Regulation of c-myc and c-fos proto-oncogene expression by animal cell growth factors

[1]  J. Vilček,et al.  Tumor necrosis factor and interleukin-1 cause a rapid and transient stimulation of c-fos and c-myc mRNA levels in human fibroblasts. , 1987, The Journal of biological chemistry.

[2]  R. Taub,et al.  Insulin as a growth factor in rat hepatoma cells. Stimulation of proto-oncogene expression. , 1987, The Journal of biological chemistry.

[3]  Bruce M. Spiegelman,et al.  Nucleoprotein complexes that regulate gene expression in adipocyte differentiation: direct participation of c-fos , 1987, Cell.

[4]  D. Bowtell,et al.  Murine c-myc retroviruses alter the growth requirements of myeloid cell lines. , 1987, Oncogene research.

[5]  A. Kinniburgh,et al.  Relatively stable population of c-myc RNA that lacks long poly(A) , 1987, Molecular and cellular biology.

[6]  E. Rozengurt,et al.  Bombesin induction of c‐fos and c‐myc proto‐oncogenes in Swiss 3T3 cells: Significance for the mitogenic response , 1987, Journal of cellular physiology.

[7]  B. Cochran,et al.  Inducible binding of a factor to the c-fos regulatory region. , 1987, Proceedings of the National Academy of Sciences of the United States of America.

[8]  P. Mccaffrey,et al.  Two independent growth factor-generated signals regulate c-fos and c-myc mRNA levels in Swiss 3T3 cells. , 1987, The Journal of biological chemistry.

[9]  K. Nishikura,et al.  Antisense RNA of proto-oncogene c-fos blocks renewed growth of quiescent 3T3 cells. , 1987, Molecular and cellular biology.

[10]  B. Calabretta.  Dissociation of c-fos induction from macrophage differentiation in human myeloid leukemic cell lines , 1987, Molecular and cellular biology.

[11]  R. Roeder,et al.  Inducible binding of a factor to the c-fos enhancer , 1986, Cell.

[12]  R. Weinberg,et al.  Multiple protein-binding sites in the 5'-flanking region regulate c-fos expression , 1986, Molecular and cellular biology.

[13]  V. Sorrentino,et al.  Potentiation of growth factor activity by exogenous c-myc expression. , 1986, Proceedings of the National Academy of Sciences of the United States of America.

[14]  J. Campisi,et al.  Induction of c-fos and c-myc mRNA by epidermal growth factor or calcium ionophore is cAMP dependent. , 1986, Proceedings of the National Academy of Sciences of the United States of America.

[15]  G. P. Studzinski,et al.  Participation of c-myc protein in DNA synthesis of human cells. , 1986, Science.

[16]  G. Shaw,et al.  A conserved AU sequence from the 3′ untranslated region of GM-CSF mRNA mediates selective mRNA degradation , 1986, Cell.

[17]  Richard Treisman,et al.  Identification of a protein-binding site that mediates transcriptional response of the c-fos gene to serum factors , 1986, Cell.

[18]  G. Colletta,et al.  Induction of the c-fos oncogene by thyrotropic hormone in rat thyroid cells in culture. , 1986, Science.

[19]  G. Sonenshein,et al.  Regulation of c-myc transcription and mRNA abundance by serum growth factors and cell contact. , 1986, The Journal of biological chemistry.

[20]  A. Nienhuis,et al.  Inducible production of c-fos antisense RNA inhibits 3T3 cell proliferation. , 1986, Proceedings of the National Academy of Sciences of the United States of America.

[21]  R. Vallee,et al.  A microtubule-activated ATPase from sea urchin eggs, distinct from cytoplasmic dynein and kinesin. , 1986, Proceedings of the National Academy of Sciences of the United States of America.

[22]  I. Verma,et al.  Proto-oncogene expression in regenerating liver is simulated in cultures of primary adult rat hepatocytes. , 1986, The Journal of biological chemistry.

[23]  Mark Groudine,et al.  A block to elongation is largely responsible for decreased transcription of c-myc in differentiated HL60 cells , 1986, Nature.

[24]  P. Nowell,et al.  Sequential expression of protooncogenes during lectin-stimulated mitogenesis of normal human lymphocytes. , 1986, Proceedings of the National Academy of Sciences of the United States of America.

[25]  R. Mitchell,et al.  c-fos expression is neither sufficient nor obligatory for differentiation of monomyelocytes to macrophages , 1986, Cell.

[26]  T. Curran,et al.  Expression of c‐fos in NIH3T3 cells is very low but inducible throughout the cell cycle. , 1986, The EMBO journal.

[27]  R. Steinman,et al.  Lymphokine and nonlymphokine mRNA levels in stimulated human T cells. Kinetics, mitogen requirements, and effects of cyclosporin A , 1986, The Journal of experimental medicine.

[28]  A. Moses,et al.  Thyrotropin and dibutyryl cyclic AMP increase levels of c-myc and c-fos mRNAs in cultured rat thyroid cells. , 1986, The Journal of biological chemistry.

[29]  B. Beutler,et al.  Identification of a common nucleotide sequence in the 3'-untranslated region of mRNA molecules specifying inflammatory mediators. , 1986, Proceedings of the National Academy of Sciences of the United States of America.

[30]  M. Sporn,et al.  Differential responsiveness of myc- and ras-transfected cells to growth factors: selective stimulation of myc-transfected cells by epidermal growth factor , 1986, Molecular and cellular biology.

[31]  J. Campisi,et al.  c-myc regulation during retinoic acid-induced differentiation of F9 cells is posttranscriptional and associated with growth arrest , 1986, Molecular and cellular biology.

[32]  B. Verrier,et al.  Interleukin‐3‐dependent expression of the c‐myc and c‐fos proto‐oncogenes in hemopoietic cell lines. , 1986, The EMBO journal.

[33]  M. Cole,et al.  Tumorigenicity of fibroblast lines expressing the adenovirus E1a, cellular p53, or normal c-myc genes , 1986, Molecular and cellular biology.

[34]  M. Groudine,et al.  Enhanced transcription of c-myc in bursal lymphoma cells requires continuous protein synthesis. , 1985, Science.

[35]  I. Verma,et al.  Identification of a transcriptional enhancer element upstream from the proto-oncogene fos. , 1985, Science.

[36]  T. Rabbitts,et al.  Truncation of exon 1 from the c‐myc gene results in prolonged c‐myc mRNa stability. , 1985, The EMBO journal.

[37]  C. Stiles,et al.  Platelet-derived growth factor and double-stranded ribonucleic acids stimulate expression of the same genes in 3T3 cells , 1985, Cell.

[38]  G. Lenoir,et al.  Aberrant c‐myc RNAs of Burkitt's lymphoma cells have longer half‐lives. , 1985, The EMBO journal.

[39]  M. Greenberg,et al.  Nerve growth factor and epidermal growth factor induce rapid transient changes in proto-oncogene transcription in PC12 cells. , 1985, The Journal of biological chemistry.

[40]  S. Coughlin,et al.  c-myc gene expression is stimulated by agents that activate protein kinase C and does not account for the mitogenic effect of PDGF , 1985, Cell.

[41]  C. Dani,et al.  c-myc gene is transcribed at high rate in G0-arrested fibroblasts and is post-transcriptionally regulated in response to growth factors , 1985, Nature.

[42]  J. Cleveland,et al.  Abrogation of IL-3 and IL-2 dependence by recombinant murine retroviruses expressing v-myc oncogenes , 1985, Nature.

[43]  Richard Treisman,et al.  Transient accumulation of c-fos RNA following serum stimulation requires a conserved 5′ element and c-fos 3′ sequences , 1985, Cell.

[44]  J. Blanchard,et al.  Posttranscriptional mechanisms are responsible for accumulation of truncated c-myc RNAs in murine plasma cell tumors , 1985, Cell.

[45]  E. Smeland,et al.  The specific induction of myc protooncogene expression in normal human B cells is not a sufficient event for acquisition of competence to proliferate. , 1985, Proceedings of the National Academy of Sciences of the United States of America.

[46]  G. Evan,et al.  Metabolism of c‐myc gene products: c‐myc mRNA and protein expression in the cell cycle. , 1985, The EMBO journal.

[47]  C. Dani,et al.  Increased rate of degradation of c-myc mRNA in interferon-treated Daudi cells. , 1985, Proceedings of the National Academy of Sciences of the United States of America.

[48]  L. Kaczmarek,et al.  Microinjected c-myc as a competence factor. , 1985, Science.

[49]  W. Leonard,et al.  Sequential expression of genes involved in human T lymphocyte growth and differentiation , 1985, The Journal of experimental medicine.

[50]  T. Curran,et al.  Stimulation and inhibition of growth by EGF in different A431 cell clones is accompanied by the rapid induction of c‐fos and c‐myc proto‐oncogenes. , 1985, The EMBO journal.

[51]  E. Antón,et al.  Interferon regulates c-myc gene expression in Daudi cells at the post-transcriptional level. , 1985, Proceedings of the National Academy of Sciences of the United States of America.

[52]  P. Golstein,et al.  Early steps of lymphocyte activation bypassed by synergy between calcium ionophores and phorbol ester , 1985, Nature.

[53]  T. Graf,et al.  Transformation of mammalian fibroblasts and macrophages in vitro by a murine retrovirus encoding an avian v‐myc oncogene. , 1984, The EMBO journal.

[54]  I. Verma,et al.  Expression of the c-fos gene and of an fos-related gene is stimulated by platelet-derived growth factor. , 1984, Science.

[55]  Michael J. Berridge,et al.  Inositol trisphosphate, a novel second messenger in cellular signal transduction , 1984, Nature.

[56]  C. Dani,et al.  Extreme instability of myc mRNA in normal and transformed human cells. , 1984, Proceedings of the National Academy of Sciences of the United States of America.

[57]  Michael E. Greenberg,et al.  Stimulation of 3T3 cells induces transcription of the c-fos proto-oncogene , 1984, Nature.

[58]  L. Lemieux,et al.  Biological activities of v-myc and rearranged c-myc oncogenes in rat fibroblast cells in culture. , 1984, Proceedings of the National Academy of Sciences of the United States of America.

[59]  P. Leder,et al.  Functional role for c-myc in mitogenic response to platelet-derived growth factor , 1984, Nature.

[60]  T. Sugimura,et al.  C-myc transcript is induced in rat liver at a very early stage of regeneration or by cycloheximide treatment , 1984, Nature.

[61]  M. Cole,et al.  Transcriptional activation of the translocated c-myc oncogene in mouse plasmacytomas: Similar RNA levels in tumor and proliferating normal cells , 1984, Cell.

[62]  Y. Nishizuka.  The role of protein kinase C in cell surface signal transduction and tumour promotion , 1984, Nature.

[63]  H. E. Gray,et al.  Cell-cycle control of c-myc but not c-ras expression is lost following chemical transformation , 1984, Cell.

[64]  P. Leder,et al.  Cell-specific regulation of the c-myc gene by lymphocyte mitogens and platelet-derived growth factor , 1983, Cell.

[65]  Brent H. Cochran,et al.  Molecular cloning of gene sequences regulated by platelet-derived growth factor , 1983, Cell.

[66]  R. Ross,et al.  Early changes in phosphatidylinositol and arachidonic acid metabolism in quiescent swiss 3T3 cells stimulated to divide by platelet-derived growth factor. , 1981, The Journal of biological chemistry.

[67]  J. Smith,et al.  Cytoplasmic transfer of the mitogenic response to platelet-derived growth factor. , 1981, Proceedings of the National Academy of Sciences of the United States of America.

[68]  C. Stiles,et al.  Dual control of cell growth by somatomedins and platelet-derived growth factor. , 1979, Proceedings of the National Academy of Sciences of the United States of America.

[69]  C. Stiles,et al.  Induction of DNA synthesis in BALB/c 3T3 cells by serum components: reevaluation of the commitment process. , 1977, Proceedings of the National Academy of Sciences of the United States of America.

[70]  R. Michell.  Inositol phospholipids and cell surface receptor function. , 1975, Biochimica et biophysica acta.

[71]  Eric Wickstrom,et al.  A c-myc antisense oligodeoxynucleotide inhibits entry into S phase but not progress from G0 to G1 , 1987, Nature.

[72]  John Koontzll.  Insulin as a Growth Factor in Rat Hepatoma Cells , 1987 .

[73]  M. Groudine,et al.  Levels of c-myc oncogene mRNA are invariant throughout the cell cycle , 1985, Nature.

[74]  Jonathan A. Cooper,et al.  Platelet-derived growth factor induces rapid but transient expression of the c-fos gene and protein , 1984, Nature.

[75]  T. Curran,et al.  Induction of c-fos gene and protein by growth factors precedes activation of c-myc , 1984, Nature.

[76]  John P. Moore,et al.  Mitogens increase phosphorylation of phosphoinositides in thymocytes , 1984, Nature.