Distinctive gene expression pattern in VH3-21 utilizing B-cell chronic lymphocytic leukemia.
暂无分享,去创建一个
Richard Rosenquist | G. Gahrton | R. Rosenquist | A. Wennborg | Ulf Thunberg | Gösta Gahrton | G. Tobin | Susann Fält | Mats Merup | Gerard Tobin | Anders Wennborg | S. Fält | M. Merup | U. Thunberg
[1] Y. Tu,et al. Gene Expression Profiling of B Cell Chronic Lymphocytic Leukemia Reveals a Homogeneous Phenotype Related to Memory B Cells , 2001, The Journal of experimental medicine.
[2] T J Hamblin,et al. Unmutated Ig V(H) genes are associated with a more aggressive form of chronic lymphocytic leukemia. , 1999, Blood.
[3] A. Hall,et al. Loss of nuclear expression of the p33ING1b inhibitor of growth protein in childhood acute lymphoblastic leukaemia , 2002, Journal of clinical pathology.
[4] D. Catovsky,et al. The immunological profile of B-cell disorders and proposal of a scoring system for the diagnosis of CLL. , 1994, Leukemia.
[5] G. Juliusson,et al. Chronic lymphocytic leukemias utilizing the VH3-21 gene display highly restricted Vlambda2-14 gene use and homologous CDR3s: implicating recognition of a common antigen epitope. , 2003, Blood.
[6] N. Dean,et al. Identification of a Functional Link for the p53 Tumor Suppressor Protein in Dexamethasone-induced Growth Suppression* , 2003, The Journal of Biological Chemistry.
[8] G. Crabtree,et al. Five SWI/SNF-related, matrix-associated, actin-dependent regulator of chromatin (SMARC) genes are dispersed in the human genome. , 1998, Genomics.
[9] Axel Benner,et al. Stromal-derived factor 1 inhibits the cycling of very primitive human hematopoietic cells in vitro and in NOD/SCID mice. , 2002, Blood.
[10] T. Adachi,et al. Involvement of cell cycle progression in survival signaling through CD40 in the B-lymphocyte line WEHI-231 , 2004, Cell Death and Differentiation.
[11] Lai See Po,et al. ING1b decreases cell proliferation through p53‐dependent and ‐independent mechanisms , 2003, FEBS letters.
[12] L. Rassenti,et al. Ig VH1 genes expressed in B cell chronic lymphocytic leukemia exhibit distinctive molecular features. , 1997, Journal of immunology.
[13] Göran Roos,et al. Subsets with restricted immunoglobulin gene rearrangement features indicate a role for antigen selection in the development of chronic lymphocytic leukemia. , 2004, Blood.
[14] N. Chiorazzi,et al. Ig V gene mutation status and CD38 expression as novel prognostic indicators in chronic lymphocytic leukemia. , 1999, Blood.
[15] Weidong Wang,et al. BAF60a Mediates Critical Interactions between Nuclear Receptors and the BRG1 Chromatin-Remodeling Complex for Transactivation , 2003, Molecular and Cellular Biology.
[16] Olivier Poch,et al. Identification of genes associated with tumorigenesis and metastatic potential of hypopharyngeal cancer by microarray analysis , 2004, Oncogene.
[17] J. Kang,et al. Comprehensive genome-wide comparison of DNA and RNA level scan using microarray technology for identification of candidate cancer-related genes in the HL-60 cell line. , 2003, Cancer genetics and cytogenetics.
[18] K. Miyazono,et al. TGF-beta signalling from cell membrane to nucleus through SMAD proteins. , 1997, Nature.
[19] Ash A. Alizadeh,et al. Distinct types of diffuse large B-cell lymphoma identified by gene expression profiling , 2000, Nature.
[20] Brian T Chait,et al. The N-CoR-HDAC3 nuclear receptor corepressor complex inhibits the JNK pathway through the integral subunit GPS2. , 2002, Molecular cell.
[21] R. Rosenquist,et al. VH gene family utilization in different B‐cell lymphoma subgroups , 1999, European journal of haematology.
[22] J. Gribben,et al. Chronic lymphocytic leukemia B cells of more than 1% of patients express virtually identical immunoglobulins. , 2004, Blood.
[23] Bing Zhang,et al. GOTree Machine (GOTM): a web-based platform for interpreting sets of interesting genes using Gene Ontology hierarchies , 2004, BMC Bioinformatics.
[24] G. Parmigiani,et al. Digital karyotyping identifies thymidylate synthase amplification as a mechanism of resistance to 5-fluorouracil in metastatic colorectal cancer patients. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[25] W. Isaacs,et al. High mobility group protein I(Y): a candidate architectural protein for chromosomal rearrangements in prostate cancer cells. , 2002, Cancer research.
[26] C. Li,et al. Model-based analysis of oligonucleotide arrays: expression index computation and outlier detection. , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[27] Arthur Weiss,et al. Expression of ZAP-70 is associated with increased B-cell receptor signaling in chronic lymphocytic leukemia. , 2002, Blood.
[28] G. Dighiero,et al. The pathogenesis of chronic lymphocytic leukemia: analysis of the antibody repertoire. , 1994, Immunology today.
[29] David Botstein,et al. Relation of Gene Expression Phenotype to Immunoglobulin Mutation Genotype in B Cell Chronic Lymphocytic Leukemia , 2001, The Journal of experimental medicine.
[30] S. Sumitomo,et al. Reduced ING1b gene expression plays an important role in carcinogenesis of non-small cell lung cancer patients. , 2003, Clinical cancer research : an official journal of the American Association for Cancer Research.
[31] D. Oscier,et al. Multivariate analysis of prognostic factors in CLL: clinical stage, IGVH gene mutational status, and loss or mutation of the p53 gene are independent prognostic factors. , 2002, Blood.
[32] Adrian Wiestner,et al. ZAP-70 expression identifies a chronic lymphocytic leukemia subtype with unmutated immunoglobulin genes, inferior clinical outcome, and distinct gene expression profile. , 2003, Blood.
[33] M. Keating,et al. Biological and Clinical Heterogeneity of B-cell Chronic Lymphocytic Leukemia , 2003, Leukemia & lymphoma.
[34] Barbara Hoffman,et al. The proto-oncogene c-myc in hematopoietic development and leukemogenesis , 2002, Oncogene.
[35] J. Mesirov,et al. Interpreting patterns of gene expression with self-organizing maps: methods and application to hematopoietic differentiation. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[36] Emili Montserrat,et al. ZAP-70 expression as a surrogate for immunoglobulin-variable-region mutations in chronic lymphocytic leukemia. , 2003, The New England journal of medicine.
[37] Pojen P. Chen,et al. Developmentally restricted immunoglobulin heavy chain variable region gene expressed at high frequency in chronic lymphocytic leukemia. , 1989, Proceedings of the National Academy of Sciences of the United States of America.
[38] Todd,et al. Diffuse large B-cell lymphoma outcome prediction by gene-expression profiling and supervised machine learning , 2002, Nature Medicine.
[39] Göran Roos,et al. Somatically mutated Ig V(H)3-21 genes characterize a new subset of chronic lymphocytic leukemia. , 2002, Blood.
[40] R. Rosenquist,et al. Clonal rearrangements in childhood and adult precursor B acute lymphoblastic leukemia: a comparative polymerase chain reaction study using multiple sets of primers , 1999, European journal of haematology.
[41] R. Rosenquist,et al. VH3-21 Gene Usage in Chronic Lymphocytic Leukemia – Characterization of a New Subgroup with Distinct Molecular Features and Poor Survival , 2004, Leukemia & lymphoma.
[42] H. Niiro,et al. Decision making in the immune system: Regulation of B-cell fate by antigen-receptor signals , 2002, Nature Reviews Immunology.
[43] Ituro Inoue,et al. Regulation of Geminin and Cdt1 expression by E2F transcription factors , 2004, Oncogene.
[44] A. Pettitt,et al. High frequency of p53 dysfunction and low level of VH mutation in chronic lymphocytic leukemia patients using the VH3-21 gene segment. , 2003, Blood.
[45] J. Mesirov,et al. Molecular classification of cancer: class discovery and class prediction by gene expression monitoring. , 1999, Science.
[46] L. Rassenti,et al. Chronic lymphocytic leukemia B cells express restricted sets of mutated and unmutated antigen receptors. , 1998, The Journal of clinical investigation.