Inhibitor of growth 1 (ING1) acts at early steps of multiple DNA repair pathways
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[1] A. Baniahmad,et al. The ING tumor suppressors in cellular senescence and chromatin , 2011, Cell & Bioscience.
[2] M. Narita,et al. The tumor suppressor ING1 contributes to epigenetic control of cellular senescence , 2011, Aging cell.
[3] Gang Li,et al. Tumour suppressor ING1b maintains genomic stability upon replication stress , 2011, Nucleic acids research.
[4] Y. Takano,et al. ING Genes Work as Tumor Suppressor Genes in the Carcinogenesis of Head and Neck Squamous Cell Carcinoma , 2010, Journal of oncology.
[5] A. Benguría,et al. Regulation of the microRNA processor DGCR8 by the tumor suppressor ING1. , 2010, Cancer research.
[6] A. von Deimling,et al. The inhibitor of growth 1 (ING1) is involved in trichostatin A-induced apoptosis and caspase 3 signaling in p53-deficient glioblastoma cells. , 2009, Oncology Research.
[7] T. Kutateladze,et al. Structural insight into histone recognition by the ING PHD fingers. , 2009, Current drug targets.
[8] P. Muñoz,et al. Chromatin dynamics coupled to DNA repair , 2009, Epigenetics.
[9] Andrew H Coles,et al. The ING gene family in the regulation of cell growth and tumorigenesis , 2009, Journal of cellular physiology.
[10] E. Brambilla,et al. The new tumor suppressor genes ING: Genomic structure and status in cancer , 2008, International journal of cancer.
[11] S. Nozell,et al. The ING4 Tumor Suppressor Attenuates NF-κB Activity at the Promoters of Target Genes , 2008, Molecular and Cellular Biology.
[12] T. Hung,et al. Histone H3K4me3 binding is required for the DNA repair and apoptotic activities of ING1 tumor suppressor. , 2008, Journal of molecular biology.
[13] Gang Li,et al. Phosphorylation of the tumor suppressor p33ING1b at Ser‐126 influences its protein stability and proliferation of melanoma cells , 2007, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[14] K. Riabowol,et al. After a decade of study-ING, a PHD for a versatile family of proteins. , 2007, Trends in biochemical sciences.
[15] M. Serrano,et al. Ing1 Mediates p53 Accumulation and Chromatin Modification in Response to Oncogenic Stress* , 2007, Journal of Biological Chemistry.
[16] Seung-Min Yang,et al. Global chromatin compaction limits the strength of the DNA damage response , 2007, The Journal of cell biology.
[17] Jung-Ae Kim,et al. Heterochromatin is refractory to γ-H2AX modification in yeast and mammals , 2007, The Journal of cell biology.
[18] Gang Li,et al. The ING1b tumor suppressor facilitates nucleotide excision repair by promoting chromatin accessibility to XPA. , 2007, Experimental cell research.
[19] Aditi Das,et al. Oxidative DNA damage repair in mammalian cells: a new perspective. , 2007, DNA repair.
[20] C. López-Otín,et al. A functional link between the tumour suppressors ARF and p33ING1 , 2006, Oncogene.
[21] J. Malvehy,et al. Mutation of the tumour suppressor p33ING1b is rare in melanoma , 2006, The British journal of dermatology.
[22] M. Zeremski,et al. Targeted disruption of the mouse ing1 locus results in reduced body size, hypersensitivity to radiation and elevated incidence of lymphomas , 2006, Oncogene.
[23] Song Tan,et al. ING tumor suppressor proteins are critical regulators of chromatin acetylation required for genome expression and perpetuation. , 2006, Molecular cell.
[24] O. Gozani,et al. The fellowships of the INGs , 2005, Journal of cellular biochemistry.
[25] Sheryl K Elkin,et al. A PHD Finger Motif in the C Terminus of RAG2 Modulates Recombination Activity*[boxs] , 2005, Journal of Biological Chemistry.
[26] K. Kumamoto,et al. ING2 Regulates the Onset of Replicative Senescence by Induction of p300-Dependent p53 Acetylation , 2005, Molecular and Cellular Biology.
[27] M. Serrano,et al. Growth Inhibition by the Tumor Suppressor p33ING1 in Immortalized and Primary Cells: Involvement of Two Silencing Domains and Effect of Ras , 2005, Molecular and Cellular Biology.
[28] Michel Nussenzweig,et al. H2AX: the histone guardian of the genome. , 2004, DNA repair.
[29] M. Martinka,et al. Mutations of the ING1 tumor suppressor gene detected in human melanoma abrogate nucleotide excision repair. , 2004, International journal of oncology.
[30] R. Jain,et al. The candidate tumour suppressor protein ING4 regulates brain tumour growth and angiogenesis , 2004, Nature.
[31] J. Qin,et al. Components of a Pathway Maintaining Histone Modification and Heterochromatin Protein 1 Binding at the Pericentric Heterochromatin in Mammalian Cells* , 2004, Journal of Biological Chemistry.
[32] Song Tan,et al. Structural and Functional Conservation of the NuA4 Histone Acetyltransferase Complex from Yeast to Humans , 2004, Molecular and Cellular Biology.
[33] Lai See Po,et al. ING1b decreases cell proliferation through p53‐dependent and ‐independent mechanisms , 2003, FEBS letters.
[34] C. Harris,et al. ING1 represses transcription by direct DNA binding and through effects on p53. , 2003, Cancer research.
[35] J. Lunec,et al. The role of the tumour suppressor p33ING1b in human neoplasia , 2003, Journal of clinical pathology.
[36] Koh Miura,et al. p29ING4 and p28ING5 bind to p53 and p300, and enhance p53 activity. , 2003, Cancer research.
[37] Li Dong Wang,et al. ING1 and p53 tumor suppressor gene alterations in adenocarcinomas of the esophagogastric junction. , 2003, Cancer letters.
[38] J. Yokota,et al. A novel PHD-finger motif protein, p47ING3, modulates p53-mediated transcription, cell cycle control, and apoptosis , 2003, Oncogene.
[39] F. Boisvert,et al. Human ING1 Proteins Differentially Regulate Histone Acetylation* , 2002, The Journal of Biological Chemistry.
[40] M. Serrano,et al. Activation of ARF by oncogenic stress in mouse fibroblasts is independent of E2F1 and E2F2 , 2002, Oncogene.
[41] D. Reinberg,et al. ING 1 Candidate Tumor Suppressor p 33 Complex in Growth Regulation by the Role of the Sin 3-Histone Deacetylase , 2001 .
[42] F. Boisvert,et al. UV-induced binding of ING1 to PCNA regulates the induction of apoptosis. , 2001, Journal of cell science.
[43] X. Wang,et al. DNA damage-inducible gene p33ING2 negatively regulates cell proliferation through acetylation of p53 , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[44] Gang Li,et al. The tumor suppressor candidate p33(ING1) mediates repair of UV-damaged DNA. , 2001, Cancer research.
[45] N. Matsubara,et al. Genetic alterations of candidate tumor suppressor ING1 in human esophageal squamous cell cancer. , 2001, Cancer research.
[46] N. Drouot,et al. Identification of a novel mutation in the autoimmune regulator (AIRE-1) gene in a French family with autoimmune polyendocrinopathy-candidiasis-ectodermal dystrophy. , 2001, European journal of endocrinology.
[47] J. Qin,et al. Differential Association of Products of Alternative Transcripts of the Candidate Tumor Suppressor ING1 with the mSin3/HDAC1 Transcriptional Corepressor Complex* , 2001, The Journal of Biological Chemistry.
[48] G. Li,et al. Expression of the novel tumour suppressor p33ING1 is independent of p53 , 2000, British Journal of Cancer.
[49] S. Nishioka,et al. Genomic structure of the human ING1 gene and tumor-specific mutations detected in head and neck squamous cell carcinomas. , 2000, Cancer research.
[50] E. Rogakou,et al. Megabase Chromatin Domains Involved in DNA Double-Strand Breaks in Vivo , 1999, The Journal of cell biology.
[51] I. Garkavtsev,et al. The candidate tumour suppressor p33ING1cooperates with p53 in cell growth control , 1998, Nature.
[52] S. Aftimos,et al. Mutations in transcriptional regulator ATRX establish the functional significance of a PHD-like domain , 1997, Nature Genetics.
[53] P. Chomczyński,et al. Single-step method of RNA isolation by acid guanidinium thiocyanate-phenol-chloroform extraction. , 1987, Analytical biochemistry.
[54] T. Ellenberger,et al. Mechanism and base specificity of DNA Breakage in intact cells by neocarzinostatin. , 1987, Biochemistry.
[55] T. Hatayama,et al. Action of neocarzinostatin on cell nuclei: release of specific chromatin. , 1982, Biochemical and biophysical research communications.