Interleukin 11 confers resistance to dextran sulfate sodium-induced colitis in mice
暂无分享,去创建一个
H. Nakano | T. Mikami | T. Nishina | S. Yamazaki | Y. Deguchi | Mika Kawauchi | Chen Xiyu
[1] A. Regev,et al. Colon stroma mediates an inflammation-driven fibroblastic response controlling matrix remodeling and healing , 2022, PLoS biology.
[2] I. Wicks,et al. Emerging roles for IL-11 in inflammatory diseases. , 2021, Cytokine.
[3] M. Jackson,et al. IL-1-driven stromal–neutrophil interactions define a subset of patients with inflammatory bowel disease that does not respond to therapies , 2021, Nature Medicine.
[4] C. Garbers,et al. The role of proteolysis in interleukin-11 signaling. , 2021, Biochimica et biophysica acta. Molecular cell research.
[5] C. Lin,et al. Mucin 1 and interleukin-11 protein expression and inflammatory reactions in the intestinal mucosa of necrotizing enterocolitis children after surgery , 2021, World Journal of Clinical Cases.
[6] S. Rose-John,et al. New insights into IL-6 family cytokines in metabolism, hepatology and gastroenterology , 2021, Nature Reviews Gastroenterology & Hepatology.
[7] J. Dear,et al. Redefining IL11 as a regeneration-limiting hepatotoxin and therapeutic target in acetaminophen-induced liver injury , 2021, Science Translational Medicine.
[8] M. van Lookeren Campagne,et al. IL-1R1–dependent signaling coordinates epithelial regeneration in response to intestinal damage , 2021, Science Immunology.
[9] K. Matsushima,et al. Interleukin-11-expressing fibroblasts have a unique gene signature correlated with poor prognosis of colorectal cancer , 2021, Nature Communications.
[10] Judy H. Cho,et al. A myeloid–stromal niche and gp130 rescue in NOD2-driven Crohn’s disease , 2021, Nature.
[11] S. Cook,et al. Transgenic interleukin 11 expression causes cross-tissue fibro-inflammation and an inflammatory bowel phenotype in mice , 2020, PloS one.
[12] Aviv Regev,et al. Intra- and Inter-cellular Rewiring of the Human Colon during Ulcerative Colitis , 2019, Cell.
[13] M. Hibbs,et al. IL-33-mediated mast cell activation promotes gastric cancer through macrophage mobilization , 2019, Nature Communications.
[14] H. Nakano,et al. A murine model of acute lung injury identifies growth factors to promote tissue repair and their biomarkers , 2018, Genes to cells : devoted to molecular & cellular mechanisms.
[15] M. Ohmuraya,et al. Generation of and characterization of anti-IL-11 antibodies using newly established Il11-deficient mice. , 2018, Biochemical and biophysical research communications.
[16] H. Shirzad,et al. The expression analysis of Fra-1 gene and IL-11 protein in Iranian patients with ulcerative colitis , 2018, BMC Immunology.
[17] U. Knaus,et al. NADPH oxidases and ROS signaling in the gastrointestinal tract , 2018, Mucosal Immunology.
[18] Michael Y. Gerner,et al. Innate and adaptive lymphocytes sequentially shape the gut microbiota and lipid metabolism , 2018, Nature.
[19] K. Kharbanda,et al. Dextran sodium sulfate colitis murine model: An indispensable tool for advancing our understanding of inflammatory bowel diseases pathogenesis , 2017, World journal of gastroenterology.
[20] G. Müller-Newen,et al. Oncostatin M drives intestinal inflammation and predicts response to tumor necrosis factor-neutralizing therapy in patients with inflammatory bowel disease , 2017, Zeitschrift für Gastroenterologie.
[21] H. Kiyono,et al. Mucosal Ecological Network of Epithelium and Immune Cells for Gut Homeostasis and Tissue Healing. , 2017, Annual review of immunology.
[22] Richard A. Flavell,et al. The Stromal Intervention: Regulation of Immunity and Inflammation at the Epithelial-Mesenchymal Barrier , 2017, Cell.
[23] H. Nakano,et al. Critical Contribution of Nuclear Factor Erythroid 2-related Factor 2 (NRF2) to Electrophile-induced Interleukin-11 Production* , 2016, The Journal of Biological Chemistry.
[24] G. Kollias,et al. Isolation of Intestinal Mesenchymal Cells from Adult Mice , 2016 .
[25] C. Fiocchi,et al. Immunopathogenesis of IBD: current state of the art , 2016, Nature Reviews Gastroenterology &Hepatology.
[26] Yan Li,et al. Tim-3 promotes intestinal homeostasis in DSS colitis by inhibiting M1 polarization of macrophages. , 2015, Clinical immunology.
[27] T. Denning,et al. Wound repair: role of immune–epithelial interactions , 2015, Mucosal Immunology.
[28] John T. Chang,et al. A gp130–Src–YAP module links inflammation to epithelial regeneration , 2015, Nature.
[29] G. Barton,et al. MyD88: a central player in innate immune signaling , 2014, F1000prime reports.
[30] R. Dobson,et al. The structure of human interleukin-11 reveals receptor-binding site features and structural differences from interleukin-6. , 2014, Acta crystallographica. Section D, Biological crystallography.
[31] S. Reddy,et al. Reactive oxygen species in inflammation and tissue injury. , 2014, Antioxidants & redox signaling.
[32] David Artis,et al. Intestinal epithelial cells: regulators of barrier function and immune homeostasis , 2014, Nature Reviews Immunology.
[33] M. Erard,et al. ROS production in phagocytes: why, when, and where? , 2013, Journal of leukocyte biology.
[34] D. Merlin,et al. Dextran sodium sulfate inhibits the activities of both polymerase and reverse transcriptase: lithium chloride purification, a rapid and efficient technique to purify RNA , 2013, BMC Research Notes.
[35] O. Sieber,et al. Interleukin-11 is the dominant IL-6 family cytokine during gastrointestinal tumorigenesis and can be targeted therapeutically. , 2013, Cancer cell.
[36] D. Threadgill,et al. Tumor fibroblast-derived epiregulin promotes growth of colitis-associated neoplasms through ERK. , 2013, The Journal of clinical investigation.
[37] K. Tsumoto,et al. Interleukin-11 Links Oxidative Stress and Compensatory Proliferation , 2012, Science Signaling.
[38] Sven Kreiborg,et al. Inactivation of IL11 signaling causes craniosynostosis, delayed tooth eruption, and supernumerary teeth. , 2011, American journal of human genetics.
[39] Xin Chen,et al. Mesenchymal cells of the intestinal lamina propria. , 2011, Annual review of physiology.
[40] M. Si-Tahar,et al. Microbiota-induced tertiary lymphoid tissues aggravate inflammatory disease in the absence of RORγt and LTi cells , 2011, The Journal of experimental medicine.
[41] P. Henderson,et al. Function of the intestinal epithelium and its dysregulation in inflammatory bowel disease , 2011, Inflammatory bowel diseases.
[42] Kristel Van Steen,et al. Predictive value of epithelial gene expression profiles for response to infliximab in Crohn's disease‡ , 2010, Inflammatory bowel diseases.
[43] Sanjoy Ghosh,et al. Interleukin-11 reduces TLR4-induced colitis in TLR2-deficient mice and restores intestinal STAT3 signaling. , 2010, Gastroenterology.
[44] J. Tschopp,et al. Colitis induced in mice with dextran sulfate sodium (DSS) is mediated by the NLRP3 inflammasome , 2010, Gut.
[45] M. Neurath,et al. STAT3 links IL-22 signaling in intestinal epithelial cells to mucosal wound healing , 2009, The Journal of experimental medicine.
[46] M. Merad,et al. Lymph-migrating, tissue-derived dendritic cells are minor constituents within steady-state lymph nodes , 2008, The Journal of experimental medicine.
[47] T. Nishimura,et al. Amphiregulin and epiregulin expression in neoplastic and inflammatory lesions in the colon. , 2008, Oncology reports.
[48] M. Boerma,et al. Local administration of interleukin-11 ameliorates intestinal radiation injury in rats. , 2007, Cancer research.
[49] Stefan Wirtz,et al. Chemically induced mouse models of intestinal inflammation , 2007, Nature Protocols.
[50] Mutsumi Matsuu-Matsuyama,et al. The protective effect of interleukin-11 on the cell death induced by X-ray irradiation in cultured intestinal epithelial cell. , 2007, Journal of radiation research.
[51] N. Van Rooijen,et al. Suppression of experimental colitis by intestinal mononuclear phagocytes , 2006, Journal of leukocyte biology.
[52] J. Schulzke,et al. Randomized, Double Blind Controlled Trial of Subcutaneous Recombinant Human Interleukin-11 Versus Prednisolone in Active Crohn's Disease , 2006, The American Journal of Gastroenterology.
[53] D. Littman,et al. The orphan nuclear receptor RORgammat directs the differentiation program of proinflammatory IL-17+ T helper cells. , 2006, Cell.
[54] A. Takayanagi,et al. Interleukin-22, a member of the IL-10 subfamily, induces inflammatory responses in colonic subepithelial myofibroblasts. , 2005, Gastroenterology.
[55] Ruili Li,et al. Interleukin‐11 Receptor Signaling Is Required for Normal Bone Remodeling , 2005, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[56] J. Keith,et al. Protection by recombinant human interleukin-11 against experimental TNB-induced colitis in rats , 1996, Digestive Diseases and Sciences.
[57] V. Godfrey,et al. Epiregulin Is Not Essential for Development of Intestinal Tumors but Is Required for Protection from Intestinal Damage , 2004, Molecular and Cellular Biology.
[58] J. Epplen,et al. A promotor polymorphism in the Interleukin 11 gene is associated with chronic obstructive pulmonary disease , 2004, Electrophoresis.
[59] Yongwon Choi,et al. An essential function for the nuclear receptor RORγt in the generation of fetal lymphoid tissue inducer cells , 2004, Nature Immunology.
[60] Shokei Kim,et al. Regulation of IL-11 expression in intestinal myofibroblasts: role of c-Jun AP-1- and MAPK-dependent pathways. , 2003, American journal of physiology. Gastrointestinal and liver physiology.
[61] M Hocke,et al. A polymorphism in the IL11 gene is associated with ulcerative colitis , 2002, Genes and Immunity.
[62] B. Sands,et al. Randomized, controlled trial of recombinant human interleukin‐11 in patients with active Crohn's disease , 2002, Alimentary pharmacology & therapeutics.
[63] K. Kuenzler,et al. Interleukin-11 enhances intestinal absorptive function after ischemia-reperfusion injury. , 2002, Journal of Pediatric Surgery.
[64] K. Rajewsky,et al. Homeostasis of Peripheral B Cells in the Absence of B Cell Influx from the Bone Marrow , 2001, The Journal of experimental medicine.
[65] Q. Hamid,et al. IL-17 is increased in asthmatic airways and induces human bronchial fibroblasts to produce cytokines. , 2001, The Journal of allergy and clinical immunology.
[66] Simon C Watkins,et al. Intestinal cytokine gene expression in infants with acute necrotizing enterocolitis: interleukin-11 mRNA expression inversely correlates with extent of disease. , 2001, Journal of pediatric surgery.
[67] H. Yonekawa,et al. Diphtheria toxin receptor–mediated conditional and targeted cell ablation in transgenic mice , 2001, Nature Biotechnology.
[68] S. Akira,et al. Targeted disruption of the MyD88 gene results in loss of IL-1- and IL-18-mediated function. , 1998, Immunity.
[69] Weiliang Tang,et al. Transforming Growth Factor-β Stimulates Interleukin-11 Transcription via Complex Activating Protein-1-dependent Pathways* , 1998, The Journal of Biological Chemistry.
[70] M. Maruyama,et al. Interleukin-1alpha and tumor necrosis factor alpha synergistically stimulate prostaglandin E2-dependent production of interleukin-11 in rheumatoid synovial fibroblasts. , 1998, Arthritis and rheumatism.
[71] R. Poulsom,et al. Keratinocyte growth factor in inflammatory bowel disease. Increased mRNA transcripts in ulcerative colitis compared with Crohn's disease in biopsies and isolated mucosal myofibroblasts. , 1997, The American journal of pathology.
[72] C. Begley,et al. Adult mice with targeted mutation of the interleukin-11 receptor (IL11Ra) display normal hematopoiesis. , 1997, Blood.
[73] C. Pothoulakis,et al. IL-11 inhibits Clostridium difficile toxin A enterotoxicity in rat ileum. , 1997, The American journal of physiology.
[74] D. Williams,et al. Interleukin-11 prevents apoptosis and accelerates recovery of small intestinal mucosa in mice treated with combined chemotherapy and radiation. , 1996, Laboratory investigation; a journal of technical methods and pathology.
[75] T. Ohkusa,et al. A novel method in the induction of reliable experimental acute and chronic ulcerative colitis in mice. , 1990, Gastroenterology.
[76] R. Renkawitz,et al. Mouse lysozyme M gene: isolation, characterization, and expression studies. , 1988, Proceedings of the National Academy of Sciences of the United States of America.