ENU induces mutations in the heart of lacZ transgenic mice.

[1]  J. Heddle.  The role of proliferation in the origin of mutations in mammalian cells. , 1998, Drug metabolism reviews.

[2]  K Rakusan,et al.  Transplantation-induced atrophy of normal and hypertrophic rat hearts: effect on cardiac myocytes and capillaries. , 1997, Journal of molecular and cellular cardiology.

[3]  J. Heddle,et al.  On the origin of spontaneous somatic mutations and sectored plaques detected in transgenic mice. , 1997, Mutation research.

[4]  S Nattel,et al.  Properties of human atrial ICa at physiological temperatures and relevance to action potential. , 1997, The American journal of physiology.

[5]  Bridges Ba.  Mutation in resting cells: the role of endogenous DNA damage. , 1996 .

[6]  T. Skopek,et al.  System Issues: Mutagenic response of the endogenous hprt gene and lacl transgene in benzo[a]pyrene‐treated Big Blue™ B6C3F1 mice , 1996 .

[7]  J. Heddle,et al.  System issues: A test for neutrality of mutations of the lacZ transgene , 1996, Environmental and molecular mutagenesis.

[8]  J. Burbach,et al.  Somatic mutations in the brain: relationship to aging? , 1995, Mutation research.

[9]  J. O’Neill,et al.  Suggestions concerning the relationship between mutant frequency and mutation rate at the hprt locus in human peripheral T-lymphocytes. , 1995, Mutation research.

[10]  J. Vijg,et al.  A selective system for lacZ- phage using a galactose-sensitive E. coli host. , 1993, BioTechniques.

[11]  K. Smith,et al.  Spontaneous mutagenesis: experimental, genetic and other factors. , 1992, Mutation research.

[12]  B. Strauss.  The origin of point mutations in human tumor cells. , 1992, Cancer research.

[13]  T. A. Marino,et al.  Proliferating cell nuclear antigen in developing and adult rat cardiac muscle cells. , 1991, Circulation research.

[14]  B. Hall,et al.  Spontaneous point mutations that occur more often when advantageous than when neutral. , 1990, Genetics.

[15]  B. Ames,et al.  Too many rodent carcinogens: mitogenesis increases mutagenesis. , 1990, Science.

[16]  J. Short,et al.  Development of a short-term, in vivo mutagenesis assay: the effects of methylation on the recovery of a lambda phage shuttle vector from transgenic mice. , 1990, Nucleic acids research.

[17]  J Vijg,et al.  Efficient rescue of integrated shuttle vectors from transgenic mice: a model for studying mutations in vivo. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[18]  P. Burbach,et al.  Age-related development of a heterozygous phenotype in solitary neurons of the homozygous Brattleboro rat. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[19]  J. Overbaugh,et al.  The origin of mutants , 1988, Nature.

[20]  A. Nag,et al.  DNA synthesis in rat heart cells after injury and the regeneration of myocardia. , 1983, Tissue & cell.

[21]  J. O’Neill,et al.  Induction and expression of mutations in mammalian cells in the absence of DNA synthesis and cell division. , 1982, Mutation research.

[22]  D. C. Sheehan,et al.  Theory and Practice of Histotechnology , 1980 .

[23]  K. R. Simmons,et al.  A quantitative histologic analysis of selected tissues in growing mice. , 1970, Developmental biology.

[24]  R. R. Rinehart.  Spontaneous sex-linked recessive lethal frequencies from aged and non-aged spermatozoa of Drosophila melanogaster. , 1969, Mutation research.

[25]  S. Zamenhof,et al.  Mutability of Stored Spores of Bacillus subtilis , 1968, Nature.

[26]  R. Engerman,et al.  Cell turnover of capillaries. , 1967, Laboratory investigation; a journal of technical methods and pathology.