Combined but not Individual Administration of β-Adrenergic and Serotonergic Antagonists Impairs Water Maze Acquisition in the Rat
暂无分享,去创建一个
[1] Francis Boon,et al. Retrosplenial cortex lesions impair water maze strategies learning or spatial place learning depending on prior experience of the rat , 2006, Behavioural Brain Research.
[2] M. E. Corcoran,et al. Thalamic and hippocampal mechanisms in spatial navigation: A dissociation between brain mechanisms for learning how versus learning where to navigate , 2006, Behavioural Brain Research.
[3] J. Raftery,et al. Long-term donepezil treatment in 565 patients with Alzheimer's disease (AD2000): randomised double-blind trial , 2004, The Lancet.
[4] D. Cain,et al. Contribution of sex differences in the acute stress response to sex differences in water maze performance in the rat , 2004, Behavioural Brain Research.
[5] Howard J Federoff,et al. Current Treatment for Alzheimer Disease and Future Prospects , 2003, Alzheimer disease and associated disorders.
[6] D. Cain,et al. Detailed behavioral analysis reveals both task strategies and spatial memory impairments in rats given bilateral middle cerebral artery stroke , 2003, Brain Research.
[7] D. Cain,et al. Combined β-Adrenergic and Cholinergic Antagonism Produces Behavioral and Cognitive Impairments in the Water Maze: Implications for Alzheimer Disease and Pharmacotherapy with β-Adrenergic Antagonists , 2003, Neuropsychopharmacology.
[8] I. Whishaw,et al. Variation in visual acuity within pigmented, and between pigmented and albino rat strains , 2002, Behavioural Brain Research.
[9] D. Cain,et al. Ethanol impairs behavioral strategy use in naive rats but does not prevent spatial learning in the water maze in pretrained rats , 2002, Psychopharmacology.
[10] M. Esiri,et al. Postmortem serotoninergic correlates of cognitive decline in Alzheimer's disease , 2002, Neuroreport.
[11] I. Whishaw,et al. Impaired Spatial Performance in Rats with Retrosplenial Lesions: Importance of the Spatial Problem and the Rat Strain in Identifying Lesion Effects in a Swimming Pool , 2002, The Journal of Neuroscience.
[12] H. Dringenberg,et al. Alzheimer's disease: more than a ‘cholinergic disorder' — evidence that cholinergic–monoaminergic interactions contribute to EEG slowing and dementia , 2000, Behavioural Brain Research.
[13] L. Cahill,et al. Impaired Memory Consolidation in Rats Produced with β-Adrenergic Blockade , 2000, Neurobiology of Learning and Memory.
[14] M. Esiri,et al. Immunocytochemical study of the dorsal and median raphe nuclei in patients with Alzheimer’s disease prospectively assessed for behavioural changes , 2000, Neuropathology and applied neurobiology.
[15] D. Cain,et al. Individual and combined manipulation of muscarinic, NMDA, and benzodiazepine receptor activity in the water maze task: implications for a rat model of Alzheimer dementia , 2000, Behavioural Brain Research.
[16] K. Davis,et al. Neuropeptide abnormalities in patients with early Alzheimer disease. , 1999, Archives of general psychiatry.
[17] B. Lawlor,et al. A randomized trial of risperidone, placebo, and haloperidol for behavioral symptoms of dementia , 1999, Neurology.
[18] N. Singewald,et al. Dependence of serotonin release in the locus coeruleus on dorsal raphe neuronal activity , 1999, Naunyn-Schmiedeberg's Archives of Pharmacology.
[19] D. Cain,et al. Complex Behavioral Strategy and Reversal Learning in the Water Maze without NMDA Receptor-Dependent Long-Term Potentiation , 1999, The Journal of Neuroscience.
[20] C. Hölscher. Stress impairs performance in spatial water maze learning tasks , 1999, Behavioural Brain Research.
[21] G. Wilcock,et al. The cholinergic hypothesis of Alzheimer’s disease: a review of progress , 1999, Journal of neurology, neurosurgery, and psychiatry.
[22] James L. McGaugh,et al. Stress and glucocorticoids impair retrieval of long-term spatial memory , 1998, Nature.
[23] D. Cain. Testing the NMDA, Long-term Potentiation, and Cholinergic Hypotheses of Spatial Learning , 1998, Neuroscience & Biobehavioral Reviews.
[24] J. Leslie,et al. Effects of Propranolol, Buspirone, pCPA, Reserpine, and Chlordiazepoxide on Open-Field Behavior , 1998, Pharmacology Biochemistry and Behavior.
[25] D. Cain,et al. The effect of nonspatial water maze pretraining in rats subjected to serotonin depletion and muscarinic receptor antagonism: a detailed behavioural assessment of spatial performance , 1997, Behavioural Brain Research.
[26] D. Cain. Prior non-spatial pretraining eliminates sensorimotor disturbances and impairments in water maze learning caused by diazepam , 1997, Psychopharmacology.
[27] D. Cain,et al. Testing hypotheses of spatial learning: the role of NMDA receptors and NMDA-mediated long-term potentiation , 1997, Behavioural Brain Research.
[28] C. de Montigny,et al. Modulation of the firing activity of noradrenergic neurones in the rat locus coeruleus by the 5‐hydroxtryptamine system , 1997, British journal of pharmacology.
[29] M. Kavaliers,et al. Sex differences in performance in the Morris water maze and the effects of initial nonstationary hidden platform training. , 1996, Behavioral neuroscience.
[30] M. T. Pyne,et al. Effects of captopril and propranolol on cognitive function and cerebral blood flow in aged hypertensive rats. , 1996, The journals of gerontology. Series A, Biological sciences and medical sciences.
[31] B. Costall,et al. Combined pCPA and muscarinic antagonist treatment produces a deficit in rat water maze acquisition , 1996, Pharmacology Biochemistry and Behavior.
[32] C. H. Vanderwolf,et al. The role of brain noradrenaline in cortical activation and behavior: A study of lesions of the locus coeruleus, medial thalamus and hippocampus-neocortex and of muscarinic blockade in the rat , 1996, Behavioural Brain Research.
[33] J. Hall,et al. Detailed behavioral analysis of water maze acquisition under APV or CNQX: contribution of sensorimotor disturbances to drug-induced acquisition deficits. , 1996, Behavioral neuroscience.
[34] C. H. Vanderwolf,et al. Detailed behavioral analysis of water maze acquisition under systemic NMDA or muscarinic antagonism: nonspatial pretraining eliminates spatial learning deficits. , 1996, Behavioral neuroscience.
[35] D. Cain,et al. Spatial learning without NMDA receptor-dependent long-term potentiation , 1995, Nature.
[36] R. Morris,et al. Distinct components of spatial learning revealed by prior training and NMDA receptor blockade , 1995, Nature.
[37] I. Whishaw,et al. Rats with fimbria-fornix lesions display a place response in a swimming pool: a dissociation between getting there and knowing where , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[38] C. H. Vanderwolf,et al. p-Chlorophenylalanine-induced serotonin depletion: reduction in exploratory locomotion but no obvious sensory-motor deficits , 1995, Behavioural Brain Research.
[39] R. J. McDonald,et al. Dissociation of the medial prefrontal, posterior parietal, and posterior temporal cortex for spatial navigation and recognition memory in the rat. , 1994, Cerebral cortex.
[40] D. P. Cain,et al. Polypropylene pellets as an inexpensive reusable substitute for milk in the Morris milk maze , 1993, Journal of Neuroscience Methods.
[41] J. Sirviö,et al. Effects of concurrent nicotinic antagonist and PCPA treatments on spatial and passive avoidance learning , 1992, Brain Research.
[42] J L McGaugh,et al. The role of interactions between the cholinergic system and other neuromodulatory systems in learing and memory , 1991, Synapse.
[43] H. Soininen,et al. Neurotransmitter changes in alzheimer's disease: Implications to diagnostics and therapy , 1990, Journal of neuroscience research.
[44] J. D. McGaugh,et al. Concurrent muscarinic and β-adrenergic blockade in rats impairs place-learning in a water maze and retention of inhibitory avoidance , 1990, Brain Research.
[45] I. Whishaw. Dissociating performance and learning deficits on spatial navigation tasks in rats subjected to cholinergic muscarinic blockade , 1989, Brain Research Bulletin.
[46] R. Morris. Synaptic plasticity and learning: selective impairment of learning rats and blockade of long-term potentiation in vivo by the N-methyl-D- aspartate receptor antagonist AP5 , 1989, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[47] J. Davis,et al. Species differences in the localization and number of CNS beta adrenergic receptors: rat versus guinea pig. , 1989, The Journal of pharmacology and experimental therapeutics.
[48] M. Segal,et al. Spatial performance is severely impaired in rats with combined reduction of serotonergic and cholinergic transmission , 1989, Brain Research.
[49] C. Fairburn,et al. Changes in Behaviour in Dementia: A Neglected Research Area , 1988, British Journal of Psychiatry.
[50] E B Larson,et al. Behavioral Disturbance in Dementia of the Alzheimer's Type , 1988, Journal of the American Geriatrics Society.
[51] I. Whishaw,et al. Cholinergic receptor blockade produces impairments in a sensorimotor subsystem for place navigation in the rat: evidence from sensory, motor, and acquisition tests in a swimming pool. , 1987, Behavioral neuroscience.
[52] C. H. Vanderwolf. Near-total loss of ‘learning’ and ‘memory’ as a result of combined cholinergic and serotonergic blockade in the rat , 1987, Behavioural Brain Research.
[53] I. Whishaw,et al. Earlier is not always better: Behavioral dysfunction and abnormal cerebral morphogenesis following neonatal cortical lesions in the rat , 1985, Behavioural Brain Research.
[54] T. Iwasaki,et al. Effects of cholinergic and monoaminergic antagonists and tranquilizers upon spatial memory in rats , 1984, Pharmacology Biochemistry and Behavior.
[55] C. Saper. Organization of cerebral cortical afferent systems in the rat. II. Magnocellular basal nucleus , 1984, The Journal of comparative neurology.
[56] J. Coyle,et al. Topographic analysis of the innervation of the rat neocortex and hippocampus by the basal forebrain cholinergic system , 1983, The Journal of comparative neurology.
[57] J. R. Rush,et al. Spatial working memory in rats: Effects of monoaminergic antagonists , 1983, Pharmacology Biochemistry and Behavior.
[58] R. Morris,et al. Place navigation impaired in rats with hippocampal lesions , 1982, Nature.
[59] T. Robinson,et al. Electrical activity of the hippocampus and neocortex in rats depleted of brain dopamine and norepinephrine: Relations to behavior and effects of atropine , 1978, Experimental Neurology.
[60] Barbara E. Jones,et al. Effects of locus coeruleus lesions upon cerebral monoamine content, sleep-wakefulness states and the response to amphetamine in the cat , 1977, Brain Research.
[61] A. Weissman,et al. p-Chlorophenylalanine: a specific depletor of brain serotonin. , 1966, The Journal of pharmacology and experimental therapeutics.
[62] C J CLEMEDSON,et al. DYNAMIC RESPONSE OF CHEST WALL AND LUNG INJURIES IN RABBITS EXPOSED TO AIR SHOCK WAVES OF SHORT DURATION. , 1964, Acta physiologica Scandinavica. Supplementum.
[63] R. Morris,et al. Dissociation between components of spatial memory in rats after recovery from the effects of retrohippocampal lesions , 2004, Experimental Brain Research.
[64] J. F. Brody. Behavioral effects of serotonin depletion and of p-chlorophenylalanine (a serotonin depletor) in rats , 2004, Psychopharmacologia.
[65] I. Whishaw,et al. Similarities vs. differences in place learning and circadian activity in rats after fimbria‐fornix section or ibotenate removal of hippocampal cells , 1995, Hippocampus.
[66] C. H. Vanderwolf,et al. Potentiation of the Effects of Antimuscarinic Drugs on Behavior by Serotonin Depletion: Specificity and Relation to Learning and Memory , 1992 .
[67] L. Butcher,et al. Neurotransmitter Interactions and Cognitive Function , 1992, Birkhäuser Boston.
[68] C. H. Vanderwolf. Cerebral activity and behavior: control by central cholinergic and serotonergic systems. , 1988, International review of neurobiology.
[69] K. Fuxe,et al. EVIDENCE FOR THE EXISTENCE OF MONOAMINE-CONTAINING NEURONS IN THE CENTRAL NERVOUS SYSTEM. I. DEMONSTRATION OF MONOAMINES IN THE CELL BODIES OF BRAIN STEM NEURONS. , 1964, Acta physiologica Scandinavica. Supplementum.