Apocynin and Nox2 regulate NF-κB by modifying thioredoxin-1 redox-state
暂无分享,去创建一个
A. Shah | C. Scavone | R. Curi | L. Lopes | F. Laurindo | F. Cunha | J. Alves-Filho | J. Hothersall | A. Ivetic | A. Cobb | T. Cunha | S. Trevelin | Min Zhang | P. Roxo-Junior | Célio Xavier dos Santos | M. Cervi | L. de Sá Lima | R. Ferreira | R. L. Silva | Andrew M. Cobb
[1] Kyungho Kim,et al. NOX2 is critical for heterotypic neutrophil-platelet interactions during vascular inflammation. , 2015, Blood.
[2] A. Holmgren,et al. The thioredoxin antioxidant system. , 2014, Free radical biology & medicine.
[3] A. Holmgren,et al. Thioredoxin 1 Is Inactivated Due to Oxidation Induced by Peroxiredoxin under Oxidative Stress and Reactivated by the Glutaredoxin System* , 2013, The Journal of Biological Chemistry.
[4] V. Visvanathan. N-acetylcysteine for sepsis and systemic inflammatory response in adults. , 2013, Critical care nurse.
[5] Jiyang Cai,et al. NADPH Oxidase Limits Lipopolysaccharide-Induced Lung Inflammation and Injury in Mice through Reduction-Oxidation Regulation of NF-κB Activity , 2013, The Journal of Immunology.
[6] P. Boggiatto,et al. NOX2 Protects against Prolonged Inflammation, Lung Injury, and Mortality following Systemic Insults , 2013, Journal of Innate Immunity.
[7] S. Houser,et al. Blockade of NOX2 and STIM1 signaling limits lipopolysaccharide-induced vascular inflammation. , 2013, The Journal of clinical investigation.
[8] L. Cesaro,et al. Inhibition of thioredoxin reductase by lanthanum chloride. , 2012, Journal of inorganic biochemistry.
[9] K. Krause,et al. Hyperinflammation of chronic granulomatous disease is abolished by NOX2 reconstitution in macrophages and dendritic cells , 2012, The Journal of pathology.
[10] C. Scavone,et al. Eye enucleation activates the transcription nuclear factor kappa-B in the rat superior colliculus , 2012, Neuroscience Letters.
[11] J. Snyder,et al. Ebselen and congeners inhibit NADPH oxidase 2-dependent superoxide generation by interrupting the binding of regulatory subunits. , 2012, Chemistry & biology.
[12] R. Linhardt,et al. Trimer hydroxylated quinone derived from apocynin targets cysteine residues of p47phox preventing the activation of human vascular NADPH oxidase. , 2012, Free radical biology & medicine.
[13] Yusuke Murayama,et al. Unravelling cerebellar pathways with high temporal precision targeting motor and extensive sensory and parietal networks , 2012, Nature Communications.
[14] F. Cunha,et al. Essential role of CCR2 in neutrophil tissue infiltration and multiple organ dysfunction in sepsis. , 2011, American journal of respiratory and critical care medicine.
[15] A. Towbin,et al. Chronic granulomatous disease , 2010, Pediatric Radiology.
[16] J. Winther,et al. An introduction to methods for analyzing thiols and disulfides: Reactions, reagents, and practical considerations. , 2009, Analytical biochemistry.
[17] Dean P. Jones,et al. Thioredoxin redox western analysis. , 2009, Current protocols in toxicology.
[18] J. Engelhardt,et al. Signaling components of redox active endosomes: the redoxosomes. , 2009, Antioxidants & redox signaling.
[19] Mauro M. Teixeira,et al. Regulation of chemokine receptor by Toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis , 2009, Proceedings of the National Academy of Sciences.
[20] A. Adler,et al. The IKK‐neutralizing compound Bay11 kills supereffector CD8 T cells by altering caspase‐dependent activation‐induced cell death , 2008, Journal of leukocyte biology.
[21] R. Hancock,et al. ROS-deficient monocytes have aberrant gene expression that correlates with inflammatory disorders of chronic granulomatous disease. , 2008, Clinical immunology.
[22] M. Gougerot-Pocidalo,et al. Priming of the neutrophil NADPH oxidase activation: role of p47phox phosphorylation and NOX2 mobilization to the plasma membrane , 2008, Seminars in Immunopathology.
[23] S. Blecher,et al. Expression of cell surface receptors and oxidative metabolism modulation in the clinical continuum of sepsis , 2008, Critical care.
[24] Shizuo Akira,et al. Signaling to NF-?B by Toll-like receptors , 2007 .
[25] I. L. Brunetti,et al. The reactivity of ortho-methoxy-substituted catechol radicals with sulfhydryl groups: contribution for the comprehension of the mechanism of inhibition of NADPH oxidase by apocynin. , 2007, Biochemical pharmacology.
[26] M. S. Galhiane,et al. The oxidation of apocynin catalyzed by myeloperoxidase: proposal for NADPH oxidase inhibition. , 2007, Archives of biochemistry and biophysics.
[27] P. Hawkins,et al. PtdIns3P binding to the PX domain of p40phox is a physiological signal in NADPH oxidase activation , 2006, The EMBO journal.
[28] B. Vargaftig,et al. Systemic administration of interleukin‐2 inhibits inflammatory neutrophil migration: role of nitric oxide , 2006, British journal of pharmacology.
[29] F. Hu,et al. Lanthanum inhibited the binding of LPS with monocyte and CD 14 expression upregulation. , 2004, Cellular & Molecular Immunology.
[30] A. Holmgren,et al. Ebselen: A substrate for human thioredoxin reductase strongly stimulating its hydroperoxide reductase activity and a superfast thioredoxin oxidant , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[31] R. Schirmer,et al. Thioredoxin reductase as a pathophysiological factor and drug target. , 2000, European journal of biochemistry.
[32] M. Matsui,et al. Demonstration of the interaction of thioredoxin with p40phox, a phagocyte oxidase component, using a yeast two-hybrid system. , 1999, Immunology letters.
[33] D. Männel,et al. Role of NFkappaB in the mortality of sepsis. , 1997, The Journal of clinical investigation.
[34] J. Yodoi,et al. Thiordoxin regulates the DNA binding activity of NF-χB by reduction of a disulphid bond involving cysteine 62 , 1992 .
[35] W. Leonard,et al. Modulation of transcription factor NF-kappa B binding activity by oxidation-reduction in vitro. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[36] N. Rajasekaran,et al. Reductive potential - a savior turns stressor in protein aggregation cardiomyopathy. , 2015, Biochimica et biophysica acta.
[37] J. Quevedo,et al. Sepsis-associated brain dysfunction in critically ill patients , 2013, Critical Care.
[38] G. Decavalas,et al. Severe Sepsis and Septic Shock , 2018 .
[39] YangWang,et al. Lanthanum Inhibited the Binding of LPS with Monocyte and CD14 Expression Upregulation , 2004 .
[40] A. Holmgren,et al. Selenite and selenodiglutathione: reactions with thioredoxin systems. , 1995, Methods in enzymology.
[41] David A. Williams,et al. Mouse model of X–linked chronic granulomatous disease, an inherited defect in phagocyte superoxide production , 1995, Nature Genetics.
[42] A. Holmgren,et al. [22] Selenite and selenodiglutathione: Reactions with thioredoxin systems , 1995 .
[43] J. Yodoi,et al. Thioredoxin regulates the DNA binding activity of NF-kappa B by reduction of a disulphide bond involving cysteine 62. , 1992, Nucleic acids research.