Integrating case-control and TDT studies.
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[1] D. Clayton,et al. A generalization of the transmission/disequilibrium test for uncertain-haplotype transmission. , 1999, American journal of human genetics.
[2] J. Witte,et al. Genetic dissection of complex traits. , 1994, Nature genetics.
[3] W J Ewens,et al. The TDT and other family-based tests for linkage disequilibrium and association. , 1996, American journal of human genetics.
[4] C. Molony,et al. Overdispersion of allele frequency differences between populations: implications for meta-analyses of genotypic disease associations , 2005, European Journal of Human Genetics.
[5] N Risch,et al. The Future of Genetic Studies of Complex Human Diseases , 1996, Science.
[6] M. Munafo,et al. Meta-analysis of genetic association studies. , 2004, Trends in genetics : TIG.
[7] L. Excoffier,et al. Maximum-likelihood estimation of molecular haplotype frequencies in a diploid population. , 1995, Molecular biology and evolution.
[8] P. Donnelly,et al. Association mapping in structured populations. , 2000, American journal of human genetics.
[9] G. Oehlert. A note on the delta method , 1992 .
[10] P. Wilson,et al. A genome scan for loci linked to quantitative insulin traits in persons without diabetes: the Framingham Offspring Study , 2003, Diabetologia.
[11] P. Donnelly,et al. The effects of human population structure on large genetic association studies , 2004, Nature Genetics.
[12] S. Gabriel,et al. Assessing the impact of population stratification on genetic association studies , 2004, Nature Genetics.
[13] L. Cardon,et al. Association study designs for complex diseases , 2001, Nature Reviews Genetics.
[14] J. Staessen,et al. The deletion/insertion polymorphism of the angiotensin converting enzyme gene and cardiovascular‐renal risk , 1997, Journal of hypertension.
[15] N. Risch. Searching for genetic determinants in the new millennium , 2000, Nature.
[16] J. Levy,et al. Polymorphisms in type II SH2 domain-containing inositol 5-phosphatase (INPPL1, SHIP2) are associated with physiological abnormalities of the metabolic syndrome. , 2004, Diabetes.
[17] S. Faraone,et al. Association between a functional catechol O-methyltransferase gene polymorphism and schizophrenia: meta-analysis of case-control and family-based studies. , 2003, The American journal of psychiatry.
[18] Lon R. Cardon,et al. The complex interplay among factors that influence allelic association , 2004, Nature Reviews Genetics.
[19] L. Wasserman,et al. Genomic control, a new approach to genetic-based association studies. , 2001, Theoretical population biology.
[20] W. Ewens,et al. Transmission test for linkage disequilibrium: the insulin gene region and insulin-dependent diabetes mellitus (IDDM). , 1993, American journal of human genetics.
[21] L. Cardon,et al. Population stratification and spurious allelic association , 2003, The Lancet.
[22] L. Hedges,et al. Statistical Methods for Meta-Analysis , 1987 .
[23] L. E. Mitchell. Relationship between case‐control studies and the transmission/disequilibrium test , 2000, Genetic epidemiology.