Hepatocellular hypoxia-induced vascular endothelial growth factor expression and angiogenesis in experimental biliary cirrhosis.

[1]  M. Phillips,et al.  Perinodular arteriolar plexus in liver cirrhosis. Scanning electron microscopy of microvascular casts. , 2008, Liver.

[2]  L. Aiello,et al.  Hypoxia induces vascular endothelial growth factor gene and protein expression in cultured rat islet cells. , 1998, Diabetes.

[3]  S. Friedman,et al.  Coordinated induction of VEGF receptors in mesenchymal cell types during rat hepatic wound healing , 1998, Oncogene.

[4]  T. Nabika,et al.  Clinical significance of microvessel density and vascular endothelial growth factor expression in hepatocellular carcinoma and surrounding liver: Possible involvement of vascular endothelial growth factor in the angiogenesis of cirrhotic liver , 1998, Hepatology.

[5]  A. Karsan,et al.  Fibroblast growth factor-2 inhibits endothelial cell apoptosis by Bcl-2-dependent and independent mechanisms. , 1997, The American journal of pathology.

[6]  J. Boyer,et al.  Extrahepatic biliary obstruction impairs microvascular perfusion and increases leukocyte adhesion in rat liver , 1997, Hepatology.

[7]  Y. Itoyama,et al.  Rapid induction of vascular endothelial growth factor gene expression after transient middle cerebral artery occlusion in rats. , 1997, Stroke.

[8]  P. Desmond,et al.  Sequential increases in the intrahepatic expression of epidermal growth factor, basic fibroblast growth factor, and transforming growth factor β in a bile duct ligated rat model of cirrhosis , 1997, Hepatology.

[9]  A. Harris,et al.  Hypoxia-inducible factor-1 modulates gene expression in solid tumors and influences both angiogenesis and tumor growth. , 1997, Proceedings of the National Academy of Sciences of the United States of America.

[10]  F. Bayard,et al.  Extracellular Cleavage of the Vascular Endothelial Growth Factor 189-Amino Acid Form by Urokinase Is Required for Its Mitogenic Effect* , 1997, The Journal of Biological Chemistry.

[11]  G. Arteel,et al.  Chronic enteral ethanol treatment causes hypoxia in rat liver tissue in vivo , 1997, Hepatology.

[12]  M. Arthur,et al.  The plasminogen‐activating system in hepatic stellate cells , 1996, Hepatology.

[13]  E. Abel Clinical applications of research on angiogenesis , 1996 .

[14]  G. Semenza,et al.  Activation of vascular endothelial growth factor gene transcription by hypoxia-inducible factor 1 , 1996, Molecular and cellular biology.

[15]  M. Goldberg,et al.  Post-transcriptional Regulation of Vascular Endothelial Growth Factor by Hypoxia (*) , 1996, The Journal of Biological Chemistry.

[16]  J. Villeneuve,et al.  The hepatic microcirculation in the isolated perfused human liver , 1996, Hepatology.

[17]  C. Kevil,et al.  Translational enhancement of FGF-2 by eIF-4 factors, and alternate utilization of CUG and AUG codons for translation initiation. , 1995, Oncogene.

[18]  G. Arteel,et al.  Evidence that hypoxia markers detect oxygen gradients in liver: pimonidazole and retrograde perfusion of rat liver. , 1995, British Journal of Cancer.

[19]  M. Neeman,et al.  Stabilization of vascular endothelial growth factor mRNA by hypoxia and hypoglycemia and coregulation with other ischemia-induced genes , 1995, Molecular and cellular biology.

[20]  M. Goldberg,et al.  Transcriptional Regulation of the Rat Vascular Endothelial Growth Factor Gene by Hypoxia (*) , 1995, The Journal of Biological Chemistry.

[21]  H. Dvorak,et al.  Vascular permeability factor/vascular endothelial growth factor, microvascular hyperpermeability, and angiogenesis. , 1995, The American journal of pathology.

[22]  J. Haratake,et al.  Alterations in bile ducts and peribiliary microcirculation in rats after common bile duct ligation , 1995, Hepatology.

[23]  D. Rifkin,et al.  Differential modulation of cell phenotype by different molecular weight forms of basic fibroblast growth factor: possible intracellular signaling by the high molecular weight forms , 1995, The Journal of cell biology.

[24]  D. Marmé,et al.  Hypoxia-induced transcription of the vascular endothelial growth factor gene is independent of functional AP-1 transcription factor. , 1995, Biochemical and biophysical research communications.

[25]  K. Alitalo,et al.  Vascular endothelial growth factor is induced in response to transforming growth factor-beta in fibroblastic and epithelial cells. , 1994, The Journal of biological chemistry.

[26]  W. Meyers,et al.  Transforming growth factor‐β1 and mannose 6‐phosphate/insulin‐like growth factor‐II receptor expression during intrahepatic bile duct hyperplasia and biliary fibrosis in the rat , 1994, Hepatology.

[27]  R. Koos,et al.  Vascular endothelial growth factor/vascular permeability factor expression in the rat uterus: rapid stimulation by estrogen correlates with estrogen-induced increases in uterine capillary permeability and growth. , 1993, Endocrinology.

[28]  D. Dhumeaux,et al.  Immunolocalization of heparinbinding growth factors (HBGF) types 1 and 2 in rat liver. Selective hyperexpression of HBGF‐2 in carbon tetrachloride‐induced fibrosis , 1993, The Journal of pathology.

[29]  E. Bauer,et al.  Vascular endothelial growth factor induces interstitial collagenase expression in human endothelial cells , 1992, Journal of cellular physiology.

[30]  E. Keshet,et al.  Vascular endothelial growth factor induced by hypoxia may mediate hypoxia-initiated angiogenesis , 1992, Nature.

[31]  L. Orci,et al.  Vascular endothelial growth factor (VEGF) induces plasminogen activators and plasminogen activator inhibitor-1 in microvascular endothelial cells. , 1991, Biochemical and biophysical research communications.

[32]  D. Morgan,et al.  Therapeutic implications of impaired hepatic oxygen diffusion in chronic liver disease , 1991, Hepatology.

[33]  D. Connolly,et al.  Effects of a variety of cytokines and inducing agents on vascular permeability factor mRNA levels in U937 cells. , 1991, Biochemical and biophysical research communications.

[34]  M. Hisaoka,et al.  Morphological changes of hepatic microcirculation in experimental rat cirrhosis: A scanning electron microscopic study , 1991, Hepatology.

[35]  H. Prats,et al.  Alternative initiation of translation determines cytoplasmic or nuclear localization of basic fibroblast growth factor , 1991, Molecular and cellular biology.

[36]  D. Connolly,et al.  Vascular permeability factor: a tumor-derived polypeptide that induces endothelial cell and monocyte procoagulant activity, and promotes monocyte migration , 1990, The Journal of experimental medicine.

[37]  J. Lélias,et al.  High molecular mass forms of basic fibroblast growth factor are initiated by alternative CUG codons. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[38]  L. Sarkozi,et al.  Biliary physiology in rats with bile ductular cell hyperplasia. Evidence for a secretory function of proliferated bile ductules. , 1988, The Journal of clinical investigation.

[39]  F Varin,et al.  Hepatic microcirculation in the perfused cirrhotic rat liver. , 1985, The Journal of clinical investigation.

[40]  Joe W. Gray,et al.  The Molecular Basis of Cancer , 1985 .

[41]  J. Villeneuve,et al.  Assessment of liver microcirculation in human cirrhosis. , 1982, The Journal of clinical investigation.

[42]  E. Brogi,et al.  Indirect angiogenic cytokines upregulate VEGF and bFGF gene expression in vascular smooth muscle cells, whereas hypoxia upregulates VEGF expression only. , 1994, Circulation.

[43]  D. Moscatelli,et al.  The FGF family of growth factors and oncogenes. , 1992, Advances in cancer research.

[44]  Roche ResearchCenter,et al.  Vascular Permeability Factor: A Tumor-derived Polypeptide that Induces Endothelial Cell and Monocyte Procoagulant Activity, and Promotes Monocyte Migration , 1990 .