Molecular and mechanical bases of focal lipid accumulation in arterial wall.
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[1] S Chien,et al. Effects of disturbed flow on endothelial cells. , 1998, Journal of biomechanical engineering.
[2] Gerrity Rg,et al. Structural correlates of arterial endothelial permeability in the Evans blue model. , 1977 .
[3] S. Chien,et al. Effects of mechanical forces on signal transduction and gene expression in endothelial cells. , 1998, Hypertension.
[4] S. Weinbaum,et al. Transendothelial Transport of Low Density Lipoprotein in Association with Cell Mitosis in Rat Aorta , 1989, Arteriosclerosis.
[5] T. Karino. Microscopic structure of disturbed flows in the arterial and venous systems, and its implication in the localization of vascular diseases. , 1986, International angiology : a journal of the International Union of Angiology.
[6] B. Helmke,et al. The convergence of haemodynamics, genomics, and endothelial structure in studies of the focal origin of atherosclerosis. , 2002, Biorheology.
[7] C. R. Ethier,et al. Flow waveform effects on end-to-side anastomotic flow patterns. , 1998, Journal of biomechanics.
[8] Clark K. Colton,et al. Microcinematographic studies of flow patterns in the excised rabbit aorta and its major branches. , 1997, Biorheology.
[9] B. Berk,et al. Mitogen-activated protein kinase (ERK1/2) activation by shear stress and adhesion in endothelial cells. Essential role for a herbimycin-sensitive kinase. , 1996, The Journal of clinical investigation.
[10] G. Truskey,et al. Measurement of endothelial permeability to 125I-low density lipoproteins in rabbit arteries by use of en face preparations. , 1992, Circulation research.
[11] M. Schwartz,et al. Integrins: emerging paradigms of signal transduction. , 1995, Annual review of cell and developmental biology.
[12] P. Lelkes,et al. Gene expression profiling of human aortic endothelial cells exposed to disturbed flow and steady laminar flow. , 2002, Physiological genomics.
[13] P. Chuang,et al. Macromolecular transport across arterial and venous endothelium in rats. Studies with Evans blue-albumin and horseradish peroxidase. , 1990, Arteriosclerosis.
[14] S. Weinbaum,et al. Shear stress induces a time- and position-dependent increase in endothelial cell membrane fluidity. , 2001, American journal of physiology. Cell physiology.
[15] J. Weiss,et al. Strain measurement in coronary arteries using intravascular ultrasound and deformable images. , 2002, Journal of biomechanical engineering.
[16] M. Davies,et al. Atherosclerotic plaque caps are locally weakened when macrophages density is increased. , 1991, Atherosclerosis.
[17] C. Lin,et al. Inhibition of neointimal formation in porcine coronary artery by a Ras mutant. , 2001, The Journal of surgical research.
[18] M. Ishida,et al. Shear Stress-mediated Extracellular Signal-regulated Kinase Activation Is Regulated by Sodium in Endothelial Cells , 1999, The Journal of Biological Chemistry.
[19] J A Frangos,et al. Role of G proteins in shear stress-mediated nitric oxide production by endothelial cells. , 1994, The American journal of physiology.
[20] D. Ku,et al. Hemodynamics and atherosclerosis. Insights and perspectives gained from studies of human arteries. , 1988, Archives of pathology & laboratory medicine.
[21] J F Cornhill,et al. Topography of human aortic sudanophilic lesions. , 1990, Monographs on atherosclerosis.
[22] Bernd Nilius,et al. Shear stress induced membrane currents and calcium transients in human vascular endothelial cells , 1992, Pflügers Archiv.
[23] J A Frangos,et al. Fluid shear stress increases membrane fluidity in endothelial cells: a study with DCVJ fluorescence. , 2000, American journal of physiology. Heart and circulatory physiology.
[24] M. Schwartz. Integrin signaling revisited. , 2001, Trends in cell biology.
[25] D. Steinberg. Lipoproteins and atherosclerosis. A look back and a look ahead. , 1983, Arteriosclerosis.
[26] An-Li Huang,et al. Role of intercellular junctions in the passage of horseradish peroxidase across aortic endothelium. , 1992, Laboratory investigation; a journal of technical methods and pathology.
[27] K. Jan,et al. Role of dying endothelial cells in transendothelial macromolecular transport. , 1990, Arteriosclerosis.
[28] S. Usami,et al. Rate sensitivity of shear‐induced changes in the lateral diffusion of endothelial cell membrane lipids: a role for membrane perturbation in shear‐induced MAPK activation , 2002, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[29] A. Barakat,et al. Topographical mapping of sites of enhanced HRP permeability in the normal rabbit aorta. , 1992, Journal of Biomechanical Engineering.
[30] Larry V. McIntire,et al. DNA microarray reveals changes in gene expression of shear stressed human umbilical vein endothelial cells , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[31] K. Fujiwara,et al. Evidence for a role of platelet endothelial cell adhesion molecule-1 in endothelial cell mechanosignal transduction , 2002, The Journal of cell biology.
[32] P. Groot,et al. Chemokines and atherosclerosis. , 1999, Atherosclerosis.
[33] B. Chen,et al. DNA microarray analysis of gene expression in endothelial cells in response to 24-h shear stress. , 2001, Physiological genomics.
[34] R M Nerem,et al. Micropipette Aspiration of Cultured Bovine Aortic Endothelial Cells Exposed to Shear Stress , 1987, Arteriosclerosis.
[35] G. Garcı́a-Cardeña,et al. Biomechanical activation of vascular endothelium as a determinant of its functional phenotype , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[36] C F Dewey,et al. Platelet-derived growth factor B chain promoter contains a cis-acting fluid shear-stress-responsive element. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[37] M J Davies,et al. Lipid and cellular constituents of unstable human aortic plaques. , 1994, Basic research in cardiology.
[38] A. Szuba,et al. The role of chemokines in human cardiovascular pathology: enhanced biological insights. , 2002, Atherosclerosis.
[39] Elisabetta Dejana,et al. VEGF receptor 2 and the adherens junction as a mechanical transducer in vascular endothelial cells , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[40] K. Yoshida,et al. New role of glycosaminoglycans on the plasma membrane proposed by their interaction with phosphatidylcholine , 2000, FEBS letters.
[41] P. Davies,et al. Flow-mediated endothelial mechanotransduction. , 1995, Physiological reviews.
[42] T. Carew,et al. Quantification In Vivo of Increased LDL Content and Rate of LDL Degradation in Normal Rabbit Aorta Occurring at Sites Susceptible to Early Atherosclerotic Lesions , 1988, Circulation research.
[43] T. Carew,et al. Initiation of atherosclerotic lesions in cholesterol-fed rabbits. I. Focal increases in arterial LDL concentration precede development of fatty streak lesions. , 1989, Arteriosclerosis.
[44] S. Diamond,et al. Tissue plasminogen activator messenger RNA levels increase in cultured human endothelial cells exposed to laminar shear stress , 1990, Journal of cellular physiology.
[45] K. Sipos,et al. Differential Effect of Shear Stress on Extracellular Signal-regulated Kinase and N-terminal Jun Kinase in Endothelial Cells , 1997, The Journal of Biological Chemistry.
[46] S Q Liu,et al. Prevention of focal intimal hyperplasia in rat vein grafts by using a tissue engineering approach. , 1998, Atherosclerosis.
[47] R M Nerem,et al. Hemodynamics and the vascular endothelium. , 1993, Journal of biomechanical engineering.
[48] T. Hunter,et al. Fluid Shear Stress Activation of Focal Adhesion Kinase , 1997, The Journal of Biological Chemistry.
[49] S. Chien,et al. Integrin-mediated mechanotransduction requires its dynamic interaction with specific extracellular matrix (ECM) ligands. , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[50] Ronald W. Davis,et al. Quantitative Monitoring of Gene Expression Patterns with a Complementary DNA Microarray , 1995, Science.
[51] Jiahuai Han,et al. The cis-acting phorbol ester "12-O-tetradecanoylphorbol 13-acetate"-responsive element is involved in shear stress-induced monocyte chemotactic protein 1 gene expression. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[52] K. Jan,et al. Ultrastructural studies on macromolecular permeability in relation to endothelial cell turnover. , 1995, Atherosclerosis.
[53] S. Weinbaum,et al. Effect of cell turnover and leaky junctions on arterial macromolecular transport. , 1985, The American journal of physiology.
[54] P. Davies,et al. Dual effect of fluid shear stress on volume-regulated anion current in bovine aortic endothelial cells. , 2002, American journal of physiology. Cell physiology.
[55] C F Dewey,et al. Turbulent fluid shear stress induces vascular endothelial cell turnover in vitro. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[56] M. Karin,et al. The Ras-JNK pathway is involved in shear-induced gene expression , 1996, Molecular and cellular biology.
[57] Shu Chien,et al. Mechanotransduction in Response to Shear Stress , 1999, The Journal of Biological Chemistry.
[58] P. Davies,et al. Haemodynamic shear stress activates a K+ current in vascular endothelial cells , 1988, Nature.
[59] D. Steinberg,et al. Role of oxidized LDL and antioxidants in atherosclerosis. , 1995, Advances in experimental medicine and biology.
[60] S. Chien,et al. Effects of active and negative mutants of Ras on rat arterial neointima formation. , 2000, The Journal of surgical research.
[61] Roger J. Davis,et al. Transcriptional regulation by MAP kinases , 1995, Molecular reproduction and development.
[62] R. Gerrity,et al. Endothelial cell morphology in areas of in vivo Evans blue uptake in the aorta of young pigs. II. Ultrastructure of the intima in areas of differing permeability to proteins. , 1977, The American journal of pathology.
[63] S. Weinbaum,et al. The role of arterial endothelial cell mitosis in macromolecular permeability. , 1988, Advances in experimental medicine and biology.
[64] S. Chien,et al. Shear stress activates p60src-Ras-MAPK signaling pathways in vascular endothelial cells. , 1998, Arteriosclerosis, thrombosis, and vascular biology.
[65] B. Chen,et al. Shear Stress Activation of SREBP1 in Endothelial Cells Is Mediated by Integrins , 2002, Arteriosclerosis, thrombosis, and vascular biology.
[66] Y. Kaneda,et al. Local Overexpression of Monocyte Chemoattractant Protein‐1 at Vessel Wall Induces Infiltration of Macrophages and Formation of Atherosclerotic Lesion: Synergism With Hypercholesterolemia , 2002, Arteriosclerosis, thrombosis, and vascular biology.
[67] S. Chien,et al. Interplay between integrins and FLK-1 in shear stress-induced signaling. , 2002, American journal of physiology. Cell physiology.
[68] S Chien,et al. Fluid shear stress induces a biphasic response of human monocyte chemotactic protein 1 gene expression in vascular endothelium. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[69] G. Truskey,et al. The distribution of intimal white blood cells in the normal rabbit aorta. , 1995, Atherosclerosis.
[70] S. Usami,et al. Molecular mechanism of endothelial growth arrest by laminar shear stress. , 2000, Proceedings of the National Academy of Sciences of the United States of America.