Do antidepressants affect motivation in conditioned place preference?
暂无分享,去创建一个
R. Sewell | F. Subhan | P. Deslandes | F. Subhan | D. Pache | David M Pache | Fazal Subhan | Robert D.E Sewell
[1] R. Sewell,et al. Potentiation of opioid-induced conditioned place preference by the selective serotonin reuptake inhibitor fluoxetine. , 2000, European journal of pharmacology.
[2] S. Cheetham,et al. A comparison of the effects on central 5‐HT function of sibutramine hydrochloride and other weight‐modifying agents , 1998, British journal of pharmacology.
[3] E. Esposito,et al. Selective serotonin reuptake inhibitors reduce the spontaneous activity of dopaminergic neurons in the ventral tegmental area , 1998, Brain Research Bulletin.
[4] M. Scheinbaum,et al. Sibutramine: a new weight loss agent without evidence of the abuse potential associated with amphetamines. , 1998, Journal of clinical psychopharmacology.
[5] R. Sewell,et al. The involvement of the opioidergic system in the antinociceptive mechanism of action of antidepressant compounds , 1998, British journal of pharmacology.
[6] C. Nemeroff,et al. Neurotransmitter receptor and transporter binding profile of antidepressants and their metabolites. , 1997, The Journal of pharmacology and experimental therapeutics.
[7] M. Collu,et al. Behavioural sensitization of mesolimbic dopamine D2 receptors in chronic fluoxetine-treated rats. , 1997, European journal of pharmacology.
[8] M. Collu,et al. Fluoxetine‐induced conditioned place preference: A preliminary study , 1997, Synapse.
[9] M. Stock,et al. Sibutramine: a review of the pharmacology of a novel anti-obesity agent. , 1997, International journal of obesity and related metabolic disorders : journal of the International Association for the Study of Obesity.
[10] E. Esposito,et al. Differential effects of acute and chronic fluoxetine administration on the spontaneous activity of dopaminergic neurones in the ventral tegmental area , 1995, British journal of pharmacology.
[11] P. Spencer,et al. Contrasting actions of acute or chronic paroxetine and fluvoxamine on morphine withdrawal-induced place conditioning. , 1995, European journal of pharmacology.
[12] E. Esposito,et al. Serotonin-dopamine interaction in the rat ventral tegmental area: an electrophysiological study in vivo. , 1994, The Journal of pharmacology and experimental therapeutics.
[13] Pagliaro La,et al. Fluoxetine abuse by an intravenous drug user. , 1993 .
[14] P. D’Aquila,et al. Role of the mesolimbic dopamine system in the mechanism of action of antidepressants. , 1992, Pharmacology & toxicology.
[15] H. Fibiger,et al. Chronic desipramine enhances the effect of locally applied amphetamine on interstitial concentrations of dopamine in the nucleus accumbens. , 1991, European journal of pharmacology.
[16] H. Fibiger,et al. Chronic desipramine enhances amphetamine-induced increases in interstitial concentrations of dopamine in the nucleus accumbens. , 1991, European journal of pharmacology.
[17] D. Jd. A case of amitriptyline abuse. , 1990 .
[18] G. Gessa,et al. Central dopaminergic transmission is selectively increased in the limbic system of rats chronically exposed to antidepressants. , 1990, European journal of pharmacology.
[19] G. Aghajanian,et al. Serotonin function and the mechanism of antidepressant action. Reversal of antidepressant-induced remission by rapid depletion of plasma tryptophan. , 1990, Archives of general psychiatry.
[20] Mariusz Papp. Differential effects of short- and long‐term antidepressant treatments on the food‐induced place preference conditioning in rats , 1989, Behavioural Pharmacology.
[21] M. Papp. Different effects of short- and long-term treatment with imipramine on the apomorphine- and food-induced place preference conditioning in rats , 1988, Pharmacology Biochemistry and Behavior.
[22] G. Di Chiara,et al. Drugs abused by humans preferentially increase synaptic dopamine concentrations in the mesolimbic system of freely moving rats. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[23] A. Beaudet,et al. Serotonin axon terminals in the ventral tegmental area of the rat: fine structure and synaptic input to dopaminergic neurons , 1987, Brain Research.
[24] M. L. de Ceballos,et al. Chronic antidepressant treatment increases enkephalin levels in n. accumbens and striatum of the rat. , 1985, European journal of pharmacology.
[25] F. Borsini,et al. Evidence of dopamine involvement in the effect of repeated treatment with various antidepressants in the behavioural 'despair' test in rats. , 1985, European journal of pharmacology.
[26] E. Richelson,et al. Blockade by antidepressants and related compounds of biogenic amine uptake into rat brain synaptosomes: most antidepressants selectively block norepinephrine uptake. , 1984, European journal of pharmacology.
[27] P. Willner. Dopamine and depression: A review of recent evidence. I. Empirical studies , 1983, Brain Research Reviews.
[28] A. Phillips,et al. Cocaine-induced place preference conditioning: Lack of effects of neuroleptics and 6-hydroxydopamine lesions , 1982, Brain Research.
[29] C. Schuster,et al. Abuse liability assessment of sibutramine, a novel weight control agent , 2000, Psychopharmacology.
[30] E. Richelson,et al. Blockade by newly-developed antidepressants of biogenic amine uptake into rat brain synaptosomes. , 1993, Life sciences.
[31] G. Koob. Drugs of abuse: anatomy, pharmacology and function of reward pathways. , 1992, Trends in pharmacological sciences.
[32] P. Willner. Animal models of depression: an overview. , 1990, Pharmacology & therapeutics.
[33] D. Charney,et al. Neuroendocrine and behavioral effects of dietary tryptophan restriction in healthy subjects. , 1989, Life sciences.
[34] H. Meltzer,et al. Psychopharmacology : the third generation of progress , 1987 .
[35] H. Hall. Relationships between receptor affinities of different antidepressants and their clinical profiles , 1983 .
[36] S. Dahl,et al. Clinical Pharmacology in Psychiatry , 1981, Palgrave Macmillan UK.