarcinoma in Egyptian and United States patients , 2006, International journal of cancer.
[12] P. A. van den Brandt,et al. Alcohol and the risk of colon and rectal cancer with mutations in the K-ras gene. , 2006, Alcohol.
[13] P. Maisonneuve,et al. Epidemiology and risk factors for pancreatic cancer. , 2006, Best practice & research. Clinical gastroenterology.
[14] S. Hecht,et al. Identification of an acetaldehyde adduct in human liver DNA and quantitation as N2-ethyldeoxyguanosine. , 2006, Chemical research in toxicology.
[15] N. Malats,et al. Exocrine pancreatic cancer: Symptoms at presentation and their relation to tumour site and stage , 2005, Clinical & translational oncology : official publication of the Federation of Spanish Oncology Societies and of the National Cancer Institute of Mexico.
[16] V. Go,et al. Alcohol and pancreatic cancer. , 2005, Alcohol.
[17] P. Rosenbaum. The Case‐Only Odds Ratio as a Causal Parameter , 2004, Biometrics.
[18] Xiang‐Dong Wang,et al. Alcohol and cancer: genetic and nutritional aspects , 2004, Proceedings of the Nutrition Society.
[19] N. Malats,et al. Validity of the hospital discharge diagnosis in epidemiologic studies of biliopancreatic pathology , 2000, European Journal of Epidemiology.
[20] N. Malats,et al. Medical Conditions in Patients with Pancreatic and Biliary Diseases (Validity and Agreement Between Data from Questionnaires and Medical Records) , 1999, Digestive Diseases and Sciences.
[21] N. Malats,et al. Occupational exposure to dyes, metals, polycyclic aromatic hydrocarbons and other agents and K‐ras activation in human exocrine pancreatic cancer , 2003, International journal of cancer.
[22] J. Cameron,et al. Multicomponent Analysis of the Pancreatic Adenocarcinoma Progression Model Using a Pancreatic Intraepithelial Neoplasia Tissue Microarray , 2003, Modern Pathology.
[23] M. Salaspuro. Alcohol consumption and cancer of the gastrointestinal tract. , 2003, Best practice & research. Clinical gastroenterology.
[24] M. Barbacid,et al. RAS oncogenes: the first 30 years , 2003, Nature Reviews Cancer.
[25] D. Purdie. Statistical Methods in Medical Research, 4th edn , 2003 .
[26] R. DePinho,et al. Pancreatic cancer biology and genetics , 2002, Nature Reviews Cancer.
[27] H. Adami,et al. Alcohol abuse and the risk of pancreatic cancer , 2002, Gut.
[28] N. Malats,et al. Family history of cancer and germline BRCA2 mutations in sporadic exocrine pancreatic cancer , 2002, Gut.
[29] T. Sarkola,et al. Ethanol, acetaldehyde, acetate, and lactate levels after alcohol intake in white men and women: effect of 4-methylpyrazole. , 2002, Alcoholism, clinical and experimental research.
[30] N. Malats,et al. Generalizing molecular results arising from incomplete biological samples: expected bias and unexpected findings. , 2002, Annals of epidemiology.
[31] N. Malats,et al. Occupational exposure to organic solvents and K-ras mutations in exocrine pancreatic cancer. , 2002, Carcinogenesis.
[32] F. Bosch,et al. Increased rectal cell proliferation following alcohol abuse , 2001, Gut.
[33] W. Willett,et al. Coffee and alcohol consumption and the risk of pancreatic cancer in two prospective United States cohorts. , 2001, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.
[34] X. Estivill,et al. Cystic fibrosis transmembrane regulator ( CFTR ) (cid:196) F508 mutation and 5T allele in patients with chronic pancreatitis and exocrine pancreatic cancer , 2000 .
[35] M. Leppert,et al. Associations between dietary intake and Ki-ras mutations in colon tumors: a population-based study. , 2000, Cancer research.
[36] N. Malats,et al. Coffee, pancreatic cancer, and K-rasmutations: updating the research agenda , 2000, Journal of epidemiology and community health.
[37] N. Malats,et al. Serum concentrations of organochlorine compounds and K-ras mutations in exocrine pancreatic cancer , 1999, The Lancet.
[38] N. Malats,et al. Association between coffee drinking and K-ras mutations in exocrine pancreatic cancer. PANKRAS II Study Group. , 1999, Journal of epidemiology and community health.
[39] N. Malats,et al. Human cancer, carcinogenic exposures and mutation spectra. , 1999, Mutation research.
[40] P. Tolbert,et al. Patterns of genetic alterations in pancreatic cancer: A pooled analysis , 1999, Environmental and molecular mutagenesis.
[41] N. Malats,et al. Learning from case reports: diagnostic issues in an epidemiologic study of pancreatic cancer. , 1998, Journal of clinical epidemiology.
[42] N. Malats,et al. Occurrence, trends and environment etiology of pancreatic cancer. , 1998, Scandinavian journal of work, environment & health.
[43] N. Malats,et al. Ki‐ras mutations in exocrine pancreatic cancer: Association with clinico‐pathological characteristics and with tobacco and alcohol consumption , 1997, International journal of cancer.
[44] E. Fernández,et al. Concordancia entre la información facilitada por el paciente y un familiar sobre antecedentes, patológicos, consumo de tabaco, de alcohol, de café, y dieta en el cáncer de páncreas exocrino y del sistema biliar extrahepático , 1995 .
[45] N. Malats,et al. Diagnostic certainty and potential for misclassification in exocrine pancreatic cancer. PANKRAS I Project Investigations. , 1994, Journal of clinical epidemiology.
[46] R. Russell,et al. Enhancement of ethanol induced rectal mucosal hyper regeneration with age in F344 rats. , 1994, Gut.
[47] C B Begg,et al. Statistical analysis of molecular epidemiology studies employing case-series. , 1994, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.
[48] Jack A. Taylor,et al. Non-hierarchical logistic models and case-only designs for assessing susceptibility in population-based case-control studies. , 1994, Statistics in medicine.
[49] M. Berger,et al. Possible role of acetaldehyde in ethanol-related rectal cocarcinogenesis in the rat. , 1990, Gastroenterology.
[50] Martin Bland,et al. An Introduction to Medical Statistics , 1987 .
[51] Sander Greenland,et al. Modern Epidemiology 3rd edition , 1986 .