Id1 regulation of cellular senescence through transcriptional repression of p16/Ink4a
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[1] G. Peters,et al. Opposing effects of Ets and Id proteins on p16INK4a expression during cellular senescence , 2001, Nature.
[2] W. Hahn,et al. Genes involved in senescence and immortalization. , 2000, Current opinion in cell biology.
[3] J D Norton,et al. ID helix-loop-helix proteins in cell growth, differentiation and tumorigenesis. , 2000, Journal of cell science.
[4] M. Diaz,et al. Id-1 delays senescence but does not immortalize keratinocytes. , 2000, The Journal of biological chemistry.
[5] R. DePinho,et al. Cellular Senescence Minireview Mitotic Clock or Culture Shock? , 2000, Cell.
[6] G. Enders,et al. p16INK4a can initiate an autonomous senescence program , 2000, Oncogene.
[7] L. Lania,et al. Class A helix-loop-helix proteins are positive regulators of several cyclin-dependent kinase inhibitors' promoter activity and negatively affect cell growth. , 2000, Cancer research.
[8] C. Murre,et al. Helix-Loop-Helix Proteins: Regulators of Transcription in Eucaryotic Organisms , 2000, Molecular and Cellular Biology.
[9] Marc Montminy,et al. Mutations in NEUROD1 are associated with the development of type 2 diabetes mellitus , 1999, Nature Genetics.
[10] W. Gerald,et al. Id1 and Id3 are required for neurogenesis, angiogenesis and vascularization of tumour xenografts , 1999, Nature.
[11] H. Friess,et al. Copyright © American Society for Investigative Pathology Id-1 and Id-2 Are Overexpressed in Pancreatic Cancer and in Dysplastic Lesions in Chronic Pancreatitis , 2022 .
[12] Robert A. Weinberg,et al. Creation of human tumour cells with defined genetic elements , 1999, Nature.
[13] M. Takata,et al. p16INK4a inactivation is not frequent in uncultured sporadic primary cutaneous melanoma , 1999, Oncogene.
[14] M. Roussel,et al. Induction of ARF tumor suppressor gene expression and cell cycle arrest by transcription factor DMP1. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[15] R. DePinho,et al. The oncogene and Polycomb-group gene bmi-1 regulates cell proliferation and senescence through the ink4a locus , 1999, Nature.
[16] T. Kiyono,et al. Both Rb/p16INK4a inactivation and telomerase activity are required to immortalize human epithelial cells , 1998, Nature.
[17] D. Sidransky,et al. Role of the p16 tumor suppressor gene in cancer. , 1998, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[18] R. Benezra,et al. High incidence of T-cell tumors in E2A-null mice and E2A/Id1 double-knockout mice , 1997, Molecular and cellular biology.
[19] D. Haber. Splicing into Senescence: The Curious Case of p16 and p19ARF , 1997, Cell.
[20] S. T. Park,et al. Regulation of the expression of cyclin-dependent kinase inhibitor p21 by E2A and Id proteins , 1997, Molecular and cellular biology.
[21] Brian D. Dynlacht,et al. Regulation of transcription by proteins that control the cell cycle , 1997, Nature.
[22] J. Campisi. The biology of replicative senescence. , 1997, European journal of cancer.
[23] C. Sherr. Cancer Cell Cycles , 1996, Science.
[24] A. Iavarone,et al. Id2 specifically alters regulation of the cell cycle by tumor suppressor proteins , 1996, Molecular and cellular biology.
[25] R. Benezra,et al. Transcription of the dominant-negative helix-loop-helix protein Id1 is regulated by a protein complex containing the immediate-early response gene Egr-1 , 1996, Molecular and cellular biology.
[26] L. Chin,et al. Role of the INK4a Locus in Tumor Suppression and Cell Mortality , 1996, Cell.
[27] Goberdhan P Dimri,et al. The helix-loop-helix protein Id-1 and a retinoblastoma protein binding mutant of SV40 T antigen synergize to reactivate DNA synthesis in senescent human fibroblasts. , 1996, Developmental genetics.
[28] C Roskelley,et al. A biomarker that identifies senescent human cells in culture and in aging skin in vivo. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[29] F. Haluska,et al. Loss of expression of the p16/cyclin-dependent kinase inhibitor 2 tumor suppressor gene in melanocytic lesions correlates with invasive stage of tumor progression. , 1995, Cancer research.
[30] R. Benezra,et al. The loop region of the helix-loop-helix protein Id1 is critical for its dominant negative activity , 1993, Molecular and cellular biology.
[31] Harold Weintraub,et al. The protein Id: A negative regulator of helix-loop-helix DNA binding proteins , 1990, Cell.