Apocynin and Nox2 regulate NF-κB by modifying thioredoxin-1 redox-state

[1]  Kyungho Kim,et al.  NOX2 is critical for heterotypic neutrophil-platelet interactions during vascular inflammation. , 2015, Blood.

[2]  A. Holmgren,et al.  The thioredoxin antioxidant system. , 2014, Free radical biology & medicine.

[3]  A. Holmgren,et al.  Thioredoxin 1 Is Inactivated Due to Oxidation Induced by Peroxiredoxin under Oxidative Stress and Reactivated by the Glutaredoxin System* , 2013, The Journal of Biological Chemistry.

[4]  V. Visvanathan N-acetylcysteine for sepsis and systemic inflammatory response in adults. , 2013, Critical care nurse.

[5]  Jiyang Cai,et al.  NADPH Oxidase Limits Lipopolysaccharide-Induced Lung Inflammation and Injury in Mice through Reduction-Oxidation Regulation of NF-κB Activity , 2013, The Journal of Immunology.

[6]  P. Boggiatto,et al.  NOX2 Protects against Prolonged Inflammation, Lung Injury, and Mortality following Systemic Insults , 2013, Journal of Innate Immunity.

[7]  S. Houser,et al.  Blockade of NOX2 and STIM1 signaling limits lipopolysaccharide-induced vascular inflammation. , 2013, The Journal of clinical investigation.

[8]  L. Cesaro,et al.  Inhibition of thioredoxin reductase by lanthanum chloride. , 2012, Journal of inorganic biochemistry.

[9]  K. Krause,et al.  Hyperinflammation of chronic granulomatous disease is abolished by NOX2 reconstitution in macrophages and dendritic cells , 2012, The Journal of pathology.

[10]  C. Scavone,et al.  Eye enucleation activates the transcription nuclear factor kappa-B in the rat superior colliculus , 2012, Neuroscience Letters.

[11]  J. Snyder,et al.  Ebselen and congeners inhibit NADPH oxidase 2-dependent superoxide generation by interrupting the binding of regulatory subunits. , 2012, Chemistry & biology.

[12]  R. Linhardt,et al.  Trimer hydroxylated quinone derived from apocynin targets cysteine residues of p47phox preventing the activation of human vascular NADPH oxidase. , 2012, Free radical biology & medicine.

[13]  Yusuke Murayama,et al.  Unravelling cerebellar pathways with high temporal precision targeting motor and extensive sensory and parietal networks , 2012, Nature Communications.

[14]  F. Cunha,et al.  Essential role of CCR2 in neutrophil tissue infiltration and multiple organ dysfunction in sepsis. , 2011, American journal of respiratory and critical care medicine.

[15]  A. Towbin,et al.  Chronic granulomatous disease , 2010, Pediatric Radiology.

[16]  J. Winther,et al.  An introduction to methods for analyzing thiols and disulfides: Reactions, reagents, and practical considerations. , 2009, Analytical biochemistry.

[17]  Dean P. Jones,et al.  Thioredoxin redox western analysis. , 2009, Current protocols in toxicology.

[18]  J. Engelhardt,et al.  Signaling components of redox active endosomes: the redoxosomes. , 2009, Antioxidants & redox signaling.

[19]  Mauro M. Teixeira,et al.  Regulation of chemokine receptor by Toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis , 2009, Proceedings of the National Academy of Sciences.

[20]  A. Adler,et al.  The IKK‐neutralizing compound Bay11 kills supereffector CD8 T cells by altering caspase‐dependent activation‐induced cell death , 2008, Journal of leukocyte biology.

[21]  R. Hancock,et al.  ROS-deficient monocytes have aberrant gene expression that correlates with inflammatory disorders of chronic granulomatous disease. , 2008, Clinical immunology.

[22]  M. Gougerot-Pocidalo,et al.  Priming of the neutrophil NADPH oxidase activation: role of p47phox phosphorylation and NOX2 mobilization to the plasma membrane , 2008, Seminars in Immunopathology.

[23]  S. Blecher,et al.  Expression of cell surface receptors and oxidative metabolism modulation in the clinical continuum of sepsis , 2008, Critical care.

[24]  Shizuo Akira,et al.  Signaling to NF-?B by Toll-like receptors , 2007 .

[25]  I. L. Brunetti,et al.  The reactivity of ortho-methoxy-substituted catechol radicals with sulfhydryl groups: contribution for the comprehension of the mechanism of inhibition of NADPH oxidase by apocynin. , 2007, Biochemical pharmacology.

[26]  M. S. Galhiane,et al.  The oxidation of apocynin catalyzed by myeloperoxidase: proposal for NADPH oxidase inhibition. , 2007, Archives of biochemistry and biophysics.

[27]  P. Hawkins,et al.  PtdIns3P binding to the PX domain of p40phox is a physiological signal in NADPH oxidase activation , 2006, The EMBO journal.

[28]  B. Vargaftig,et al.  Systemic administration of interleukin‐2 inhibits inflammatory neutrophil migration: role of nitric oxide , 2006, British journal of pharmacology.

[29]  F. Hu,et al.  Lanthanum inhibited the binding of LPS with monocyte and CD 14 expression upregulation. , 2004, Cellular & Molecular Immunology.

[30]  A. Holmgren,et al.  Ebselen: A substrate for human thioredoxin reductase strongly stimulating its hydroperoxide reductase activity and a superfast thioredoxin oxidant , 2002, Proceedings of the National Academy of Sciences of the United States of America.

[31]  R. Schirmer,et al.  Thioredoxin reductase as a pathophysiological factor and drug target. , 2000, European journal of biochemistry.

[32]  M. Matsui,et al.  Demonstration of the interaction of thioredoxin with p40phox, a phagocyte oxidase component, using a yeast two-hybrid system. , 1999, Immunology letters.

[33]  D. Männel,et al.  Role of NFkappaB in the mortality of sepsis. , 1997, The Journal of clinical investigation.

[34]  J. Yodoi,et al.  Thiordoxin regulates the DNA binding activity of NF-χB by reduction of a disulphid bond involving cysteine 62 , 1992 .

[35]  W. Leonard,et al.  Modulation of transcription factor NF-kappa B binding activity by oxidation-reduction in vitro. , 1991, Proceedings of the National Academy of Sciences of the United States of America.

[36]  N. Rajasekaran,et al.  Reductive potential - a savior turns stressor in protein aggregation cardiomyopathy. , 2015, Biochimica et biophysica acta.

[37]  J. Quevedo,et al.  Sepsis-associated brain dysfunction in critically ill patients , 2013, Critical Care.

[38]  G. Decavalas,et al.  Severe Sepsis and Septic Shock , 2018 .

[39]  YangWang,et al.  Lanthanum Inhibited the Binding of LPS with Monocyte and CD14 Expression Upregulation , 2004 .

[40]  A. Holmgren,et al.  Selenite and selenodiglutathione: reactions with thioredoxin systems. , 1995, Methods in enzymology.

[41]  David A. Williams,et al.  Mouse model of X–linked chronic granulomatous disease, an inherited defect in phagocyte superoxide production , 1995, Nature Genetics.

[42]  A. Holmgren,et al.  [22] Selenite and selenodiglutathione: Reactions with thioredoxin systems , 1995 .

[43]  J. Yodoi,et al.  Thioredoxin regulates the DNA binding activity of NF-kappa B by reduction of a disulphide bond involving cysteine 62. , 1992, Nucleic acids research.